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Journal Abstract Search

95 related items for PubMed ID: 11293243

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  • 3. [The roles of mitochondrial permeability transition in brain ischemia].
    Kobayashi T.
    Hokkaido Igaku Zasshi; 2000 Jul; 75(4):243-52. PubMed ID: 10976404
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  • 4. Glycine enhances extracellular 45Ca2+ response to NMDA application investigated with microdialysis of rabbit hippocampus in vivo.
    Lazarewicz JW, Salińska E, Puka M.
    Acta Neurobiol Exp (Wars); 1992 Jul; 52(2):83-91. PubMed ID: 1414510
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  • 5. beta-estradiol, dehydroepiandrosterone, and dehydroepiandrosterone sulfate protect against N-methyl-D-aspartate-induced neurotoxicity in rat hippocampal neurons by different mechanisms.
    Kurata K, Takebayashi M, Morinobu S, Yamawaki S.
    J Pharmacol Exp Ther; 2004 Oct; 311(1):237-45. PubMed ID: 15175425
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  • 6. N-methyl-D-aspartate-evoked release of cyclo-oxygenase products in rabbit hippocampus: an in vivo microdialysis study.
    Lazarewicz JW, Salińska E.
    J Neurosci Res; 1995 Apr 01; 40(5):660-6. PubMed ID: 7602616
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  • 7. Role of calcium in glutamate-mediated toxicity: mechanisms of calcium fluxes in rabbit hippocampus in vivo investigated with microdialysis.
    Lazarewicz JW, Salińska E.
    Acta Neurobiol Exp (Wars); 1993 Apr 01; 53(1):3-13. PubMed ID: 8100377
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  • 8. Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.
    Sullivan PG, Thompson MB, Scheff SW.
    Exp Neurol; 1999 Nov 01; 160(1):226-34. PubMed ID: 10630207
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  • 13. Role of intracellular Ca(2+) stores shaping normal activity in brain.
    Nyitrai G, Kovács I, Szárics E, Skuban N, Juhász G, Kardos J.
    J Neurosci Res; 1999 Sep 15; 57(6):906-15. PubMed ID: 10467262
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  • 14. Nitric oxide applications prior and simultaneous to potentially excitotoxic NMDA-evoked calcium transients: cell death or survival.
    Schröter A, Andrabi SA, Wolf G, Horn TF.
    Brain Res; 2005 Oct 26; 1060(1-2):1-15. PubMed ID: 16199018
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  • 15. Cyclosporin A enhances survival, ameliorates brain damage, and prevents secondary mitochondrial dysfunction after a 30-minute period of transient cerebral ischemia.
    Li PA, Kristián T, He QP, Siesjö BK.
    Exp Neurol; 2000 Sep 26; 165(1):153-63. PubMed ID: 10964494
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  • 16. N-methyl-D-aspartic acid-induced and Ca-dependent neuronal swelling and its retardation by brain-derived neurotrophic factor in the epileptic hippocampus.
    Isokawa M.
    Neuroscience; 2005 Sep 26; 131(4):801-12. PubMed ID: 15749335
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  • 17. Inhibition of Calpain Prevents N-Methyl-D-aspartate-Induced Degeneration of the Nucleus Basalis and Associated Behavioral Dysfunction.
    Nimmrich V, Szabo R, Nyakas C, Granic I, Reymann KG, Schröder UH, Gross G, Schoemaker H, Wicke K, Möller A, Luiten P.
    J Pharmacol Exp Ther; 2008 Nov 26; 327(2):343-52. PubMed ID: 18701765
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  • 18. Neuroprotective efficacy of FR901459, a novel derivative of cyclosporin A, in in vitro mitochondrial damage and in vivo transient cerebral ischemia models.
    Muramatsu Y, Furuichi Y, Tojo N, Moriguchi A, Maemoto T, Nakada H, Hino M, Matsuoka N.
    Brain Res; 2007 May 29; 1149():181-90. PubMed ID: 17391653
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  • 20. Susceptibility to excitotoxicity in aged hippocampal cultures and neuroprotection by non-steroidal anti-inflammatory drugs: role of mitochondrial calcium.
    Calvo M, Sanz-Blasco S, Caballero E, Villalobos C, Núñez L.
    J Neurochem; 2015 Feb 29; 132(4):403-17. PubMed ID: 25492611
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