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Journal Abstract Search

356 related items for PubMed ID: 122331

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  • 2. Raised sodium pump activity and a circulating sodium transport inhibitor demonstrated on red blood cells of patients with untreated essential hypertension: correlation of pump activity with potassium permeability.
    Skrabal F, Hamberger L, Cerny E, Schönegger J, Gernhold M.
    Klin Wochenschr; 1985; 63 Suppl 3():117-21. PubMed ID: 2987607
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  • 5. Erythrocyte cation fluxes in essential hypertension of children and adolescents.
    Mongeau JG.
    Int J Pediatr Nephrol; 1985; 6(1):41-6. PubMed ID: 3997372
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  • 8. Altered erythrocyte cation transport related to hypertension or oral contraception.
    Stokes GS, Monaghan JC, Middleton A, Gunn J, Marwood JF.
    Klin Wochenschr; 1985; 63 Suppl 3():42-4. PubMed ID: 3999641
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  • 12. Evidence against a circulating ouabain-like transport inhibitor as a cause of increased red cell sodium in essential hypertension.
    Millar JA, Bramley PM, Paulin JM, Simpson FO.
    J Hypertens Suppl; 1984 Dec; 2(3):S461-3. PubMed ID: 6599699
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  • 16. [Membrane transport of cations in erythrocytes during treatment of essential hypertension with angiotensin-converting enzyme inhibitors].
    Smrcková I, Stríbrná J, Petrásek R, Skibová J, Lánská V.
    Cas Lek Cesk; 1992 May 25; 131(10):294-6. PubMed ID: 1638592
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  • 19. Evaluation of ouabain-insensitive red blood cell cation transport in uremic patients.
    Boero R, Quarello F, Guarena C, Piccoli G.
    Boll Soc Ital Biol Sper; 1985 Feb 28; 61(2):243-8. PubMed ID: 3994843
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  • 20. Coexisting independent sodium-sensitive and sodium-insensitive mechanisms of genetic hypertension in spontaneously hypertensive rats (SHR).
    Wells IC, Blotcky AJ.
    Can J Physiol Pharmacol; 2001 Sep 28; 79(9):779-84. PubMed ID: 11599778
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