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Journal Abstract Search

289 related items for PubMed ID: 12149655

  • 1. Repression of c-Cbl leads to enhanced G-CSF Jak-STAT signaling without increased cell proliferation.
    Wang L, Rudert WA, Loutaev I, Roginskaya V, Corey SJ.
    Oncogene; 2002 Aug 08; 21(34):5346-55. PubMed ID: 12149655
    [Abstract] [Full Text] [Related]

  • 2. Involvement of Shc and Cbl-PI 3-kinase in Lyn-dependent proliferative signaling pathways for G-CSF.
    Grishin A, Sinha S, Roginskaya V, Boyer MJ, Gomez-Cambronero J, Zuo S, Kurosaki T, Romero G, Corey SJ.
    Oncogene; 2000 Jan 06; 19(1):97-105. PubMed ID: 10644984
    [Abstract] [Full Text] [Related]

  • 3. Interleukin-7 signaling in human B cell precursor acute lymphoblastic leukemia cells and murine BAF3 cells involves activation of STAT1 and STAT5 mediated via the interleukin-7 receptor alpha chain.
    van der Plas DC, Smiers F, Pouwels K, Hoefsloot LH, Löwenberg B, Touw IP.
    Leukemia; 1996 Aug 06; 10(8):1317-25. PubMed ID: 8709637
    [Abstract] [Full Text] [Related]

  • 4. Dissociation of the Jak kinase pathway from G-CSF receptor signaling in neutrophils.
    Avalos BR, Parker JM, Ware DA, Hunter MG, Sibert KA, Druker BJ.
    Exp Hematol; 1997 Feb 06; 25(2):160-8. PubMed ID: 9015216
    [Abstract] [Full Text] [Related]

  • 5. Jak1 plays an essential role for receptor phosphorylation and Stat activation in response to granulocyte colony-stimulating factor.
    Shimoda K, Feng J, Murakami H, Nagata S, Watling D, Rogers NC, Stark GR, Kerr IM, Ihle JN.
    Blood; 1997 Jul 15; 90(2):597-604. PubMed ID: 9226159
    [Abstract] [Full Text] [Related]

  • 6. Engagement of the B-cell antigen receptor activates STAT through Lyn in a Jak-independent pathway.
    Wang L, Kurosaki T, Corey SJ.
    Oncogene; 2007 May 03; 26(20):2851-9. PubMed ID: 17146444
    [Abstract] [Full Text] [Related]

  • 7. Multiple signaling pathways induced by granulocyte colony-stimulating factor involving activation of JAKs, STAT5, and/or STAT3 are required for regulation of three distinct classes of immediate early genes.
    Tian SS, Tapley P, Sincich C, Stein RB, Rosen J, Lamb P.
    Blood; 1996 Dec 15; 88(12):4435-44. PubMed ID: 8977235
    [Abstract] [Full Text] [Related]

  • 8. Requirement of Src kinase Lyn for induction of DNA synthesis by granulocyte colony-stimulating factor.
    Corey SJ, Dombrosky-Ferlan PM, Zuo S, Krohn E, Donnenberg AD, Zorich P, Romero G, Takata M, Kurosaki T.
    J Biol Chem; 1998 Feb 06; 273(6):3230-5. PubMed ID: 9452436
    [Abstract] [Full Text] [Related]

  • 9. Tryptophan 650 of human granulocyte colony-stimulating factor (G-CSF) receptor, implicated in the activation of JAK2, is also required for G-CSF-mediated activation of signaling complexes of the p21ras route.
    Barge RM, de Koning JP, Pouwels K, Dong F, Löwenberg B, Touw IP.
    Blood; 1996 Mar 15; 87(6):2148-53. PubMed ID: 8630373
    [Abstract] [Full Text] [Related]

  • 10. Regulation of STAT protein synthesis by c-Cbl.
    Blesofsky WA, Mowen K, Arduini RM, Baker DP, Murphy MA, Bowtell DD, David M.
    Oncogene; 2001 Nov 01; 20(50):7326-33. PubMed ID: 11704862
    [Abstract] [Full Text] [Related]

