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995 related items for PubMed ID: 11749118

  • 1. Tissue injury following inhalation of fine particulate matter and hydrogen peroxide is associated with altered production of inflammatory mediators and antioxidants by alveolar macrophages.
    Morio LA, Hooper KA, Brittingham J, Li TH, Gordon RE, Turpin BJ, Laskin DL.
    Toxicol Appl Pharmacol; 2001 Dec 15; 177(3):188-99. PubMed ID: 11749118
    [Abstract] [Full Text] [Related]

  • 2. Peroxides and macrophages in the toxicity of fine particulate matter in rats.
    Laskin DL, Morio L, Hooper K, Li TH, Buckley B, Turpin B.
    Res Rep Health Eff Inst; 2003 Dec 15; (117):1-51; discussion 53-63. PubMed ID: 15035594
    [Abstract] [Full Text] [Related]

  • 3. Inhalation of particulate lead oxide disrupts pulmonary macrophage-mediated functions important for host defense and tumor surveillance in the lung.
    Zelikoff JT, Parsons E, Schlesinger RB.
    Environ Res; 1993 Aug 15; 62(2):207-22. PubMed ID: 8393781
    [Abstract] [Full Text] [Related]

  • 4. Enhanced production of nitric oxide by rat alveolar macrophages after inhalation of a pulmonary irritant is associated with increased expression of nitric oxide synthase.
    Pendino KJ, Laskin JD, Shuler RL, Punjabi CJ, Laskin DL.
    J Immunol; 1993 Dec 15; 151(12):7196-205. PubMed ID: 7505023
    [Abstract] [Full Text] [Related]

  • 5. NTP Toxicology and Carcinogenesis Studies of Talc (CAS No. 14807-96-6)(Non-Asbestiform) in F344/N Rats and B6C3F1 Mice (Inhalation Studies).
    National Toxicology Program .
    Natl Toxicol Program Tech Rep Ser; 1993 Sep 15; 421():1-287. PubMed ID: 12616290
    [Abstract] [Full Text] [Related]

  • 6. Inhalation of toluene diisocyanate is associated with increased production of nitric oxide by rat bronchoalveolar lavage cells.
    Huffman LJ, Judy DJ, Frazer D, Shapiro RE, Castranova V, Billie M, Dedhia HV.
    Toxicol Appl Pharmacol; 1997 Jul 15; 145(1):61-7. PubMed ID: 9221824
    [Abstract] [Full Text] [Related]

  • 7. Assessing toxicity of fine and nanoparticles: comparing in vitro measurements to in vivo pulmonary toxicity profiles.
    Sayes CM, Reed KL, Warheit DB.
    Toxicol Sci; 2007 May 15; 97(1):163-80. PubMed ID: 17301066
    [Abstract] [Full Text] [Related]

  • 8. Response of alveolar macrophages from inducible nitric oxide synthase knockout or wild-type mice to an in vitro lipopolysaccharide or silica exposure.
    Zeidler PC, Roberts JR, Castranova V, Chen F, Butterworth L, Andrew ME, Robinson VA, Porter DW.
    J Toxicol Environ Health A; 2003 Jun 13; 66(11):995-1013. PubMed ID: 12775513
    [Abstract] [Full Text] [Related]

  • 9. Pulmonary particulate matter and systemic microvascular dysfunction.
    Nurkiewicz TR, Porter DW, Hubbs AF, Stone S, Moseley AM, Cumpston JL, Goodwill AG, Frisbee SJ, Perrotta PL, Brock RW, Frisbee JC, Boegehold MA, Frazer DG, Chen BT, Castranova V, HEI Health Review Committee.
    Res Rep Health Eff Inst; 2011 Dec 13; (164):3-48. PubMed ID: 22329339
    [Abstract] [Full Text] [Related]

  • 10. Effects of vanadium upon polyl:C-induced responses in rat lung and alveolar macrophages.
    Cohen MD, Becker S, Devlin R, Schlesinger RB, Zelikoff JT.
    J Toxicol Environ Health; 1997 Aug 29; 51(6):591-608. PubMed ID: 9242230
    [Abstract] [Full Text] [Related]

  • 11. Immunotoxicologic effects of inhaled chromium: role of particle solubility and co-exposure to ozone.
    Cohen MD, Zelikoff JT, Chen LC, Schlesinger RB.
    Toxicol Appl Pharmacol; 1998 Sep 29; 152(1):30-40. PubMed ID: 9772197
    [Abstract] [Full Text] [Related]

  • 12. Nitric oxide and reactive oxygen species production causes progressive damage in rats after cessation of silica inhalation.
    Porter DW, Millecchia LL, Willard P, Robinson VA, Ramsey D, McLaurin J, Khan A, Brumbaugh K, Beighley CM, Teass A, Castranova V.
    Toxicol Sci; 2006 Mar 29; 90(1):188-97. PubMed ID: 16339787
    [Abstract] [Full Text] [Related]

  • 13. The spontaneously hypertensive rat as a model of human cardiovascular disease: evidence of exacerbated cardiopulmonary injury and oxidative stress from inhaled emission particulate matter.
    Kodavanti UP, Schladweiler MC, Ledbetter AD, Watkinson WP, Campen MJ, Winsett DW, Richards JR, Crissman KM, Hatch GE, Costa DL.
    Toxicol Appl Pharmacol; 2000 May 01; 164(3):250-63. PubMed ID: 10799335
    [Abstract] [Full Text] [Related]

  • 14. Inhibition of macrophages with gadolinium chloride abrogates ozone-induced pulmonary injury and inflammatory mediator production.
    Pendino KJ, Meidhof TM, Heck DE, Laskin JD, Laskin DL.
    Am J Respir Cell Mol Biol; 1995 Aug 01; 13(2):125-32. PubMed ID: 7542894
    [Abstract] [Full Text] [Related]

  • 15. Deficiency in inducible nitric oxide synthase protects mice from ozone-induced lung inflammation and tissue injury.
    Fakhrzadeh L, Laskin JD, Laskin DL.
    Am J Respir Cell Mol Biol; 2002 Apr 01; 26(4):413-9. PubMed ID: 11919077
    [Abstract] [Full Text] [Related]

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  • 17. Toxicology and carcinogenesis studies of indium phosphide (CAS No. 22398-90-7) in F344/N rats and B6C3F1 mice (inhalation studies).
    National Toxicology Program.
    Natl Toxicol Program Tech Rep Ser; 2001 Jul 01; (499):7-340. PubMed ID: 12087422
    [Abstract] [Full Text] [Related]

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  • 19. Inhibition of ozone-induced nitric oxide synthase expression in the lung by endotoxin.
    Pendino KJ, Gardner CR, Shuler RL, Laskin JD, Durham SK, Barton DS, Ohnishi ST, Ohnishi T, Laskin DL.
    Am J Respir Cell Mol Biol; 1996 Jun 01; 14(6):516-25. PubMed ID: 8652180
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