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Journal Abstract Search

444 related items for PubMed ID: 11284033

  • 21. Two new translocations involving the 11q23 region map outside the MLL locus in myeloid leukemias.
    Giugliano E, Rege-Cambrin G, Scaravaglio P, Wlodarska I, Emanuel B, Stul M, Serra A, Tonso A, Pini M, Saglio G, Hagemeijer A.
    Haematologica; 2002 Oct; 87(10):1014-20. PubMed ID: 12368154
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  • 22. MLL gene amplification in acute myeloid leukemia and myelodysplastic syndromes is associated with characteristic clinicopathological findings and TP53 gene mutation.
    Tang G, DiNardo C, Zhang L, Ravandi F, Khoury JD, Huh YO, Muzzafar T, Medeiros LJ, Wang SA, Bueso-Ramos CE.
    Hum Pathol; 2015 Jan; 46(1):65-73. PubMed ID: 25387813
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  • 23. Comparison of cytogenetics, Southern blotting, and fluorescence in situ hybridization as methods for detecting MLL gene rearrangements in children with acute leukemia and with 11q23 abnormalities.
    Mathew S, Behm F, Dalton J, Raimondi S.
    Leukemia; 1999 Nov; 13(11):1713-20. PubMed ID: 10557043
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  • 24. Acute myeloid leukemia with MLL rearrangements: clinicobiological features, prognostic impact and value of flow cytometry in the detection of residual leukemic cells.
    Muñoz L, Nomdedéu JF, Villamor N, Guardia R, Colomer D, Ribera JM, Torres JP, Berlanga JJ, Fernández C, Llorente A, Queipo de Llano MP, Sánchez JM, Brunet S, Sierra J, Spanish CETLAM Group.
    Leukemia; 2003 Jan; 17(1):76-82. PubMed ID: 12529663
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  • 26. Internal tandem duplications of the FLT3 and MLL genes are mainly observed in atypical cases of therapy-related acute myeloid leukemia with a normal karyotype and are unrelated to type of previous therapy.
    Christiansen DH, Pedersen-Bjergaard J.
    Leukemia; 2001 Dec; 15(12):1848-51. PubMed ID: 11753604
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  • 29. Detection of 11q23/MLL rearrangements in infant leukemias with fluorescence in situ hybridization and molecular analysis.
    Martinez-Climent JA, Thirman MJ, Espinosa R, Le Beau MM, Rowley JD.
    Leukemia; 1995 Aug; 9(8):1299-304. PubMed ID: 7643616
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  • 31. Mutations with loss of heterozygosity of p53 are common in therapy-related myelodysplasia and acute myeloid leukemia after exposure to alkylating agents and significantly associated with deletion or loss of 5q, a complex karyotype, and a poor prognosis.
    Christiansen DH, Andersen MK, Pedersen-Bjergaard J.
    J Clin Oncol; 2001 Mar 01; 19(5):1405-13. PubMed ID: 11230485
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  • 32. Identification of cytogenetic subclasses and recurring chromosomal aberrations in AML and MDS with complex karyotypes using M-FISH.
    Van Limbergen H, Poppe B, Michaux L, Herens C, Brown J, Noens L, Berneman Z, De Bock R, De Paepe A, Speleman F.
    Genes Chromosomes Cancer; 2002 Jan 01; 33(1):60-72. PubMed ID: 11746988
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  • 33. MLL amplification in acute myeloid leukemia.
    Pajuelo-Gámez JC, Cervera J, García-Casado Z, Mena-Durán AV, Valencia A, Barragán E, Such E, Bolufer P, Sanz MA.
    Cancer Genet Cytogenet; 2007 Apr 15; 174(2):127-31. PubMed ID: 17452254
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  • 34. A novel t(11;12)(q23-24;q24) in a case of minimally-differentiated acute myeloid leukemia (AML-M0).
    Cox MC, Scanzani A, Del Poeta G, Venditti A, Panetta P, Derme V, Sgro R, Masi M, Amadori S.
    Cancer Genet Cytogenet; 2000 Apr 01; 118(1):76-9. PubMed ID: 10731597
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  • 37. Centromeric breakage and highly rearranged chromosome derivatives associated with mutations of TP53 are common in therapy-related MDS and AML after therapy with alkylating agents: an M-FISH study.
    Andersen MK, Christiansen DH, Pedersen-Bjergaard J.
    Genes Chromosomes Cancer; 2005 Apr 01; 42(4):358-71. PubMed ID: 15645489
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  • 40. Double minute chromosomes in acute myeloid leukemia, myelodysplastic syndromes, and chronic myelomonocytic leukemia are associated with micronuclei, MYC or MLL amplification, and complex karyotype.
    Huh YO, Tang G, Talwalkar SS, Khoury JD, Ohanian M, Bueso-Ramos CE, Abruzzo LV.
    Cancer Genet; 2016 Apr 01; 209(7-8):313-20. PubMed ID: 27318442
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