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PUBMED FOR HANDHELDS

Journal Abstract Search


261 related items for PubMed ID: 11298356

  • 1.
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  • 2. LACK-reactive CD4+ T cells require autocrine IL-2 to mediate susceptibility to Leishmania major.
    Gumy A, Aseffa A, Rachinel N, Breton M, Otten L, Tacchini-Cottier F, Röcken M, Doyen N, Acha-Orbea H, Locksley RM, MacDonald HR, Launois P, Louis J.
    Eur J Immunol; 2006 Jun; 36(6):1465-73. PubMed ID: 16637008
    [Abstract] [Full Text] [Related]

  • 3. The IL-4 rapidly produced in BALB/c mice after infection with Leishmania major down-regulates IL-12 receptor beta 2-chain expression on CD4+ T cells resulting in a state of unresponsiveness to IL-12.
    Himmelrich H, Parra-Lopez C, Tacchini-Cottier F, Louis JA, Launois P.
    J Immunol; 1998 Dec 01; 161(11):6156-63. PubMed ID: 9834101
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  • 5. IL-4 instructs TH1 responses and resistance to Leishmania major in susceptible BALB/c mice.
    Biedermann T, Zimmermann S, Himmelrich H, Gumy A, Egeter O, Sakrauski AK, Seegmüller I, Voigt H, Launois P, Levine AD, Wagner H, Heeg K, Louis JA, Röcken M.
    Nat Immunol; 2001 Nov 01; 2(11):1054-60. PubMed ID: 11600887
    [Abstract] [Full Text] [Related]

  • 6. Early production of IL-4 and induction of Th2 responses in the lymph node originate from an MHC class I-independent CD4+NK1.1- T cell population.
    von der Weid T, Beebe AM, Roopenian DC, Coffman RL.
    J Immunol; 1996 Nov 15; 157(10):4421-7. PubMed ID: 8906817
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  • 7. LeIF: a recombinant Leishmania protein that induces an IL-12-mediated Th1 cytokine profile.
    Skeiky YA, Kennedy M, Kaufman D, Borges MM, Guderian JA, Scholler JK, Ovendale PJ, Picha KS, Morrissey PJ, Grabstein KH, Campos-Neto A, Reed SG.
    J Immunol; 1998 Dec 01; 161(11):6171-9. PubMed ID: 9834103
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  • 8.
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  • 9. Induction of a Th2 population from a polarized Leishmania-specific Th1 population by in vitro culture with IL-4.
    Mocci S, Coffman RL.
    J Immunol; 1995 Apr 15; 154(8):3779-87. PubMed ID: 7706719
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  • 11. The mechanism of in vitro T helper cell type 1 to T helper cell type 2 switching in highly polarized Leishmania major-specific T cell populations.
    Mocci S, Coffman RL.
    J Immunol; 1997 Feb 15; 158(4):1559-64. PubMed ID: 9029090
    [Abstract] [Full Text] [Related]

  • 12. Some of the early events underlying Th2 cell maturation and susceptibility to Leishmania major infection in BALB/c mice.
    Himmelrich H, Launois P, Tacchini-Cottier F, Louis JA.
    Biol Chem; 1999 Feb 15; 380(7-8):909-14. PubMed ID: 10494841
    [Abstract] [Full Text] [Related]

  • 13. Blockade of CD86 in BALB/c mice infected with Leishmania major does not prevent the expansion of low avidity T cells.
    Moro M, Filippi C, Gallard A, Malherbe L, Foucras G, Akiba H, Yagita H, Guéry JC, Glaichenhaus N.
    Eur J Immunol; 2002 Dec 15; 32(12):3566-75. PubMed ID: 12516542
    [Abstract] [Full Text] [Related]

  • 14. Leishmania major-infected C3H mice treated with anti-IL-12 mAb develop but do not maintain a Th2 response.
    Hondowicz BD, Scharton-Kersten TM, Jones DE, Scott P.
    J Immunol; 1997 Nov 15; 159(10):5024-31. PubMed ID: 9366430
    [Abstract] [Full Text] [Related]

  • 15. Early IL-4 production does not predict susceptibility to Leishmania major.
    Scott P, Eaton A, Gause WC, di Zhou X, Hondowicz B.
    Exp Parasitol; 1996 Nov 15; 84(2):178-87. PubMed ID: 8932767
    [Abstract] [Full Text] [Related]

  • 16. IL-4- and IL-4 receptor-deficient BALB/c mice reveal differences in susceptibility to Leishmania major parasite substrains.
    Noben-Trauth N, Paul WE, Sacks DL.
    J Immunol; 1999 May 15; 162(10):6132-40. PubMed ID: 10229856
    [Abstract] [Full Text] [Related]

  • 17. In susceptible mice, Leishmania major induce very rapid interleukin-4 production by CD4+ T cells which are NK1.1-.
    Launois P, Ohteki T, Swihart K, MacDonald HR, Louis JA.
    Eur J Immunol; 1995 Dec 15; 25(12):3298-307. PubMed ID: 8566015
    [Abstract] [Full Text] [Related]

  • 18. Rapid IL-4 production by Leishmania homolog of mammalian RACK1-reactive CD4(+) T cells in resistant mice treated once with anti-IL-12 or -IFN-gamma antibodies at the onset of infection with Leishmania major instructs Th2 cell development, resulting in nonhealing lesions.
    Launois P, Gumy A, Himmelrich H, Locksley RM, Röcken M, Louis JA.
    J Immunol; 2002 May 01; 168(9):4628-35. PubMed ID: 11971011
    [Abstract] [Full Text] [Related]

  • 19. T-cell responses to immunodominant LACK antigen do not play a critical role in determining susceptibility of BALB/c mice to Leishmania mexicana.
    Torrentera FA, Glaichenhaus N, Laman JD, Carlier Y.
    Infect Immun; 2001 Jan 01; 69(1):617-21. PubMed ID: 11119565
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  • 20. Changes in the precursor frequencies of IL-4 and IFN-gamma secreting CD4+ cells correlate with resolution of lesions in murine cutaneous leishmaniasis.
    Morris L, Troutt AB, Handman E, Kelso A.
    J Immunol; 1992 Oct 15; 149(8):2715-21. PubMed ID: 1357029
    [Abstract] [Full Text] [Related]


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