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Journal Abstract Search

109 related items for PubMed ID: 11796206

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  • 2. A familial amyotrophic lateral sclerosis-associated A4V Cu, Zn-superoxide dismutase mutant has a lower Km for hydrogen peroxide. Correlation between clinical severity and the Km value.
    Yim HS, Kang JH, Chock PB, Stadtman ER, Yim MB.
    J Biol Chem; 1997 Apr 04; 272(14):8861-3. PubMed ID: 9083002
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  • 5. Mutations associated with amyotrophic lateral sclerosis convert superoxide dismutase from an antiapoptotic gene to a proapoptotic gene: studies in yeast and neural cells.
    Rabizadeh S, Gralla EB, Borchelt DR, Gwinn R, Valentine JS, Sisodia S, Wong P, Lee M, Hahn H, Bredesen DE.
    Proc Natl Acad Sci U S A; 1995 Mar 28; 92(7):3024-8. PubMed ID: 7708768
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  • 8. Cell death mechanisms in ALS.
    Bredesen DE, Wiedau-Pazos M, Goto JJ, Rabizadeh S, Roe JA, Gralla EB, Ellerby LM, Valentine JS.
    Neurology; 1996 Oct 28; 47(4 Suppl 2):S36-8; discussion S38-9. PubMed ID: 8858049
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  • 9. Release of copper ions from the familial amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutants.
    Eum WS, Kang JH.
    Mol Cells; 1999 Feb 28; 9(1):110-4. PubMed ID: 10102580
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  • 10. Reduced reactivation rate in mutant CuZnSOD and progression rate of amyotrophic lateral sclerosis.
    Völkel H, Selzle M, Walk T, Jung G, Link J, Ludolph AC, Reuter A.
    Eur J Neurol; 2004 Jun 28; 11(6):397-404. PubMed ID: 15171736
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  • 11. Implications of fALS Mutations on Sod1 Function and Oligomerization in Cell Models.
    Brasil AA, Magalhães RSS, De Carvalho MDC, Paiva I, Gerhardt E, Pereira MD, Outeiro TF, Eleutherio ECA.
    Mol Neurobiol; 2018 Jun 28; 55(6):5269-5281. PubMed ID: 28884318
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  • 12. The perplexing role of copper-zinc superoxide dismutase in amyotrophic lateral sclerosis (Lou Gehrig's disease).
    Potter SZ, Valentine JS.
    J Biol Inorg Chem; 2003 Apr 28; 8(4):373-80. PubMed ID: 12644909
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  • 14. A gain-of-function of an amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutant: An enhancement of free radical formation due to a decrease in Km for hydrogen peroxide.
    Yim MB, Kang JH, Yim HS, Kwak HS, Chock PB, Stadtman ER.
    Proc Natl Acad Sci U S A; 1996 Jun 11; 93(12):5709-14. PubMed ID: 8650157
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  • 15. Loss of oxidation-reduction specificity in amyotrophic lateral sclerosis-associated CuZnSOD mutants.
    Cafe C, Testa MP, Sheldon PJ, French WP, Ellerby LM, Bredesen DE.
    J Mol Neurosci; 2000 Oct 11; 15(2):71-83. PubMed ID: 11220787
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  • 16. Reexamination of the mechanism of hydroxyl radical adducts formed from the reaction between familial amyotrophic lateral sclerosis-associated Cu,Zn superoxide dismutase mutants and H2O2.
    Singh RJ, Karoui H, Gunther MR, Beckman JS, Mason RP, Kalyanaraman B.
    Proc Natl Acad Sci U S A; 1998 Jun 09; 95(12):6675-80. PubMed ID: 9618471
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  • 19. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 09; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 20. Copper(2+) binding to the surface residue cysteine 111 of His46Arg human copper-zinc superoxide dismutase, a familial amyotrophic lateral sclerosis mutant.
    Liu H, Zhu H, Eggers DK, Nersissian AM, Faull KF, Goto JJ, Ai J, Sanders-Loehr J, Gralla EB, Valentine JS.
    Biochemistry; 2000 Jul 18; 39(28):8125-32. PubMed ID: 10889018
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