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Journal Abstract Search

452 related items for PubMed ID: 11425855

  • 1. Hsp90 ensures the transition from the early Ca2+-dependent to the late phosphorylation-dependent activation of the endothelial nitric-oxide synthase in vascular endothelial growth factor-exposed endothelial cells.
    Brouet A, Sonveaux P, Dessy C, Balligand JL, Feron O.
    J Biol Chem; 2001 Aug 31; 276(35):32663-9. PubMed ID: 11425855
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  • 2. Synergistic activation of endothelial nitric-oxide synthase (eNOS) by HSP90 and Akt: calcium-independent eNOS activation involves formation of an HSP90-Akt-CaM-bound eNOS complex.
    Takahashi S, Mendelsohn ME.
    J Biol Chem; 2003 Aug 15; 278(33):30821-7. PubMed ID: 12799359
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  • 3. Domain mapping studies reveal that the M domain of hsp90 serves as a molecular scaffold to regulate Akt-dependent phosphorylation of endothelial nitric oxide synthase and NO release.
    Fontana J, Fulton D, Chen Y, Fairchild TA, McCabe TJ, Fujita N, Tsuruo T, Sessa WC.
    Circ Res; 2002 May 03; 90(8):866-73. PubMed ID: 11988487
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  • 4. Hsp90 and caveolin are key targets for the proangiogenic nitric oxide-mediated effects of statins.
    Brouet A, Sonveaux P, Dessy C, Moniotte S, Balligand JL, Feron O.
    Circ Res; 2001 Nov 09; 89(10):866-73. PubMed ID: 11701613
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  • 5. Transactivation of vascular endothelial growth factor (VEGF) receptor Flk-1/KDR is involved in sphingosine 1-phosphate-stimulated phosphorylation of Akt and endothelial nitric-oxide synthase (eNOS).
    Tanimoto T, Jin ZG, Berk BC.
    J Biol Chem; 2002 Nov 08; 277(45):42997-3001. PubMed ID: 12226078
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  • 6. Immediate and delayed VEGF-mediated NO synthesis in endothelial cells: role of PI3K, PKC and PLC pathways.
    Gélinas DS, Bernatchez PN, Rollin S, Bazan NG, Sirois MG.
    Br J Pharmacol; 2002 Dec 08; 137(7):1021-30. PubMed ID: 12429574
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  • 7. Oxidized LDL inhibits vascular endothelial growth factor-induced endothelial cell migration by an inhibitory effect on the Akt/endothelial nitric oxide synthase pathway.
    Chavakis E, Dernbach E, Hermann C, Mondorf UF, Zeiher AM, Dimmeler S.
    Circulation; 2001 Apr 24; 103(16):2102-7. PubMed ID: 11319202
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  • 8. The Akt kinase signals directly to endothelial nitric oxide synthase.
    Michell BJ, Griffiths JE, Mitchelhill KI, Rodriguez-Crespo I, Tiganis T, Bozinovski S, de Montellano PR, Kemp BE, Pearson RB.
    Curr Biol; 2001 Apr 24; 9(15):845-8. PubMed ID: 10469573
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  • 9. Insulin-stimulated activation of eNOS is independent of Ca2+ but requires phosphorylation by Akt at Ser(1179).
    Montagnani M, Chen H, Barr VA, Quon MJ.
    J Biol Chem; 2001 Aug 10; 276(32):30392-8. PubMed ID: 11402048
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  • 11. TNF-alpha inhibits flow and insulin signaling leading to NO production in aortic endothelial cells.
    Kim F, Gallis B, Corson MA.
    Am J Physiol Cell Physiol; 2001 May 10; 280(5):C1057-65. PubMed ID: 11287317
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  • 12. VEGF increases permeability of the endothelial cell monolayer by activation of PKB/akt, endothelial nitric-oxide synthase, and MAP kinase pathways.
    Lal BK, Varma S, Pappas PJ, Hobson RW, Durán WN.
    Microvasc Res; 2001 Nov 10; 62(3):252-62. PubMed ID: 11678628
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  • 15. Phosphorylation of the endothelial nitric oxide synthase at ser-1177 is required for VEGF-induced endothelial cell migration.
    Dimmeler S, Dernbach E, Zeiher AM.
    FEBS Lett; 2000 Jul 21; 477(3):258-62. PubMed ID: 10908731
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  • 17. Dephosphorylation of endothelial nitric oxide synthase contributes to the anti-angiogenic effects of endostatin.
    Urbich C, Reissner A, Chavakis E, Dernbach E, Haendeler J, Fleming I, Zeiher AM, Kaszkin M, Dimmeler S.
    FASEB J; 2002 May 21; 16(7):706-8. PubMed ID: 11978735
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  • 20. Dephosphorylation of endothelial nitric-oxide synthase by vascular endothelial growth factor. Implications for the vascular responses to cyclosporin A.
    Kou R, Greif D, Michel T.
    J Biol Chem; 2002 Aug 16; 277(33):29669-73. PubMed ID: 12050171
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