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197 related items for PubMed ID: 11862715
1. [Role of the TCF phosphorylation state and the chromatin structure in the negative transcription regulation of the c-fos proto-oncogene in E1A + c-Ha-ras transformed cells]. Abramova MV, Kukushkin AN, Pospelova TV, Svetlikova SB, Pospelov VA. Mol Biol (Mosk); 2002; 36(1):66-75. PubMed ID: 11862715 [Abstract] [Full Text] [Related]
2. Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter. Kukushkin AN, Abramova MV, Svetlikova SB, Darieva ZA, Pospelova TV, Pospelov VA. Oncogene; 2002 Jan 24; 21(5):719-30. PubMed ID: 11850800 [Abstract] [Full Text] [Related]
3. Transient expression of E1A and Ras oncogenes causes downregulation of c-fos gene transcription in nontransformed REF52 cells. Usenko T, Kukushkin A, Pospelova T, Pospelov V. Oncogene; 2003 Oct 23; 22(48):7661-6. PubMed ID: 14576829 [Abstract] [Full Text] [Related]
4. FLI1 and EWS-FLI1 function as ternary complex factors and ELK1 and SAP1a function as ternary and quaternary complex factors on the Egr1 promoter serum response elements. Watson DK, Robinson L, Hodge DR, Kola I, Papas TS, Seth A. Oncogene; 1997 Jan 16; 14(2):213-21. PubMed ID: 9010223 [Abstract] [Full Text] [Related]
5. Integration of growth factor signals at the c-fos serum response element. Price MA, Hill C, Treisman R. Philos Trans R Soc Lond B Biol Sci; 1996 Apr 29; 351(1339):551-9. PubMed ID: 8735278 [Abstract] [Full Text] [Related]
6. Anisomycin abrogates repression of protooncogene c-fos transcription in E1A + cHa-ras-transformed cells through activation of MEK/ERK kinase cascade. Kukushkin AN, Svetlikova SB, Amanzholov RA, Pospelov VA. J Cell Biochem; 2008 Feb 15; 103(3):1005-12. PubMed ID: 17647273 [Abstract] [Full Text] [Related]
7. Crystal structure of a ternary SAP-1/SRF/c-fos SRE DNA complex. Mo Y, Ho W, Johnston K, Marmorstein R. J Mol Biol; 2001 Nov 30; 314(3):495-506. PubMed ID: 11846562 [Abstract] [Full Text] [Related]
8. Myocardin and ternary complex factors compete for SRF to control smooth muscle gene expression. Wang Z, Wang DZ, Hockemeyer D, McAnally J, Nordheim A, Olson EN. Nature; 2004 Mar 11; 428(6979):185-9. PubMed ID: 15014501 [Abstract] [Full Text] [Related]
9. [Transformation with the E1A + cHa-ras oncogenes enhances the trans-repressor function of the Elk-1 transcription factor]. Usenko TN, Pospelova TV, Pospelov VA. Mol Biol (Mosk); 2002 Mar 11; 36(5):825-32. PubMed ID: 12391846 [Abstract] [Full Text] [Related]
10. Heparin and heparan sulfate inhibit extracellular signal-regulated kinase activation and myocardial cell hypertrophy induced by endothelin-1. Mizutani K, Kawashima S, Ueyama T, Sakoda T, Takeuchi S, Kobayashi S, Yokoyama M, Hayashi Y, Itoh H. Kobe J Med Sci; 2001 Apr 11; 47(2):47-58. PubMed ID: 11599124 [Abstract] [Full Text] [Related]
11. [The c-fos proto-oncogene promotor is not regulated by serum, epidermal growth factor, and phorbol ester in embryonal fibroblasts transformed by E1Aad5+cHa-ras-oncogenes]. Pospelova TV, Medvedev AV, Kisliakova TV, Svetlikova SB, Pospelov VA. Mol Biol (Mosk); 1991 Apr 11; 25(1):105-15. PubMed ID: 1716733 [Abstract] [Full Text] [Related]
12. Net, a negative Ras-switchable TCF, contains a second inhibition domain, the CID, that mediates repression through interactions with CtBP and de-acetylation. Criqui-Filipe P, Ducret C, Maira SM, Wasylyk B. EMBO J; 1999 Jun 15; 18(12):3392-403. PubMed ID: 10369679 [Abstract] [Full Text] [Related]
13. [Effect of anisomycin on activation of early response genes c-fos, c-jun, Egr-1 in cells transformed by E1A and cHa-ras oncogenes]. Kukushkin AN, Svetlikova SB, Pospelov VA. Mol Biol (Mosk); 2005 Jun 15; 39(1):80-8. PubMed ID: 15773551 [Abstract] [Full Text] [Related]
14. Delta elk-1, a variant of elk-1, fails to interact with the serum response factor and binds to DNA with modulated specificity. Rao VN, Reddy ES. Cancer Res; 1993 Jan 15; 53(2):215-20. PubMed ID: 8417810 [Abstract] [Full Text] [Related]
15. [Constitutive activity of MAP kinase cascades in REF cells transformed by E1A and cHa-ras oncogenes]. Svetlikova SB, Abramova MV, Kukushkin AN, Darieva ZA, Pospelova TV, Pospelov VA. Tsitologiia; 2001 Jan 15; 43(10):961-8. PubMed ID: 11769129 [Abstract] [Full Text] [Related]
16. The p38 and ERK MAP kinase pathways cooperate to activate Ternary Complex Factors and c-fos transcription in response to UV light. Price MA, Cruzalegui FH, Treisman R. EMBO J; 1996 Dec 02; 15(23):6552-63. PubMed ID: 8978682 [Abstract] [Full Text] [Related]
17. Age-associated changes in basal c-fos transcription factor binding activity in rat hearts. Tsou H, Azhar G, Lu XG, Kovacs S, Peacocke M, Wei JY. Exp Cell Res; 1996 Dec 15; 229(2):432-7. PubMed ID: 8986626 [Abstract] [Full Text] [Related]
18. Sequences at the 3' side of the c-fos SRE mediate gene expression via an Sob1-dependent, TCF-independent pathway. Omoike OI, Benson BA, Chan MA, Benedict SH. Biochem Biophys Res Commun; 1999 Aug 27; 262(2):523-9. PubMed ID: 10462507 [Abstract] [Full Text] [Related]
19. [Transcription factors STAT1 and STAT3 are localized in different compartments of rat embryo fibroblasts transformed by E1A and Ha-Ras oncogenes]. Poselova TV, Evdonin AL, Medvedeva ND, Pospelov VA. Tsitologiia; 1998 Aug 27; 40(12):1074-9. PubMed ID: 10188224 [Abstract] [Full Text] [Related]
20. The c-fos serum response element (SRE) confers negative response to glucocorticoids. Karagianni N, Tsawdaroglou N. Oncogene; 1994 Aug 27; 9(8):2327-34. PubMed ID: 8036015 [Abstract] [Full Text] [Related] Page: [Next] [New Search]