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Journal Abstract Search

270 related items for PubMed ID: 11743991

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  • 2. Brevetoxin-induced autocrine excitotoxicity is associated with manifold routes of Ca2+ influx.
    Berman FW, Murray TF.
    J Neurochem; 2000 Apr; 74(4):1443-51. PubMed ID: 10737600
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  • 3. Domoic acid neurotoxicity in cultured cerebellar granule neurons is mediated predominantly by NMDA receptors that are activated as a consequence of excitatory amino acid release.
    Berman FW, Murray TF.
    J Neurochem; 1997 Aug; 69(2):693-703. PubMed ID: 9231729
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  • 6. Calcium influx constitutes the ionic basis for the maintenance of glutamate-induced extended neuronal depolarization associated with hippocampal neuronal death.
    Limbrick DD, Sombati S, DeLorenzo RJ.
    Cell Calcium; 2003 Feb; 33(2):69-81. PubMed ID: 12531183
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  • 8. Instrumental role of Na+ in NMDA excitotoxicity in glucose-deprived and depolarized cerebellar granule cells.
    Czyz A, Baranauskas G, Kiedrowski L.
    J Neurochem; 2002 Apr; 81(2):379-89. PubMed ID: 12064485
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  • 11. Disruption of endoplasmic reticulum calcium stores is involved in neuronal death induced by glycolysis inhibition in cultured hippocampal neurons.
    Hernández-Fonseca K, Massieu L.
    J Neurosci Res; 2005 Oct 15; 82(2):196-205. PubMed ID: 16175570
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  • 12. Ca(2+)- and Cl(-)-dependent, NMDA receptor-mediated neuronal death induced by depolarization in rat hippocampal organotypic cultures.
    Takahashi M, Liou SY, Kunihara M.
    Brain Res; 1995 Mar 27; 675(1-2):249-56. PubMed ID: 7796136
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  • 13. Apoptosis induced by domoic acid in mouse cerebellar granule neurons involves activation of p38 and JNK MAP kinases.
    Giordano G, Klintworth HM, Kavanagh TJ, Costa LG.
    Neurochem Int; 2008 May 27; 52(6):1100-5. PubMed ID: 18164102
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  • 14. Na+/K+-ATPase inhibitor palytoxin enhances vulnerability of cultured cerebellar neurons to domoic acid via sodium-dependent mechanisms.
    Pérez-Gómez A, Novelli A, Fernández-Sánchez MT.
    J Neurochem; 2010 Jul 27; 114(1):28-38. PubMed ID: 20089129
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  • 15. Failure of glycine site NMDA receptor antagonists to protect against L-2-chloropropionic acid-induced neurotoxicity highlights the uniqueness of cerebellar NMDA receptors.
    Widdowson PS, Gyte AJ, Upton R, Wyatt I.
    Brain Res; 1996 Nov 04; 738(2):236-42. PubMed ID: 8955518
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