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Journal Abstract Search

302 related items for PubMed ID: 11347199

  • 1. [The role of TEL and AML1 genes in the pathogenesis of hematologic malignancies].
    Zuna J.
    Cas Lek Cesk; 2001 Mar 15; 140(5):131-7. PubMed ID: 11347199
    [Abstract] [Full Text] [Related]

  • 2. Expression levels of TEL, AML1, and the fusion products TEL-AML1 and AML1-TEL versus drug sensitivity and clinical outcome in t(12;21)-positive pediatric acute lymphoblastic leukemia.
    Stams WA, den Boer ML, Beverloo HB, Meijerink JP, van Wering ER, Janka-Schaub GE, Pieters R.
    Clin Cancer Res; 2005 Apr 15; 11(8):2974-80. PubMed ID: 15837750
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  • 3. Fluorescence in situ hybridization study of TEL/AML1 fusion and other abnormalities involving TEL and AML1 genes. Correlation with cytogenetic findings and prognostic value in children with acute lymphocytic leukemia.
    Martínez-Ramírez A, Urioste M, Contra T, Cantalejo A, Tavares A, Portero JA, López-Ibor B, Bernacer M, Soto C, Cigudosa JC, Benítez J.
    Haematologica; 2001 Dec 15; 86(12):1245-53. PubMed ID: 11726315
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  • 4. The AML1 gene: a transcription factor involved in the pathogenesis of myeloid and lymphoid leukemias.
    Lo Coco F, Pisegna S, Diverio D.
    Haematologica; 1997 Dec 15; 82(3):364-70. PubMed ID: 9234595
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  • 5. Analysis of TEL proteins in human leukemias.
    Poirel H, Lacronique V, Mauchauffé M, Le Coniat M, Raffoux E, Daniel MT, Erickson P, Drabkin H, MacLeod RA, Drexler HG, Ghysdael J, Berger R, Bernard OA.
    Oncogene; 1998 Jun 04; 16(22):2895-903. PubMed ID: 9671410
    [Abstract] [Full Text] [Related]

  • 6. TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis.
    Shurtleff SA, Buijs A, Behm FG, Rubnitz JE, Raimondi SC, Hancock ML, Chan GC, Pui CH, Grosveld G, Downing JR.
    Leukemia; 1995 Dec 04; 9(12):1985-9. PubMed ID: 8609706
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  • 7. Lack of TEL-AML1 fusion transcript resulting from a cryptic t(12;21) in adult B lineage acute lymphoblastic leukemia in Taiwan.
    Shih LY, Chou TB, Liang DC, Tzeng YS, Rubnitz JE, Downing JR, Pui CH.
    Leukemia; 1996 Sep 04; 10(9):1456-8. PubMed ID: 8751462
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  • 9. TEL deletion analysis supports a novel view of relapse in childhood acute lymphoblastic leukemia.
    Zuna J, Ford AM, Peham M, Patel N, Saha V, Eckert C, Köchling J, Panzer-Grümayer R, Trka J, Greaves M.
    Clin Cancer Res; 2004 Aug 15; 10(16):5355-60. PubMed ID: 15328172
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  • 11. High incidence of TEL/AML1 fusion resulting from a cryptic t(12;21) in childhood B-lineage acute lymphoblastic leukemia in Taiwan.
    Liang DC, Chou TB, Chen JS, Shurtleff SA, Rubnitz JE, Downing JR, Pui CH, Shih LY.
    Leukemia; 1996 Jun 15; 10(6):991-3. PubMed ID: 8667657
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  • 13. [Detection of BCR/ABL, MLL/AF4 and TEL/AML1 hybrid genes and monitoring of minimal residual disease in pediatric patients with acute lymphoblastic leukemia].
    Trka J, Zuna J, Haskovec C, Brabencová A, Kalinová M, Muzíková K, Paukertová R, Hrusák O, Zemanová Z, Michalová K, Starý J.
    Cas Lek Cesk; 1999 Jan 04; 138(1):12-7. PubMed ID: 10953429
    [Abstract] [Full Text] [Related]

  • 14. TEL/AML1 shows dominant-negative effects over TEL as well as AML1.
    Gunji H, Waga K, Nakamura F, Maki K, Sasaki K, Nakamura Y, Mitani K.
    Biochem Biophys Res Commun; 2004 Sep 17; 322(2):623-30. PubMed ID: 15325275
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  • 16. TEL/AML1-positive patients lacking TEL exon 5 resemble canonical TEL/AML1 cases.
    Zaliova M, Meyer C, Cario G, Vaskova M, Marschalek R, Stary J, Zuna J, Trka J.
    Pediatr Blood Cancer; 2011 Feb 17; 56(2):217-25. PubMed ID: 21157892
    [Abstract] [Full Text] [Related]

  • 17. PRDX4, a member of the peroxiredoxin family, is fused to AML1 (RUNX1) in an acute myeloid leukemia patient with a t(X;21)(p22;q22).
    Zhang Y, Emmanuel N, Kamboj G, Chen J, Shurafa M, Van Dyke DL, Wiktor A, Rowley JD.
    Genes Chromosomes Cancer; 2004 Aug 17; 40(4):365-70. PubMed ID: 15188461
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