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Journal Abstract Search

144 related items for PubMed ID: 11900326

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  • 4. Mitochondrial "movement" and lens optics following oxidative stress from UV-B irradiation: cultured bovine lenses and human retinal pigment epithelial cells (ARPE-19) as examples.
    Bantseev V, Youn HY.
    Ann N Y Acad Sci; 2006 Dec; 1091():17-33. PubMed ID: 17341599
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  • 7. Review: Age-related cataract: immunity and lens epithelium-derived growth factor (LEDGF).
    Shinohara T, Singh DP, Chylack LT.
    J Ocul Pharmacol Ther; 2000 Apr; 16(2):181-91. PubMed ID: 10803429
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  • 16. Evolution of damage in the lens after in vivo close to threshold exposure to UV-B radiation: cytomorphological study of apoptosis.
    Galichanin K, Löfgren S, Bergmanson J, Söderberg P.
    Exp Eye Res; 2010 Sep; 91(3):369-77. PubMed ID: 20599969
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  • 17. Experimental cataract induced by ultraviolet radiation.
    Söderberg PG.
    Acta Ophthalmol Suppl (1985); 1990 Sep; (196):1-75. PubMed ID: 2161611
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  • 18. Repair in the rat lens after threshold ultraviolet radiation injury.
    Michael R, Vrensen GF, van Marle J, Löfgren S, Söderberg PG.
    Invest Ophthalmol Vis Sci; 2000 Jan; 41(1):204-12. PubMed ID: 10634622
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  • 19. Reactive Oxygen Species and the Aging Eye: Specific Role of Metabolically Active Mitochondria in Maintaining Lens Function and in the Initiation of the Oxidation-Induced Maturity Onset Cataract--A Novel Platform of Mitochondria-Targeted Antioxidants With Broad Therapeutic Potential for Redox Regulation and Detoxification of Oxidants in Eye Diseases.
    Babizhayev MA, Yegorov YE.
    Am J Ther; 2016 Jan; 23(1):e98-117. PubMed ID: 21048433
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