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731 related items for PubMed ID: 11266442

  • 1. Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells.
    Shimizu S, Matsuoka Y, Shinohara Y, Yoneda Y, Tsujimoto Y.
    J Cell Biol; 2001 Jan 22; 152(2):237-50. PubMed ID: 11266442
    [Abstract] [Full Text] [Related]

  • 2. Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity.
    Shimizu S, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Jan 18; 97(2):577-82. PubMed ID: 10639121
    [Abstract] [Full Text] [Related]

  • 3. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
    Shimizu S, Narita M, Tsujimoto Y.
    Nature; 1999 Jun 03; 399(6735):483-7. PubMed ID: 10365962
    [Abstract] [Full Text] [Related]

  • 4. Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim.
    Sugiyama T, Shimizu S, Matsuoka Y, Yoneda Y, Tsujimoto Y.
    Oncogene; 2002 Jul 25; 21(32):4944-56. PubMed ID: 12118373
    [Abstract] [Full Text] [Related]

  • 5. Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator.
    Shimizu S, Shinohara Y, Tsujimoto Y.
    Oncogene; 2000 Sep 07; 19(38):4309-18. PubMed ID: 10980606
    [Abstract] [Full Text] [Related]

  • 6. VDAC2 inhibits BAK activation and mitochondrial apoptosis.
    Cheng EH, Sheiko TV, Fisher JK, Craigen WJ, Korsmeyer SJ.
    Science; 2003 Jul 25; 301(5632):513-7. PubMed ID: 12881569
    [Abstract] [Full Text] [Related]

  • 7. Bid, but not Bax, regulates VDAC channels.
    Rostovtseva TK, Antonsson B, Suzuki M, Youle RJ, Colombini M, Bezrukov SM.
    J Biol Chem; 2004 Apr 02; 279(14):13575-83. PubMed ID: 14729675
    [Abstract] [Full Text] [Related]

  • 8. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
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  • 11. BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death.
    Shimizu S, Konishi A, Kodama T, Tsujimoto Y.
    Proc Natl Acad Sci U S A; 2000 Mar 28; 97(7):3100-5. PubMed ID: 10737788
    [Abstract] [Full Text] [Related]

  • 12. Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis.
    Desagher S, Osen-Sand A, Nichols A, Eskes R, Montessuit S, Lauper S, Maundrell K, Antonsson B, Martinou JC.
    J Cell Biol; 1999 Mar 08; 144(5):891-901. PubMed ID: 10085289
    [Abstract] [Full Text] [Related]

  • 13. Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death.
    Wei MC, Zong WX, Cheng EH, Lindsten T, Panoutsakopoulou V, Ross AJ, Roth KA, MacGregor GR, Thompson CB, Korsmeyer SJ.
    Science; 2001 Apr 27; 292(5517):727-30. PubMed ID: 11326099
    [Abstract] [Full Text] [Related]

  • 14. Direct addition of BimL to mitochondria does not lead to cytochrome c release.
    Terradillos O, Montessuit S, Huang DC, Martinou JC.
    FEBS Lett; 2002 Jul 03; 522(1-3):29-34. PubMed ID: 12095614
    [Abstract] [Full Text] [Related]

  • 15. Two pathways for tBID-induced cytochrome c release from rat brain mitochondria: BAK- versus BAX-dependence.
    Brustovetsky N, Dubinsky JM, Antonsson B, Jemmerson R.
    J Neurochem; 2003 Jan 03; 84(1):196-207. PubMed ID: 12485416
    [Abstract] [Full Text] [Related]

  • 16. Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.
    Kim TH, Zhao Y, Barber MJ, Kuharsky DK, Yin XM.
    J Biol Chem; 2000 Dec 15; 275(50):39474-81. PubMed ID: 10982793
    [Abstract] [Full Text] [Related]

  • 17. Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax.
    Werner AB, de Vries E, Tait SW, Bontjer I, Borst J.
    J Biol Chem; 2002 Jun 21; 277(25):22781-8. PubMed ID: 11929871
    [Abstract] [Full Text] [Related]

  • 18. Apoptosis. Key to the mitochondrial gate.
    Martinou JC.
    Nature; 1999 Jun 03; 399(6735):411-2. PubMed ID: 10365950
    [No Abstract] [Full Text] [Related]

  • 19. Mouse uterine epithelial apoptosis is associated with expression of mitochondrial voltage-dependent anion channels, release of cytochrome C from mitochondria, and the ratio of Bax to Bcl-2 or Bcl-X.
    Takagi-Morishita Y, Yamada N, Sugihara A, Iwasaki T, Tsujimura T, Terada N.
    Biol Reprod; 2003 Apr 03; 68(4):1178-84. PubMed ID: 12606449
    [Abstract] [Full Text] [Related]

  • 20. Induction of cell death by the BH3-only Bcl-2 homolog Nbk/Bik is mediated by an entirely Bax-dependent mitochondrial pathway.
    Gillissen B, Essmann F, Graupner V, Stärck L, Radetzki S, Dörken B, Schulze-Osthoff K, Daniel PT.
    EMBO J; 2003 Jul 15; 22(14):3580-90. PubMed ID: 12853473
    [Abstract] [Full Text] [Related]

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