  • 11. Distinct regions of the granulocyte colony-stimulating factor receptor are required for tyrosine phosphorylation of the signaling molecules JAK2, Stat3, and p42, p44MAPK.
    Nicholson SE, Novak U, Ziegler SF, Layton JE.
    Blood; 1995 Nov 15; 86(10):3698-704. PubMed ID: 7579336
    [Abstract] [Full Text] [Related]

  • 12. c-Cbl is a negative regulator of GH-stimulated STAT5-mediated transcription.
    Goh EL, Zhu T, Leong WY, Lobie PE.
    Endocrinology; 2002 Sep 15; 143(9):3590-603. PubMed ID: 12193575
    [Abstract] [Full Text] [Related]

  • 13. APS, an adaptor protein containing Pleckstrin homology (PH) and Src homology-2 (SH2) domains inhibits the JAK-STAT pathway in collaboration with c-Cbl.
    Wakioka T, Sasaki A, Mitsui K, Yokouchi M, Inoue A, Komiya S, Yoshimura A.
    Leukemia; 1999 May 15; 13(5):760-7. PubMed ID: 10374881
    [Abstract] [Full Text] [Related]

  • 14. Platelet-derived growth factor (PDGF)-induced activation of signal transducer and activator of transcription (Stat) 5 is mediated by PDGF beta-receptor and is not dependent on c-src, fyn, jak1 or jak2 kinases.
    Paukku K, Valgeirsdóttir S, Saharinen P, Bergman M, Heldin CH, Silvennoinen O.
    Biochem J; 2000 Feb 01; 345 Pt 3(Pt 3):759-66. PubMed ID: 10642538
    [Abstract] [Full Text] [Related]

  • 15. Yeast two-hybrid in vivo association of the Src kinase Lyn with the proto-oncogene product Cbl but not with the p85 subunit of PI 3-kinase.
    Dombrosky-Ferlan PM, Corey SJ.
    Oncogene; 1997 May 01; 14(17):2019-24. PubMed ID: 9160881
    [Abstract] [Full Text] [Related]

  • 16. G-CSF-induced tyrosine phosphorylation of Gab2 is Lyn kinase dependent and associated with enhanced Akt and differentiative, not proliferative, responses.
    Zhu QS, Robinson LJ, Roginskaya V, Corey SJ.
    Blood; 2004 May 01; 103(9):3305-12. PubMed ID: 14656892
    [Abstract] [Full Text] [Related]

  • 17. A beta 1 integrin signaling pathway involving Src-family kinases, Cbl and PI-3 kinase is required for macrophage spreading and migration.
    Meng F, Lowell CA.
    EMBO J; 1998 Aug 03; 17(15):4391-403. PubMed ID: 9687507
    [Abstract] [Full Text] [Related]

  • 18. Tyrosine phosphorylation of Shc is not required for proliferation or viability signaling by granulocyte-macrophage colony-stimulating factor in hematopoietic cell lines.
    Durstin M, Inhorn RC, Griffin JD.
    J Immunol; 1996 Jul 15; 157(2):534-40. PubMed ID: 8752899
    [Abstract] [Full Text] [Related]

  • 19. CSF-1 signal transduction.
    Hamilton JA.
    J Leukoc Biol; 1997 Aug 15; 62(2):145-55. PubMed ID: 9261328
    [Abstract] [Full Text] [Related]

  • 20. Macrophages from motheaten and viable motheaten mutant mice show increased proliferative responses to GM-CSF: detection of potential HCP substrates in GM-CSF signal transduction.
    Jiao H, Yang W, Berrada K, Tabrizi M, Shultz L, Yi T.
    Exp Hematol; 1997 Jul 15; 25(7):592-600. PubMed ID: 9216734
    [Abstract] [Full Text] [Related]

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