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100 related items for PubMed ID: 11908943

  • 1. Absence of CD5 dramatically reduces progression of pulmonary inflammatory lesions in SHP-1 protein-tyrosine phosphatase-deficient 'viable motheaten' mice.
    Joliat MJ, Lang PA, Lyons BL, Burzenski L, Lynes MA, Yi T, Sundberg JP, Shultz LD.
    J Autoimmun; 2002 Mar; 18(2):105-17. PubMed ID: 11908943
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  • 2. T cell developmental defects in 'viable motheaten' mice deficient in SHP-1 protein-tyrosine phosphatase. Developmental defects are corrected in vitro in the presence of normal hematopoietic-origin stromal cells and in vivo by exogenous IL-7.
    Christianson SW, Greiner DL, Deluca D, Leif J, Phillips NE, Hayes SM, Hayashi S, Joliat MJ, Lyons BL, Shultz LD.
    J Autoimmun; 2002 Mar; 18(2):119-30. PubMed ID: 11908944
    [Abstract] [Full Text] [Related]

  • 3. Deficiency of SHP-1 protein-tyrosine phosphatase in "viable motheaten" mice results in retinal degeneration.
    Lyons BL, Smith RS, Hurd RE, Hawes NL, Burzenski LM, Nusinowitz S, Hasham MG, Chang B, Shultz LD.
    Invest Ophthalmol Vis Sci; 2006 Mar; 47(3):1201-9. PubMed ID: 16505059
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  • 4. DNA-binding antibodies from viable motheaten mutant mice: implications for B cell tolerance.
    Westhoff CM, Whittier A, Kathol S, McHugh J, Zajicek C, Shultz LD, Wylie DE.
    J Immunol; 1997 Sep 15; 159(6):3024-33. PubMed ID: 9300728
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  • 5. Hematopoietic cell protein-tyrosine phosphatase-deficient motheaten mice exhibit T cell apoptosis defect.
    Su X, Zhou T, Yang PA, Wang Z, Mountz JD.
    J Immunol; 1996 Jun 01; 156(11):4198-208. PubMed ID: 8666788
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  • 7. Increased G-CSF responsiveness of bone marrow cells from hematopoietic cell phosphatase deficient viable motheaten mice.
    Tapley P, Shevde NK, Schweitzer PA, Gallina M, Christianson SW, Lin IL, Stein RB, Shultz LD, Rosen J, Lamb P.
    Exp Hematol; 1997 Feb 01; 25(2):122-31. PubMed ID: 9015212
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  • 8. SHP-1 regulates STAT6 phosphorylation and IL-4-mediated function in a cell type-specific manner.
    Huang Z, Coleman JM, Su Y, Mann M, Ryan J, Shultz LD, Huang H.
    Cytokine; 2005 Feb 07; 29(3):118-24. PubMed ID: 15613279
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  • 9. Mutation of the hematopoietic cell phosphatase (Hcph) gene is associated with resistance to gamma-irradiation-induced apoptosis in Src homology protein tyrosine phosphatase (SHP)-1-deficient "motheaten" mutant mice.
    Hsu HC, Shultz LD, Su X, Shi J, Yang PA, Relyea MJ, Zhang HG, Mountz JD.
    J Immunol; 2001 Jan 15; 166(2):772-80. PubMed ID: 11145649
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  • 10. Functional consequences of the SHP-1 defect in motheaten viable mice: role of NF-kappa B.
    Khaled AR, Butfiloski EJ, Sobel ES, Schiffenbauer J.
    Cell Immunol; 1998 Apr 10; 185(1):49-58. PubMed ID: 9636682
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  • 11. Motheaten (me/me) mice deficient in SHP-1 are less susceptible to focal cerebral ischemia.
    Beamer CA, Brooks DM, Lurie DI.
    J Neurosci Res; 2006 May 15; 83(7):1220-30. PubMed ID: 16528752
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  • 13. Motheaten and viable motheaten mice have mutations in the haematopoietic cell phosphatase gene.
    Tsui HW, Siminovitch KA, de Souza L, Tsui FW.
    Nat Genet; 1993 Jun 15; 4(2):124-9. PubMed ID: 8348149
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  • 16. Estrogen induces normal murine CD5+ B cells to produce autoantibodies.
    Ansar Ahmed S, Dauphinée MJ, Montoya AI, Talal N.
    J Immunol; 1989 Apr 15; 142(8):2647-53. PubMed ID: 2467934
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  • 17. Loss of SHP-1 phosphatase alters cytokine expression in the mouse hindbrain following cochlear ablation.
    Zhao J, Lurie DI.
    Cytokine; 2004 Oct 07; 28(1):1-9. PubMed ID: 15341919
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  • 18. Genetic ablation of protein tyrosine phosphatase 1B accelerates lymphomagenesis of p53-null mice through the regulation of B-cell development.
    Dubé N, Bourdeau A, Heinonen KM, Cheng A, Loy AL, Tremblay ML.
    Cancer Res; 2005 Nov 01; 65(21):10088-95. PubMed ID: 16267035
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  • 19. Increased negative selection impairs neonatal B cell repertoire but does not directly lead to generation of disease-associated IgM auto-antibodies.
    Cassady-Cain RL, Kaushik AK.
    Int Immunol; 2006 May 01; 18(5):661-9. PubMed ID: 16569683
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  • 20. Defective lymphopoiesis in the bone marrow of motheaten (me/me) and viable motheaten (mev/mev) mutant mice. II. Description of a microenvironmental defect for the generation of terminal deoxynucleotidyltransferase-positive bone marrow cells in vitro.
    Medlock ES, Goldschneider I, Greiner DL, Shultz L.
    J Immunol; 1987 Jun 01; 138(11):3590-7. PubMed ID: 3584969
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