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200 related items for PubMed ID: 11714811

  • 1. The carboxyl terminus of the granulocyte colony-stimulating factor receptor, truncated in patients with severe congenital neutropenia/acute myeloid leukemia, is required for SH2-containing phosphatase-1 suppression of Stat activation.
    Dong F, Qiu Y, Yi T, Touw IP, Larner AC.
    J Immunol; 2001 Dec 01; 167(11):6447-52. PubMed ID: 11714811
    [Abstract] [Full Text] [Related]

  • 2. Defective internalization and sustained activation of truncated granulocyte colony-stimulating factor receptor found in severe congenital neutropenia/acute myeloid leukemia.
    Ward AC, van Aesch YM, Schelen AM, Touw IP.
    Blood; 1999 Jan 15; 93(2):447-58. PubMed ID: 9885206
    [Abstract] [Full Text] [Related]

  • 3. Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene.
    Hermans MH, Antonissen C, Ward AC, Mayen AE, Ploemacher RE, Touw IP.
    J Exp Med; 1999 Feb 15; 189(4):683-92. PubMed ID: 9989983
    [Abstract] [Full Text] [Related]

  • 4. G-CSF receptor truncations found in SCN/AML relieve SOCS3-controlled inhibition of STAT5 but leave suppression of STAT3 intact.
    van de Geijn GJ, Gits J, Aarts LH, Heijmans-Antonissen C, Touw IP.
    Blood; 2004 Aug 01; 104(3):667-74. PubMed ID: 15069015
    [Abstract] [Full Text] [Related]

  • 5. The SH2 domain-containing protein tyrosine phosphatase SHP-1 is induced by granulocyte colony-stimulating factor (G-CSF) and modulates signaling from the G-CSF receptor.
    Ward AC, Oomen SP, Smith L, Gits J, van Leeuwen D, Soede-Bobok AA, Erpelinck-Verschueren CA, Yi T, Touw IP.
    Leukemia; 2000 Jul 01; 14(7):1284-91. PubMed ID: 10914554
    [Abstract] [Full Text] [Related]

  • 6. Loss of SHIP and CIS recruitment to the granulocyte colony-stimulating factor receptor contribute to hyperproliferative responses in severe congenital neutropenia/acute myelogenous leukemia.
    Hunter MG, Jacob A, O'donnell LC, Agler A, Druhan LJ, Coggeshall KM, Avalos BR.
    J Immunol; 2004 Oct 15; 173(8):5036-45. PubMed ID: 15470047
    [Abstract] [Full Text] [Related]

  • 7. The G-CSF receptor carboxyl terminus, truncated in AML/SCN, is required for induction of a Stat5 protease activity.
    Qiu Y, Zhuang D, MacRae A, Dong F.
    Leuk Res; 2005 Oct 15; 29(10):1153-62. PubMed ID: 16111533
    [Abstract] [Full Text] [Related]

  • 8. Expression of granulocyte colony-stimulating factor- and granulocyte-macrophage colony-stimulating factor-associated signal transduction proteins of the JAK/STAT pathway in normal granulopoiesis and in blast cells of acute myelogenous leukemia.
    Biethahn S, Alves F, Wilde S, Hiddemann W, Spiekermann K.
    Exp Hematol; 1999 May 15; 27(5):885-94. PubMed ID: 10340405
    [Abstract] [Full Text] [Related]

  • 9. Mutations in the gene for the granulocyte colony-stimulating-factor receptor in patients with acute myeloid leukemia preceded by severe congenital neutropenia.
    Dong F, Brynes RK, Tidow N, Welte K, Löwenberg B, Touw IP.
    N Engl J Med; 1995 Aug 24; 333(8):487-93. PubMed ID: 7542747
    [Abstract] [Full Text] [Related]

  • 10. Tyrosine-dependent and -independent mechanisms of STAT3 activation by the human granulocyte colony-stimulating factor (G-CSF) receptor are differentially utilized depending on G-CSF concentration.
    Ward AC, Hermans MH, Smith L, van Aesch YM, Schelen AM, Antonissen C, Touw IP.
    Blood; 1999 Jan 01; 93(1):113-24. PubMed ID: 9864153
    [Abstract] [Full Text] [Related]

