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Journal Abstract Search


651 related items for PubMed ID: 11988019

  • 1. Retinal pathology and function in a Cln3 knockout mouse model of juvenile Neuronal Ceroid Lipofuscinosis (batten disease).
    Seigel GM, Lotery A, Kummer A, Bernard DJ, Greene ND, Turmaine M, Derksen T, Nussbaum RL, Davidson B, Wagner J, Mitchison HM.
    Mol Cell Neurosci; 2002 Apr; 19(4):515-27. PubMed ID: 11988019
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  • 2. Altered gene expression in the eye of a mouse model for batten disease.
    Chattopadhyay S, Kingsley E, Serour A, Curran TM, Brooks AI, Pearce DA.
    Invest Ophthalmol Vis Sci; 2004 Sep; 45(9):2893-905. PubMed ID: 15326100
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  • 3. A mouse gene knockout model for juvenile ceroid-lipofuscinosis (Batten disease).
    Katz ML, Shibuya H, Liu PC, Kaur S, Gao CL, Johnson GS.
    J Neurosci Res; 1999 Aug 15; 57(4):551-6. PubMed ID: 10440905
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  • 4. Progression of early postnatal retinal pathology in a mouse model of neuronal ceroid lipofuscinosis.
    Seigel GM, Wagner J, Wronska A, Campbell L, Ju W, Zhong N.
    Eye (Lond); 2005 Dec 15; 19(12):1306-12. PubMed ID: 15565184
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  • 5. Targeted disruption of the Cln3 gene provides a mouse model for Batten disease. The Batten Mouse Model Consortium [corrected].
    Mitchison HM, Bernard DJ, Greene ND, Cooper JD, Junaid MA, Pullarkat RK, de Vos N, Breuning MH, Owens JW, Mobley WC, Gardiner RM, Lake BD, Taschner PE, Nussbaum RL.
    Neurobiol Dis; 1999 Oct 15; 6(5):321-34. PubMed ID: 10527801
    [Abstract] [Full Text] [Related]

  • 6. Immunochemical localization of the Batten disease (CLN3) protein in retina.
    Katz ML, Gao CL, Prabhakaram M, Shibuya H, Liu PC, Johnson GS.
    Invest Ophthalmol Vis Sci; 1997 Oct 15; 38(11):2375-86. PubMed ID: 9344361
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  • 7. Developmental impairments of select neurotransmitter systems in brains of Cln3(Deltaex7/8) knock-in mice, an animal model of juvenile neuronal ceroid lipofuscinosis.
    Herrmann P, Druckrey-Fiskaaen C, Kouznetsova E, Heinitz K, Bigl M, Cotman SL, Schliebs R.
    J Neurosci Res; 2008 Jun 15; 86(8):1857-70. PubMed ID: 18265413
    [Abstract] [Full Text] [Related]

  • 8. Altered arginine metabolism in the central nervous system (CNS) of the Cln3-/- mouse model of juvenile Batten disease.
    Chan CH, Ramirez-Montealegre D, Pearce DA.
    Neuropathol Appl Neurobiol; 2009 Apr 15; 35(2):189-207. PubMed ID: 19284480
    [Abstract] [Full Text] [Related]

  • 9. Protracted course of juvenile ceroid lipofuscinosis associated with a novel CLN3 mutation (p.Y199X).
    Sarpong A, Schottmann G, Rüther K, Stoltenburg G, Kohlschütter A, Hübner C, Schuelke M.
    Clin Genet; 2009 Jul 15; 76(1):38-45. PubMed ID: 19489875
    [Abstract] [Full Text] [Related]

  • 10. A murine model for juvenile NCL: gene targeting of mouse Cln3.
    Greene ND, Bernard DL, Taschner PE, Lake BD, de Vos N, Breuning MH, Gardiner RM, Mole SE, Nussbaum RL, Mitchison HM.
    Mol Genet Metab; 1999 Apr 15; 66(4):309-13. PubMed ID: 10191119
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  • 18. A knock-in reporter mouse model for Batten disease reveals predominant expression of Cln3 in visual, limbic and subcortical motor structures.
    Ding SL, Tecedor L, Stein CS, Davidson BL.
    Neurobiol Dis; 2011 Feb 15; 41(2):237-48. PubMed ID: 20875858
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  • 20. Optic nerve degeneration in a murine model of juvenile ceroid lipofuscinosis.
    Sappington RM, Pearce DA, Calkins DJ.
    Invest Ophthalmol Vis Sci; 2003 Sep 15; 44(9):3725-31. PubMed ID: 12939285
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