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Journal Abstract Search

371 related items for PubMed ID: 11950933

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  • 2. Inhibition of protein prenylation by bisphosphonates causes sustained activation of Rac, Cdc42, and Rho GTPases.
    Dunford JE, Rogers MJ, Ebetino FH, Phipps RJ, Coxon FP.
    J Bone Miner Res; 2006 May; 21(5):684-94. PubMed ID: 16734383
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  • 4. Dissociation of recruitment and activation of the small G-protein Rac during Fcgamma receptor-mediated phagocytosis.
    Cougoule C, Hoshino S, Dart A, Lim J, Caron E.
    J Biol Chem; 2006 Mar 31; 281(13):8756-64. PubMed ID: 16434390
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  • 5. Temporally and spatially coordinated roles for Rho, Rac, Cdc42 and their effectors in growth cone guidance by a physiological electric field.
    Rajnicek AM, Foubister LE, McCaig CD.
    J Cell Sci; 2006 May 01; 119(Pt 9):1723-35. PubMed ID: 16595546
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  • 6. How Vav proteins discriminate the GTPases Rac1 and RhoA from Cdc42.
    Movilla N, Dosil M, Zheng Y, Bustelo XR.
    Oncogene; 2001 Dec 06; 20(56):8057-65. PubMed ID: 11781818
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  • 8. Vav transformation requires activation of multiple GTPases and regulation of gene expression.
    Palmby TR, Abe K, Karnoub AE, Der CJ.
    Mol Cancer Res; 2004 Dec 06; 2(12):702-11. PubMed ID: 15634759
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  • 13. In vitro guanine nucleotide exchange activity of DHR-2/DOCKER/CZH2 domains.
    Côté JF, Vuori K.
    Methods Enzymol; 2006 Dec 06; 406():41-57. PubMed ID: 16472648
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  • 14. Requirements for Vav guanine nucleotide exchange factors and Rho GTPases in FcgammaR- and complement-mediated phagocytosis.
    Hall AB, Gakidis MA, Glogauer M, Wilsbacher JL, Gao S, Swat W, Brugge JS.
    Immunity; 2006 Mar 06; 24(3):305-16. PubMed ID: 16546099
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  • 15. Activation of Rac by cadherin through the c-Src-Rap1-phosphatidylinositol 3-kinase-Vav2 pathway.
    Fukuyama T, Ogita H, Kawakatsu T, Inagaki M, Takai Y.
    Oncogene; 2006 Jan 05; 25(1):8-19. PubMed ID: 16170364
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  • 16. Dock6, a Dock-C subfamily guanine nucleotide exchanger, has the dual specificity for Rac1 and Cdc42 and regulates neurite outgrowth.
    Miyamoto Y, Yamauchi J, Sanbe A, Tanoue A.
    Exp Cell Res; 2007 Feb 15; 313(4):791-804. PubMed ID: 17196961
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  • 17. Vav2 as a Rac-GDP/GTP exchange factor responsible for the nectin-induced, c-Src- and Cdc42-mediated activation of Rac.
    Kawakatsu T, Ogita H, Fukuhara T, Fukuyama T, Minami Y, Shimizu K, Takai Y.
    J Biol Chem; 2005 Feb 11; 280(6):4940-7. PubMed ID: 15485841
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  • 18. MyD88-independent activation of a novel actin-Cdc42/Rac pathway is required for Toll-like receptor-stimulated phagocytosis.
    Kong L, Ge BX.
    Cell Res; 2008 Jul 11; 18(7):745-55. PubMed ID: 18542102
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  • 19. Oncogenic Dbl, Cdc42, and p21-activated kinase form a ternary signaling intermediate through the minimum interactive domains.
    Wang L, Zhu K, Zheng Y.
    Biochemistry; 2004 Nov 23; 43(46):14584-93. PubMed ID: 15544329
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  • 20. Activated c-Fms recruits Vav and Rac during CSF-1-induced cytoskeletal remodeling and spreading in osteoclasts.
    Sakai H, Chen Y, Itokawa T, Yu KP, Zhu ML, Insogna K.
    Bone; 2006 Dec 23; 39(6):1290-301. PubMed ID: 16950670
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