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213 related items for PubMed ID: 11578614

  • 1. Aberrant induction of Par-4 is involved in apoptosis of hippocampal neurons in presenilin-1 M146V mutant knock-in mice.
    Xie J, Chang X, Zhang X, Guo Q.
    Brain Res; 2001 Oct 05; 915(1):1-10. PubMed ID: 11578614
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  • 2. Par-4 is a mediator of neuronal degeneration associated with the pathogenesis of Alzheimer disease.
    Guo Q, Fu W, Xie J, Luo H, Sells SF, Geddes JW, Bondada V, Rangnekar VM, Mattson MP.
    Nat Med; 1998 Aug 05; 4(8):957-62. PubMed ID: 9701251
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  • 3. Presenilin-1 mutations sensitize neurons to DNA damage-induced death by a mechanism involving perturbed calcium homeostasis and activation of calpains and caspase-12.
    Chan SL, Culmsee C, Haughey N, Klapper W, Mattson MP.
    Neurobiol Dis; 2002 Oct 05; 11(1):2-19. PubMed ID: 12460542
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  • 4. Presenilin-1 P264L knock-in mutation: differential effects on abeta production, amyloid deposition, and neuronal vulnerability.
    Siman R, Reaume AG, Savage MJ, Trusko S, Lin YG, Scott RW, Flood DG.
    J Neurosci; 2000 Dec 01; 20(23):8717-26. PubMed ID: 11102478
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  • 5. Neurons overexpressing mutant presenilin-1 are more sensitive to apoptosis induced by endoplasmic reticulum-Golgi stress.
    Terro F, Czech C, Esclaire F, Elyaman W, Yardin C, Baclet MC, Touchet N, Tremp G, Pradier L, Hugon J.
    J Neurosci Res; 2002 Aug 15; 69(4):530-9. PubMed ID: 12210846
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  • 6. Alzheimer's disease-like alterations in peripheral cells from presenilin-1 transgenic mice.
    Eckert A, Schindowski K, Leutner S, Luckhaus C, Touchet N, Czech C, Müller WE.
    Neurobiol Dis; 2001 Apr 15; 8(2):331-42. PubMed ID: 11300728
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  • 7. Increased vulnerability of hippocampal neurons from presenilin-1 mutant knock-in mice to amyloid beta-peptide toxicity: central roles of superoxide production and caspase activation.
    Guo Q, Sebastian L, Sopher BL, Miller MW, Ware CB, Martin GM, Mattson MP.
    J Neurochem; 1999 Mar 15; 72(3):1019-29. PubMed ID: 10037473
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  • 9. Perturbed endoplasmic reticulum function, synaptic apoptosis and the pathogenesis of Alzheimer's disease.
    Mattson MP, Gary DS, Chan SL, Duan W.
    Biochem Soc Symp; 2001 Mar 15; (67):151-62. PubMed ID: 11447832
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  • 11. Hippocampal spatial memory impairments caused by the familial Alzheimer's disease-linked presenilin 1 M146V mutation.
    Sun X, Beglopoulos V, Mattson MP, Shen J.
    Neurodegener Dis; 2005 Mar 15; 2(1):6-15. PubMed ID: 16908998
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  • 12. Prostate apoptosis response-4 production in synaptic compartments following apoptotic and excitotoxic insults: evidence for a pivotal role in mitochondrial dysfunction and neuronal degeneration.
    Duan W, Rangnekar VM, Mattson MP.
    J Neurochem; 1999 Jun 15; 72(6):2312-22. PubMed ID: 10349840
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  • 13. Alzheimer's disease-causing presenilin-1 mutations have deleterious effects on mitochondrial function.
    Han J, Park H, Maharana C, Gwon AR, Park J, Baek SH, Bae HG, Cho Y, Kim HK, Sul JH, Lee J, Kim E, Kim J, Cho Y, Park S, Palomera LF, Arumugam TV, Mattson MP, Jo DG.
    Theranostics; 2021 Jun 15; 11(18):8855-8873. PubMed ID: 34522215
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  • 16. Neurotrophic factors [activity-dependent neurotrophic factor (ADNF) and basic fibroblast growth factor (bFGF)] interrupt excitotoxic neurodegenerative cascades promoted by a PS1 mutation.
    Guo Q, Sebastian L, Sopher BL, Miller MW, Glazner GW, Ware CB, Martin GM, Mattson MP.
    Proc Natl Acad Sci U S A; 1999 Mar 30; 96(7):4125-30. PubMed ID: 10097174
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  • 17. Cell cycle-driven neuronal apoptosis specifically linked to amyloid peptide Abeta1-42 exposure is not exacerbated in a mouse model of presenilin-1 familial Alzheimer's disease.
    Malik B, Currais A, Soriano S.
    J Neurochem; 2008 Jul 30; 106(2):912-6. PubMed ID: 18466334
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  • 18. Functional phenotype in transgenic mice expressing mutant human presenilin-1.
    Barrow PA, Empson RM, Gladwell SJ, Anderson CM, Killick R, Yu X, Jefferys JG, Duff K.
    Neurobiol Dis; 2000 Apr 30; 7(2):119-26. PubMed ID: 10783295
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  • 19. Increased vulnerability of hippocampal neurons to excitotoxic necrosis in presenilin-1 mutant knock-in mice.
    Guo Q, Fu W, Sopher BL, Miller MW, Ware CB, Martin GM, Mattson MP.
    Nat Med; 1999 Jan 30; 5(1):101-6. PubMed ID: 9883847
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  • 20. Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid beta-peptide (1-42), HO and kainic acid: implications for Alzheimer's disease.
    Mohmmad Abdul H, Sultana R, Keller JN, St Clair DK, Markesbery WR, Butterfield DA.
    J Neurochem; 2006 Mar 30; 96(5):1322-35. PubMed ID: 16478525
    [Abstract] [Full Text] [Related]

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