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258 related items for PubMed ID: 11886899

  • 1. Mutant Cu, Zn superoxide dismutase that causes motoneuron degeneration is present in mitochondria in the CNS.
    Higgins CM, Jung C, Ding H, Xu Z.
    J Neurosci; 2002 Mar 15; 22(6):RC215. PubMed ID: 11886899
    [Abstract] [Full Text] [Related]

  • 2. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 15; 7(6 Pt B):623-43. PubMed ID: 11114261
    [Abstract] [Full Text] [Related]

  • 3. Toxicity of familial ALS-linked SOD1 mutants from selective recruitment to spinal mitochondria.
    Liu J, Lillo C, Jonsson PA, Vande Velde C, Ward CM, Miller TM, Subramaniam JR, Rothstein JD, Marklund S, Andersen PM, Brännström T, Gredal O, Wong PC, Williams DS, Cleveland DW.
    Neuron; 2004 Jul 08; 43(1):5-17. PubMed ID: 15233913
    [Abstract] [Full Text] [Related]

  • 4. The neuroprotective factor Wlds does not attenuate mutant SOD1-mediated motor neuron disease.
    Vande Velde C, Garcia ML, Yin X, Trapp BD, Cleveland DW.
    Neuromolecular Med; 2004 Jul 08; 5(3):193-203. PubMed ID: 15626820
    [Abstract] [Full Text] [Related]

  • 5. CuZn superoxide dismutase (SOD1) accumulates in vacuolated mitochondria in transgenic mice expressing amyotrophic lateral sclerosis-linked SOD1 mutations.
    Jaarsma D, Rognoni F, van Duijn W, Verspaget HW, Haasdijk ED, Holstege JC.
    Acta Neuropathol; 2001 Oct 08; 102(4):293-305. PubMed ID: 11603803
    [Abstract] [Full Text] [Related]

  • 6. Mutant SOD1 linked to familial amyotrophic lateral sclerosis, but not wild-type SOD1, induces ER stress in COS7 cells and transgenic mice.
    Tobisawa S, Hozumi Y, Arawaka S, Koyama S, Wada M, Nagai M, Aoki M, Itoyama Y, Goto K, Kato T.
    Biochem Biophys Res Commun; 2003 Apr 04; 303(2):496-503. PubMed ID: 12659845
    [Abstract] [Full Text] [Related]

  • 7. Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity.
    Bendotti C, Calvaresi N, Chiveri L, Prelle A, Moggio M, Braga M, Silani V, De Biasi S.
    J Neurol Sci; 2001 Oct 15; 191(1-2):25-33. PubMed ID: 11676989
    [Abstract] [Full Text] [Related]

  • 8. Mouse motor neuron disease caused by truncated SOD1 with or without C-terminal modification.
    Watanabe Y, Yasui K, Nakano T, Doi K, Fukada Y, Kitayama M, Ishimoto M, Kurihara S, Kawashima M, Fukuda H, Adachi Y, Inoue T, Nakashima K.
    Brain Res Mol Brain Res; 2005 Apr 27; 135(1-2):12-20. PubMed ID: 15857664
    [Abstract] [Full Text] [Related]

  • 9. Cytochrome c association with the inner mitochondrial membrane is impaired in the CNS of G93A-SOD1 mice.
    Kirkinezos IG, Bacman SR, Hernandez D, Oca-Cossio J, Arias LJ, Perez-Pinzon MA, Bradley WG, Moraes CT.
    J Neurosci; 2005 Jan 05; 25(1):164-72. PubMed ID: 15634778
    [Abstract] [Full Text] [Related]

  • 10. Mutant SOD1 in neuronal mitochondria causes toxicity and mitochondrial dynamics abnormalities.
    Magrané J, Hervias I, Henning MS, Damiano M, Kawamata H, Manfredi G.
    Hum Mol Genet; 2009 Dec 01; 18(23):4552-64. PubMed ID: 19779023
    [Abstract] [Full Text] [Related]

  • 11. ALS-associated mutant SOD1G93A causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes.
    Higgins CM, Jung C, Xu Z.
    BMC Neurosci; 2003 Jul 15; 4():16. PubMed ID: 12864925
    [Abstract] [Full Text] [Related]

  • 12. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 15; 64():48-59. PubMed ID: 24361555
    [Abstract] [Full Text] [Related]

  • 13. Ultrastructural study of mitochondria in the spinal cord of transgenic mice with a G93A mutant SOD1 gene.
    Sasaki S, Warita H, Murakami T, Abe K, Iwata M.
    Acta Neuropathol; 2004 May 15; 107(5):461-74. PubMed ID: 15029445
    [Abstract] [Full Text] [Related]

  • 14. Calcium-permeable AMPA receptors promote misfolding of mutant SOD1 protein and development of amyotrophic lateral sclerosis in a transgenic mouse model.
    Tateno M, Sadakata H, Tanaka M, Itohara S, Shin RM, Miura M, Masuda M, Aosaki T, Urushitani M, Misawa H, Takahashi R.
    Hum Mol Genet; 2004 Oct 01; 13(19):2183-96. PubMed ID: 15294873
    [Abstract] [Full Text] [Related]

  • 15. The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease.
    Mourelatos Z, Gonatas NK, Stieber A, Gurney ME, Dal Canto MC.
    Proc Natl Acad Sci U S A; 1996 May 28; 93(11):5472-7. PubMed ID: 8643599
    [Abstract] [Full Text] [Related]

  • 16. Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS).
    Howland DS, Liu J, She Y, Goad B, Maragakis NJ, Kim B, Erickson J, Kulik J, DeVito L, Psaltis G, DeGennaro LJ, Cleveland DW, Rothstein JD.
    Proc Natl Acad Sci U S A; 2002 Feb 05; 99(3):1604-9. PubMed ID: 11818550
    [Abstract] [Full Text] [Related]

  • 17. Mutant SOD1G93A triggers mitochondrial fragmentation in spinal cord motor neurons: neuroprotection by SIRT3 and PGC-1α.
    Song W, Song Y, Kincaid B, Bossy B, Bossy-Wetzel E.
    Neurobiol Dis; 2013 Mar 05; 51():72-81. PubMed ID: 22819776
    [Abstract] [Full Text] [Related]

  • 18. Increased mitochondrial antioxidative activity or decreased oxygen free radical propagation prevent mutant SOD1-mediated motor neuron cell death and increase amyotrophic lateral sclerosis-like transgenic mouse survival.
    Liu R, Li B, Flanagan SW, Oberley LW, Gozal D, Qiu M.
    J Neurochem; 2002 Feb 05; 80(3):488-500. PubMed ID: 11905995
    [Abstract] [Full Text] [Related]

  • 19. Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology.
    Son M, Puttaparthi K, Kawamata H, Rajendran B, Boyer PJ, Manfredi G, Elliott JL.
    Proc Natl Acad Sci U S A; 2007 Apr 03; 104(14):6072-7. PubMed ID: 17389365
    [Abstract] [Full Text] [Related]

  • 20. Restricted expression of G86R Cu/Zn superoxide dismutase in astrocytes results in astrocytosis but does not cause motoneuron degeneration.
    Gong YH, Parsadanian AS, Andreeva A, Snider WD, Elliott JL.
    J Neurosci; 2000 Jan 15; 20(2):660-5. PubMed ID: 10632595
    [Abstract] [Full Text] [Related]

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