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Journal Abstract Search

232 related items for PubMed ID: 11748861

  • 1. Role of nitric oxide in the pathogenesis of muscular dystrophies: a "two hit" hypothesis of the cause of muscle necrosis.
    Rando TA.
    Microsc Res Tech; 2001 Nov 15; 55(4):223-35. PubMed ID: 11748861
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  • 3. Inhibition of dystroglycan binding to laminin disrupts the PI3K/AKT pathway and survival signaling in muscle cells.
    Langenbach KJ, Rando TA.
    Muscle Nerve; 2002 Nov 15; 26(5):644-53. PubMed ID: 12402286
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  • 5. Role for alpha-dystrobrevin in the pathogenesis of dystrophin-dependent muscular dystrophies.
    Grady RM, Grange RW, Lau KS, Maimone MM, Nichol MC, Stull JT, Sanes JR.
    Nat Cell Biol; 1999 Aug 15; 1(4):215-20. PubMed ID: 10559919
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  • 6. Vasomodulation by skeletal muscle-derived nitric oxide requires alpha-syntrophin-mediated sarcolemmal localization of neuronal Nitric oxide synthase.
    Thomas GD, Shaul PW, Yuhanna IS, Froehner SC, Adams ME.
    Circ Res; 2003 Mar 21; 92(5):554-60. PubMed ID: 12600881
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  • 7. Nitric oxide synthase is up-regulated in muscle fibers in muscular dystrophy.
    Punkt K, Schering S, Löffler S, Minin EA, Samoilova VE, Hasselblatt M, Paulus W, Müller-Werdan U, Demus U, Koehler G, Boecker W, Buchwalow IB.
    Biochem Biophys Res Commun; 2006 Sep 15; 348(1):259-64. PubMed ID: 16876119
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  • 8. CAPON expression in skeletal muscle is regulated by position, repair, NOS activity, and dystrophy.
    Ségalat L, Grisoni K, Archer J, Vargas C, Bertrand A, Anderson JE.
    Exp Cell Res; 2005 Jan 15; 302(2):170-9. PubMed ID: 15561099
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  • 9. Nitric oxide synthase I (NOS-I) is deficient in the sarcolemma of striated muscle fibers in patients with Duchenne muscular dystrophy, suggesting an association with dystrophin.
    Grozdanovic Z, Gosztonyi G, Gossrau R.
    Acta Histochem; 1996 Jan 15; 98(1):61-9. PubMed ID: 9054190
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  • 11. The susceptibility of muscle cells to oxidative stress is independent of nitric oxide synthase expression.
    Zhuang W, Eby JC, Cheong M, Mohapatra PK, Bredt DS, Disatnik MH, Rando TA.
    Muscle Nerve; 2001 Apr 15; 24(4):502-11. PubMed ID: 11268022
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  • 12. Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) regulates endothelial nitric oxide synthase (eNOS) activity and its localization within the human vein endothelial cells (HUVEC) in culture.
    Di Pietro R, Mariggiò MA, Guarnieri S, Sancilio S, Giardinelli A, Di Silvestre S, Consoli A, Zauli G, Pandolfi A.
    J Cell Biochem; 2006 Mar 01; 97(4):782-94. PubMed ID: 16229016
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  • 13. The dystrophin-glycoprotein complex, cellular signaling, and the regulation of cell survival in the muscular dystrophies.
    Rando TA.
    Muscle Nerve; 2001 Dec 01; 24(12):1575-94. PubMed ID: 11745966
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  • 14. Expression of a NOS transgene in dystrophin-deficient muscle reduces muscle membrane damage without increasing the expression of membrane-associated cytoskeletal proteins.
    Tidball JG, Wehling-Henricks M.
    Mol Genet Metab; 2004 Aug 01; 82(4):312-20. PubMed ID: 15308129
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  • 15. [Association between dystrophin and neuronal nitric oxide synthase in muscles of progressive muscular dystrophy].
    Wang S, Shen D.
    Zhonghua Yi Xue Za Zhi; 2002 Feb 10; 82(3):155-7. PubMed ID: 11953148
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  • 19. Protective effects of Ca2+ handling drugs against abnormal Ca2+ homeostasis and cell damage in myopathic skeletal muscle cells.
    Iwata Y, Katanosaka Y, Shijun Z, Kobayashi Y, Hanada H, Shigekawa M, Wakabayashi S.
    Biochem Pharmacol; 2005 Sep 01; 70(5):740-51. PubMed ID: 16009351
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  • 20. A caveolin-3 mutant that causes limb girdle muscular dystrophy type 1C disrupts Src localization and activity and induces apoptosis in skeletal myotubes.
    Smythe GM, Eby JC, Disatnik MH, Rando TA.
    J Cell Sci; 2003 Dec 01; 116(Pt 23):4739-49. PubMed ID: 14600260
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