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PUBMED FOR HANDHELDS

Journal Abstract Search


249 related items for PubMed ID: 11378241

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  • 48. Aβ43 levels determine the onset of pathological amyloid deposition.
    Tambini MD, Yin T, Yesiltepe M, Breuillaud L, Zehntner SP, d'Abramo C, Giliberto L, D'Adamio L.
    J Biol Chem; 2023 Jul; 299(7):104868. PubMed ID: 37257821
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  • 52. Presenilin-1 280Glu-->Ala mutation alters C-terminal APP processing yielding longer abeta peptides: implications for Alzheimer's disease.
    Van Vickle GD, Esh CL, Kokjohn TA, Patton RL, Kalback WM, Luehrs DC, Beach TG, Newel AJ, Lopera F, Ghetti B, Vidal R, Castaño EM, Roher AE.
    Mol Med; 2008 Jul; 14(3-4):184-94. PubMed ID: 18317569
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  • 56. A pathogenic presenilin-1 deletion causes abberrant Abeta 42 production in the absence of congophilic amyloid plaques.
    Steiner H, Revesz T, Neumann M, Romig H, Grim MG, Pesold B, Kretzschmar HA, Hardy J, Holton JL, Baumeister R, Houlden H, Haass C.
    J Biol Chem; 2001 Mar 09; 276(10):7233-9. PubMed ID: 11084029
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  • 59. Molecular genetics of Alzheimer's disease.
    Goate AM.
    Geriatrics; 1997 Sep 09; 52 Suppl 2():S9-12. PubMed ID: 9307578
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  • 60. The effect of focal brain injury on beta-amyloid plaque deposition, inflammation and synapses in the APP/PS1 mouse model of Alzheimer's disease.
    Collins JM, King AE, Woodhouse A, Kirkcaldie MT, Vickers JC.
    Exp Neurol; 2015 May 09; 267():219-29. PubMed ID: 25747037
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