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Journal Abstract Search

109 related items for PubMed ID: 11786529

  • 1. Expression and intracellular localization of an SCN5A double mutant R1232W/T1620M implicated in Brugada syndrome.
    Baroudi G, Acharfi S, Larouche C, Chahine M.
    Circ Res; 2002 Jan 11; 90(1):E11-6. PubMed ID: 11786529
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  • 3. A novel mutation in the SCN5A gene is associated with Brugada syndrome.
    Shin DJ, Kim E, Park SB, Jang WC, Bae Y, Han J, Jang Y, Joung B, Lee MH, Kim SS, Huang H, Chahine M, Yoon SK.
    Life Sci; 2007 Jan 30; 80(8):716-24. PubMed ID: 17141278
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  • 6. A novel SCN5A mutation, F1344S, identified in a patient with Brugada syndrome and fever-induced ventricular fibrillation.
    Keller DI, Huang H, Zhao J, Frank R, Suarez V, Delacrétaz E, Brink M, Osswald S, Schwick N, Chahine M.
    Cardiovasc Res; 2006 Jun 01; 70(3):521-9. PubMed ID: 16616735
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  • 7. Clinical and electrophysiological characteristics of Brugada syndrome caused by a missense mutation in the S5-pore site of SCN5A.
    Itoh H, Shimizu M, Mabuchi H, Imoto K.
    J Cardiovasc Electrophysiol; 2005 Apr 01; 16(4):378-83. PubMed ID: 15828879
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  • 8. Electrocardiographic characteristics and SCN5A mutations in idiopathic ventricular fibrillation associated with early repolarization.
    Watanabe H, Nogami A, Ohkubo K, Kawata H, Hayashi Y, Ishikawa T, Makiyama T, Nagao S, Yagihara N, Takehara N, Kawamura Y, Sato A, Okamura K, Hosaka Y, Sato M, Fukae S, Chinushi M, Oda H, Okabe M, Kimura A, Maemura K, Watanabe I, Kamakura S, Horie M, Aizawa Y, Shimizu W, Makita N.
    Circ Arrhythm Electrophysiol; 2011 Dec 01; 4(6):874-81. PubMed ID: 22028457
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  • 9. Novel Brugada syndrome-causing mutation in ion-conducting pore of cardiac Na+ channel does not affect ion selectivity properties.
    Amin AS, Verkerk AO, Bhuiyan ZA, Wilde AA, Tan HL.
    Acta Physiol Scand; 2005 Dec 01; 185(4):291-301. PubMed ID: 16266370
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  • 11. Enhanced Na(+) channel intermediate inactivation in Brugada syndrome.
    Wang DW, Makita N, Kitabatake A, Balser JR, George AL.
    Circ Res; 2000 Oct 13; 87(8):E37-43. PubMed ID: 11029409
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  • 13. A novel mutation L619F in the cardiac Na+ channel SCN5A associated with long-QT syndrome (LQT3): a role for the I-II linker in inactivation gating.
    Wehrens XH, Rossenbacker T, Jongbloed RJ, Gewillig M, Heidbüchel H, Doevendans PA, Vos MA, Wellens HJ, Kass RS.
    Hum Mutat; 2003 May 13; 21(5):552. PubMed ID: 12673799
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  • 15. A novel missense mutation in the SCN5A gene associated with Brugada syndrome bidirectionally affecting blocking actions of antiarrhythmic drugs.
    Itoh H, Shimizu M, Takata S, Mabuchi H, Imoto K.
    J Cardiovasc Electrophysiol; 2005 May 13; 16(5):486-93. PubMed ID: 15877619
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  • 17. A mutation in the beta 3 subunit of the cardiac sodium channel associated with Brugada ECG phenotype.
    Hu D, Barajas-Martinez H, Burashnikov E, Springer M, Wu Y, Varro A, Pfeiffer R, Koopmann TT, Cordeiro JM, Guerchicoff A, Pollevick GD, Antzelevitch C.
    Circ Cardiovasc Genet; 2009 Jun 13; 2(3):270-8. PubMed ID: 20031595
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  • 19. Long-QT syndrome-related sodium channel mutations probed by the dynamic action potential clamp technique.
    Berecki G, Zegers JG, Bhuiyan ZA, Verkerk AO, Wilders R, van Ginneken AC.
    J Physiol; 2006 Jan 15; 570(Pt 2):237-50. PubMed ID: 16254012
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