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700 related items for PubMed ID: 12082612
1. Nup98-HoxA9 immortalizes myeloid progenitors, enforces expression of Hoxa9, Hoxa7 and Meis1, and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1. Calvo KR, Sykes DB, Pasillas MP, Kamps MP. Oncogene; 2002 Jun 20; 21(27):4247-56. PubMed ID: 12082612 [Abstract] [Full Text] [Related]
2. NUP98-NSD1 links H3K36 methylation to Hox-A gene activation and leukaemogenesis. Wang GG, Cai L, Pasillas MP, Kamps MP. Nat Cell Biol; 2007 Jul 20; 9(7):804-12. PubMed ID: 17589499 [Abstract] [Full Text] [Related]
3. NUP98-HOXA9 induces long-term proliferation and blocks differentiation of primary human CD34+ hematopoietic cells. Takeda A, Goolsby C, Yaseen NR. Cancer Res; 2006 Jul 01; 66(13):6628-37. PubMed ID: 16818636 [Abstract] [Full Text] [Related]
4. Meis1a suppresses differentiation by G-CSF and promotes proliferation by SCF: potential mechanisms of cooperativity with Hoxa9 in myeloid leukemia. Calvo KR, Knoepfler PS, Sykes DB, Pasillas MP, Kamps MP. Proc Natl Acad Sci U S A; 2001 Nov 06; 98(23):13120-5. PubMed ID: 11687616 [Abstract] [Full Text] [Related]
5. The chromosome translocation t(7;11)(p15;p15) in acute myeloid leukemia results in fusion of the NUP98 gene with a HOXA cluster gene, HOXA13, but not HOXA9. Taketani T, Taki T, Ono R, Kobayashi Y, Ida K, Hayashi Y. Genes Chromosomes Cancer; 2002 Aug 06; 34(4):437-43. PubMed ID: 12112533 [Abstract] [Full Text] [Related]
6. Leukemogenic properties of NUP98-PMX1 are linked to NUP98 and homeodomain sequence functions but not to binding properties of PMX1 to serum response factor. Hirose K, Abramovich C, Argiropoulos B, Humphries RK. Oncogene; 2008 Oct 09; 27(46):6056-67. PubMed ID: 18604245 [Abstract] [Full Text] [Related]
7. Enforced expression of NUP98-HOXA9 in human CD34(+) cells enhances stem cell proliferation. Chung KY, Morrone G, Schuringa JJ, Plasilova M, Shieh JH, Zhang Y, Zhou P, Moore MA. Cancer Res; 2006 Dec 15; 66(24):11781-91. PubMed ID: 17178874 [Abstract] [Full Text] [Related]
8. NUP98 dysregulation in myeloid leukemogenesis. Moore MA, Chung KY, Plasilova M, Schuringa JJ, Shieh JH, Zhou P, Morrone G. Ann N Y Acad Sci; 2007 Jun 15; 1106():114-42. PubMed ID: 17442773 [Abstract] [Full Text] [Related]
9. Oncoprotein E2A-Pbx1 immortalizes a myeloid progenitor in primary marrow cultures without abrogating its factor-dependence. Kamps MP, Wright DD. Oncogene; 1994 Nov 15; 9(11):3159-66. PubMed ID: 7936638 [Abstract] [Full Text] [Related]
11. HoxB8 requires its Pbx-interaction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation. Knoepfler PS, Sykes DB, Pasillas M, Kamps MP. Oncogene; 2001 Sep 06; 20(39):5440-8. PubMed ID: 11571641 [Abstract] [Full Text] [Related]
12. NUP98-HOXA9-transgenic zebrafish develop a myeloproliferative neoplasm and provide new insight into mechanisms of myeloid leukaemogenesis. Forrester AM, Grabher C, McBride ER, Boyd ER, Vigerstad MH, Edgar A, Kai FB, Da'as SI, Payne E, Look AT, Berman JN. Br J Haematol; 2011 Oct 06; 155(2):167-81. PubMed ID: 21810091 [Abstract] [Full Text] [Related]
13. E2a/Pbx1 oncogene inhibits terminal differentiation but not myeloid potential of pro-T cells. Bourette RP, Grasset MF, Mouchiroud G. Oncogene; 2007 Jan 11; 26(2):234-47. PubMed ID: 16819510 [Abstract] [Full Text] [Related]
14. Hoxa9 immortalizes a granulocyte-macrophage colony-stimulating factor-dependent promyelocyte capable of biphenotypic differentiation to neutrophils or macrophages, independent of enforced meis expression. Calvo KR, Sykes DB, Pasillas M, Kamps MP. Mol Cell Biol; 2000 May 11; 20(9):3274-85. PubMed ID: 10757811 [Abstract] [Full Text] [Related]
15. Persistent transactivation by meis1 replaces hox function in myeloid leukemogenesis models: evidence for co-occupancy of meis1-pbx and hox-pbx complexes on promoters of leukemia-associated genes. Wang GG, Pasillas MP, Kamps MP. Mol Cell Biol; 2006 May 11; 26(10):3902-16. PubMed ID: 16648484 [Abstract] [Full Text] [Related]
16. A distinct epigenetic signature at targets of a leukemia protein. Rossetti S, Hoogeveen AT, Liang P, Stanciu C, van der Spek P, Sacchi N. BMC Genomics; 2007 Feb 01; 8():38. PubMed ID: 17266773 [Abstract] [Full Text] [Related]
17. In vitro induction of inhibitory macrophage differentiation by granulocyte-macrophage colony-stimulating factor, stem cell factor and interferon-gamma from lineage phenotypes-negative c-kit-positive murine hematopoietic progenitor cells. Ferret-Bernard S, Saï P, Bach JM. Immunol Lett; 2004 Feb 15; 91(2-3):221-7. PubMed ID: 15019293 [Abstract] [Full Text] [Related]
18. [HUP98-HOXA9 transgenic mice are susceptible to N-ethyl-N-nitrosourea stimulation in leukemogenesis]. Lu SY, Gu MM, Wang Y, Tan Y, Sun YP, Wang L, Kong H, Lu ZY, Wang ZG. Zhonghua Xue Ye Xue Za Zhi; 2004 May 15; 25(5):257-61. PubMed ID: 15182530 [Abstract] [Full Text] [Related]
19. Acceleration of chronic myeloproliferation by enforced expression of Meis1 or Meis3 in Icsbp-deficient bone marrow cells. Hara T, Schwieger M, Kazama R, Okamoto S, Minehata K, Ziegler M, Löhler J, Stocking C. Oncogene; 2008 Jun 19; 27(27):3865-9. PubMed ID: 18223676 [Abstract] [Full Text] [Related]
20. Hoxa9 transforms primary bone marrow cells through specific collaboration with Meis1a but not Pbx1b. Kroon E, Krosl J, Thorsteinsdottir U, Baban S, Buchberg AM, Sauvageau G. EMBO J; 1998 Jul 01; 17(13):3714-25. PubMed ID: 9649441 [Abstract] [Full Text] [Related] Page: [Next] [New Search]