  • 11. G-CSF receptor mutations in patients with severe congenital neutropenia do not abrogate Jak2 activation and stat1/stat3 translocation.
    Herbst A, Koester M, Wirth D, Hauser H, Welte K.
    Ann N Y Acad Sci; 1999 Apr 30; 872():320-5; discussion 325-7. PubMed ID: 10372134
    [Abstract] [Full Text] [Related]

  • 12. Stimulation of Stat5 by granulocyte colony-stimulating factor (G-CSF) is modulated by two distinct cytoplasmic regions of the G-CSF receptor.
    Dong F, Liu X, de Koning JP, Touw IP, Hennighausen L, Larner A, Grimley PM.
    J Immunol; 1998 Dec 15; 161(12):6503-9. PubMed ID: 9862674
    [Abstract] [Full Text] [Related]

  • 13. Activation of Akt kinase by granulocyte colony-stimulating factor (G-CSF): evidence for the role of a tyrosine kinase activity distinct from the Janus kinases.
    Dong F, Larner AC.
    Blood; 2000 Mar 01; 95(5):1656-62. PubMed ID: 10688821
    [Abstract] [Full Text] [Related]

  • 14. Granulocyte colony-stimulating factor receptor mutations in severe congenital neutropenia transforming to acute myelogenous leukemia confer resistance to apoptosis and enhance cell survival.
    Hunter MG, Avalos BR.
    Blood; 2000 Mar 15; 95(6):2132-7. PubMed ID: 10706885
    [Abstract] [Full Text] [Related]

  • 15. Novel point mutation in the extracellular domain of the granulocyte colony-stimulating factor (G-CSF) receptor in a case of severe congenital neutropenia hyporesponsive to G-CSF treatment.
    Ward AC, van Aesch YM, Gits J, Schelen AM, de Koning JP, van Leeuwen D, Freedman MH, Touw IP.
    J Exp Med; 1999 Aug 16; 190(4):497-507. PubMed ID: 10449521
    [Abstract] [Full Text] [Related]

  • 16. Mutations in the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia.
    Dong F, Dale DC, Bonilla MA, Freedman M, Fasth A, Neijens HJ, Palmblad J, Briars GL, Carlsson G, Veerman AJ, Welte K, Löwenberg B, Touw IP.
    Leukemia; 1997 Jan 16; 11(1):120-5. PubMed ID: 9001427
    [Abstract] [Full Text] [Related]

  • 17. A novel mutation in the juxtamembrane intracellular sequence of the granulocyte colony-stimulating factor (G-CSF) receptor gene in a patient with severe congenital neutropenia augments GCSF proliferation activity but not through the MAP kinase cascade.
    Yokoyama T, Okamura S, Asano Y, Kamezaki K, Numata A, Kakumitsu H, Shide K, Nakashima H, Taisuke K, Sekine Y, Mizuno Y, Okamura J, Matsuda T, Harada M, Yoshiyuki N, Shimoda K.
    Int J Hematol; 2005 Jul 16; 82(1):28-34. PubMed ID: 16229088
    [Abstract] [Full Text] [Related]

  • 18. Involvement of STAT3 in the granulocyte colony-stimulating factor-induced differentiation of myeloid cells.
    Shimozaki K, Nakajima K, Hirano T, Nagata S.
    J Biol Chem; 1997 Oct 03; 272(40):25184-9. PubMed ID: 9312131
    [Abstract] [Full Text] [Related]

  • 19. Increased CCAAT enhancer-binding protein epsilon (C/EBPepsilon) expression and premature apoptosis in myeloid cells expressing Gfi-1 N382S mutant associated with severe congenital neutropenia.
    Zhuang D, Qiu Y, Kogan SC, Dong F.
    J Biol Chem; 2006 Apr 21; 281(16):10745-51. PubMed ID: 16500901
    [Abstract] [Full Text] [Related]

  • 20. Severe congenital neutropenia terminating in acute myeloid leukemia: disease progression associated with mutations in the granulocyte-colony stimulating factor receptor gene.
    Touw IP, Dong F.
    Leuk Res; 1996 Aug 21; 20(8):629-31. PubMed ID: 8913314
    [Abstract] [Full Text] [Related]

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