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PUBMED FOR HANDHELDS

Journal Abstract Search


318 related items for PubMed ID: 11801688

  • 1. CXCR2 is necessary for the development and persistence of chronic fungal asthma in mice.
    Schuh JM, Blease K, Hogaboam CM.
    J Immunol; 2002 Feb 01; 168(3):1447-56. PubMed ID: 11801688
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  • 2. Airway remodeling is absent in CCR1-/- mice during chronic fungal allergic airway disease.
    Blease K, Mehrad B, Standiford TJ, Lukacs NW, Kunkel SL, Chensue SW, Lu B, Gerard CJ, Hogaboam CM.
    J Immunol; 2000 Aug 01; 165(3):1564-72. PubMed ID: 10903765
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  • 3. Enhanced pulmonary allergic responses to Aspergillus in CCR2-/- mice.
    Blease K, Mehrad B, Standiford TJ, Lukacs NW, Gosling J, Boring L, Charo IF, Kunkel SL, Hogaboam CM.
    J Immunol; 2000 Sep 01; 165(5):2603-11. PubMed ID: 10946288
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  • 4. Stat6-deficient mice develop airway hyperresponsiveness and peribronchial fibrosis during chronic fungal asthma.
    Blease K, Schuh JM, Jakubzick C, Lukacs NW, Kunkel SL, Joshi BH, Puri RK, Kaplan MH, Hogaboam CM.
    Am J Pathol; 2002 Feb 01; 160(2):481-90. PubMed ID: 11839568
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  • 5. Chronic airway hyperreactivity, goblet cell hyperplasia, and peribronchial fibrosis during allergic airway disease induced by Aspergillus fumigatus.
    Hogaboam CM, Blease K, Mehrad B, Steinhauser ML, Standiford TJ, Kunkel SL, Lukacs NW.
    Am J Pathol; 2000 Feb 01; 156(2):723-32. PubMed ID: 10666400
    [Abstract] [Full Text] [Related]

  • 6. Airway hyperresponsiveness, but not airway remodeling, is attenuated during chronic pulmonary allergic responses to Aspergillus in CCR4-/- mice.
    Schuh JM, Power C, Proudfoot AE, Kunkel SL, Lukacs NW, Hogaboam CM.
    FASEB J; 2002 Aug 01; 16(10):1313-5. PubMed ID: 12154006
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  • 14. Surfactant protein D inhibits early airway response in Aspergillus fumigatus-sensitized mice.
    Erpenbeck VJ, Ziegert M, Cavalet-Blanco D, Martin C, Baelder R, Glaab T, Braun A, Steinhilber W, Luettig B, Uhlig S, Hoymann HG, Krug N, Hohlfeld JM.
    Clin Exp Allergy; 2006 Jul 01; 36(7):930-40. PubMed ID: 16839409
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  • 16. IL-17A promotes the exacerbation of IL-33-induced airway hyperresponsiveness by enhancing neutrophilic inflammation via CXCR2 signaling in mice.
    Mizutani N, Nabe T, Yoshino S.
    J Immunol; 2014 Feb 15; 192(4):1372-84. PubMed ID: 24446518
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  • 18. Absence of the complement anaphylatoxin C3a receptor suppresses Th2 effector functions in a murine model of pulmonary allergy.
    Drouin SM, Corry DB, Hollman TJ, Kildsgaard J, Wetsel RA.
    J Immunol; 2002 Nov 15; 169(10):5926-33. PubMed ID: 12421977
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