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123 related items for PubMed ID: 12369824

  • 1. Antiapoptotic Cdc42 mutants are potent activators of cellular transformation.
    Tu SS, Wu WJ, Yang W, Nolbant P, Hahn K, Cerione RA.
    Biochemistry; 2002 Oct 15; 41(41):12350-8. PubMed ID: 12369824
    [Abstract] [Full Text] [Related]

  • 2. Influencing cellular transformation by modulating the rates of GTP hydrolysis by Cdc42.
    Fidyk N, Wang JB, Cerione RA.
    Biochemistry; 2006 Jun 27; 45(25):7750-62. PubMed ID: 16784226
    [Abstract] [Full Text] [Related]

  • 3. Cdc42 and Ras cooperate to mediate cellular transformation by intersectin-L.
    Wang JB, Wu WJ, Cerione RA.
    J Biol Chem; 2005 Jun 17; 280(24):22883-91. PubMed ID: 15824104
    [Abstract] [Full Text] [Related]

  • 4. The gamma-subunit of the coatomer complex binds Cdc42 to mediate transformation.
    Wu WJ, Erickson JW, Lin R, Cerione RA.
    Nature; 2000 Jun 15; 405(6788):800-4. PubMed ID: 10866202
    [Abstract] [Full Text] [Related]

  • 5. Characterisation of the nucleotide exchange factor ITSN1L: evidence for a kinetic discrimination of GEF-stimulated nucleotide release from Cdc42.
    Kintscher C, Groemping Y.
    J Mol Biol; 2009 Mar 27; 387(2):270-83. PubMed ID: 19356586
    [Abstract] [Full Text] [Related]

  • 6. RhoGDI is required for Cdc42-mediated cellular transformation.
    Lin Q, Fuji RN, Yang W, Cerione RA.
    Curr Biol; 2003 Sep 02; 13(17):1469-79. PubMed ID: 12956948
    [Abstract] [Full Text] [Related]

  • 7. RhoGEF specificity mutants implicate RhoA as a target for Dbs transforming activity.
    Cheng L, Rossman KL, Mahon GM, Worthylake DK, Korus M, Sondek J, Whitehead IP.
    Mol Cell Biol; 2002 Oct 02; 22(19):6895-905. PubMed ID: 12215546
    [Abstract] [Full Text] [Related]

  • 8. Maintenance of CDC42 GDP-bound state by Rho-GDI inhibits MAP kinase activation by the exchange factor Ras-GRF. evidence for Ras-GRF function being inhibited by Cdc42-GDP but unaffected by CDC42-GTP.
    Arozarena I, Matallanas D, Crespo P.
    J Biol Chem; 2001 Jun 15; 276(24):21878-84. PubMed ID: 11285260
    [Abstract] [Full Text] [Related]

  • 9. Understanding the catalytic mechanism of GTPase-activating proteins: demonstration of the importance of switch domain stabilization in the stimulation of GTP hydrolysis.
    Fidyk NJ, Cerione RA.
    Biochemistry; 2002 Dec 31; 41(52):15644-53. PubMed ID: 12501193
    [Abstract] [Full Text] [Related]

  • 10. A novel Cdc42Hs mutant induces cellular transformation.
    Lin R, Bagrodia S, Cerione R, Manor D.
    Curr Biol; 1997 Oct 01; 7(10):794-7. PubMed ID: 9368762
    [Abstract] [Full Text] [Related]

  • 11. Cdc42 is a substrate for caspases and influences Fas-induced apoptosis.
    Tu S, Cerione RA.
    J Biol Chem; 2001 Jun 01; 276(22):19656-63. PubMed ID: 11278572
    [Abstract] [Full Text] [Related]

  • 12. Constitutively active Cdc42 mutant confers growth disadvantage in cell transformation.
    Vanni C, Ottaviano C, Guo F, Puppo M, Varesio L, Zheng Y, Eva A.
    Cell Cycle; 2005 Nov 01; 4(11):1675-82. PubMed ID: 16294011
    [Abstract] [Full Text] [Related]

  • 13. MSE55, a Cdc42 effector protein, induces long cellular extensions in fibroblasts.
    Burbelo PD, Snow DM, Bahou W, Spiegel S.
    Proc Natl Acad Sci U S A; 1999 Aug 03; 96(16):9083-8. PubMed ID: 10430899
    [Abstract] [Full Text] [Related]

  • 14. Active and Inactive Cdc42 Differ in Their Insert Region Conformational Dynamics.
    Haspel N, Jang H, Nussinov R.
    Biophys J; 2021 Jan 19; 120(2):306-318. PubMed ID: 33347888
    [Abstract] [Full Text] [Related]

  • 15. The CDC42-specific inhibitor derived from ACK-1 blocks v-Ha-Ras-induced transformation.
    Nur-E-Kamal MS, Kamal JM, Qureshi MM, Maruta H.
    Oncogene; 1999 Dec 16; 18(54):7787-93. PubMed ID: 10618719
    [Abstract] [Full Text] [Related]

  • 16. Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growth.
    Murphy GA, Solski PA, Jillian SA, Pérez de la Ossa P, D'Eustachio P, Der CJ, Rush MG.
    Oncogene; 1999 Jul 01; 18(26):3831-45. PubMed ID: 10445846
    [Abstract] [Full Text] [Related]

  • 17. Guanine nucleotide induced conformational change of Cdc42 revealed by hydrogen/deuterium exchange mass spectrometry.
    Yang SW, Ting HC, Lo YT, Wu TY, Huang HW, Yang CJ, Chan JF, Chuang MC, Hsu YH.
    Biochim Biophys Acta; 2016 Jan 01; 1864(1):42-51. PubMed ID: 26542736
    [Abstract] [Full Text] [Related]

  • 18. Distinct involvement of cdc42 and RhoA GTPases in actin organization and cell shape in untransformed and Dbl oncogene transformed NIH3T3 cells.
    Olivo C, Vanni C, Mancini P, Silengo L, Torrisi MR, Tarone G, Defilippi P, Eva A.
    Oncogene; 2000 Mar 09; 19(11):1428-36. PubMed ID: 10723134
    [Abstract] [Full Text] [Related]

  • 19. The Rho family GTPase Cdc42 regulates the activation of Ras/MAP kinase by the exchange factor Ras-GRF.
    Arozarena I, Aaronson DS, Matallanas D, Sanz V, Ajenjo N, Tenbaum SP, Teramoto H, Ighishi T, Zabala JC, Gutkind JS, Crespo P.
    J Biol Chem; 2000 Aug 25; 275(34):26441-8. PubMed ID: 10840034
    [Abstract] [Full Text] [Related]

  • 20. R-Ras is regulated by activators and effectors distinct from those that control Ras function.
    Huff SY, Quilliam LA, Cox AD, Der CJ.
    Oncogene; 1997 Jan 16; 14(2):133-43. PubMed ID: 9010215
    [Abstract] [Full Text] [Related]

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