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Journal Abstract Search

167 related items for PubMed ID: 11988605

  • 1. Brain representation of hemifield stimulation in poststroke visual field defects.
    Nelles G, Widman G, de Greiff A, Meistrowitz A, Dimitrova A, Weber J, Forsting M, Esser J, Diener HC.
    Stroke; 2002 May; 33(5):1286-93. PubMed ID: 11988605
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  • 2. Cortical activation in hemianopia after stroke.
    Nelles G, de Greiff A, Pscherer A, Forsting M, Gerhard H, Esser J, Diener HC.
    Neurosci Lett; 2007 Oct 09; 426(1):34-8. PubMed ID: 17881128
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  • 3. Saccade induced cortical activation in patients with post-stroke visual field defects.
    Nelles G, de Greiff A, Pscherer A, Stude P, Forsting M, Hufnagel A, Gerhard H, Esser J, Diener HC.
    J Neurol; 2007 Sep 09; 254(9):1244-52. PubMed ID: 17694385
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  • 4. Eye-movement training-induced changes of visual field representation in patients with post-stroke hemianopia.
    Nelles G, Pscherer A, de Greiff A, Gerhard H, Forsting M, Esser J, Diener HC.
    J Neurol; 2010 Nov 09; 257(11):1832-40. PubMed ID: 20532906
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  • 6. Eye-movement training-induced plasticity in patients with post-stroke hemianopia.
    Nelles G, Pscherer A, de Greiff A, Forsting M, Gerhard H, Esser J, Diener HC.
    J Neurol; 2009 May 09; 256(5):726-33. PubMed ID: 19240963
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  • 7. Brain activity associated with stimulation therapy of the visual borderzone in hemianopic stroke patients.
    Marshall RS, Ferrera JJ, Barnes A, Xian Zhang, O'Brien KA, Chmayssani M, Hirsch J, Lazar RM.
    Neurorehabil Neural Repair; 2008 May 09; 22(2):136-44. PubMed ID: 17698955
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  • 8. An FMRI investigation of the cortical network underlying detection and categorization abilities in hemianopic patients.
    Perez C, Peyrin C, Cavézian C, Coubard O, Caetta F, Raz N, Levin N, Doucet G, Andersson F, Obadia M, Gout O, Héran F, Savatovsky J, Chokron S.
    Brain Topogr; 2013 Apr 09; 26(2):264-77. PubMed ID: 22878845
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  • 9. Specific impairments in visual processing following lesion side in hemianopic patients.
    Cavézian C, Gaudry I, Perez C, Coubard O, Doucet G, Peyrin C, Marendaz C, Obadia M, Gout O, Chokron S.
    Cortex; 2010 Oct 09; 46(9):1123-31. PubMed ID: 19880103
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  • 14. Hemifield pattern-reversal visual evoked potentials (VEPs) in retrochiasmal lesions with homonymous visual field defect.
    Maccolini E, Andreoli A, Valdé G, Ghini M, Fulco L.
    Ital J Neurol Sci; 1986 Aug 09; 7(4):437-42. PubMed ID: 3759417
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  • 15. Sectoral analysis of the retinal nerve fiber layer thinning and its association with visual field loss in homonymous hemianopia caused by post-geniculate lesions using spectral-domain optical coherence tomography.
    Goto K, Miki A, Yamashita T, Araki S, Takizawa G, Nakagawa M, Ieki Y, Kiryu J.
    Graefes Arch Clin Exp Ophthalmol; 2016 Apr 09; 254(4):745-56. PubMed ID: 26446718
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  • 16. Clinical study of the visual field defects caused by occipital lobe lesions.
    Ogawa K, Ishikawa H, Suzuki Y, Oishi M, Kamei S.
    Cerebrovasc Dis; 2014 Apr 09; 37(2):102-8. PubMed ID: 24435066
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  • 17. Combining visual rehabilitative training and noninvasive brain stimulation to enhance visual function in patients with hemianopia: a comparative case study.
    Plow EB, Obretenova SN, Halko MA, Kenkel S, Jackson ML, Pascual-Leone A, Merabet LB.
    PM R; 2011 Sep 09; 3(9):825-35. PubMed ID: 21944300
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  • 18. The cortical representation of foveal stimuli: evidence from quadrantanopia and TMS-induced suppression.
    Chiang TC, Walsh V, Lavidor M.
    Brain Res Cogn Brain Res; 2004 Nov 09; 21(3):309-16. PubMed ID: 15511647
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