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Journal Abstract Search

137 related items for PubMed ID: 12391846

  • 1. [Transformation with the E1A + cHa-ras oncogenes enhances the trans-repressor function of the Elk-1 transcription factor].
    Usenko TN, Pospelova TV, Pospelov VA.
    Mol Biol (Mosk); 2002; 36(5):825-32. PubMed ID: 12391846
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  • 2. Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter.
    Kukushkin AN, Abramova MV, Svetlikova SB, Darieva ZA, Pospelova TV, Pospelov VA.
    Oncogene; 2002 Jan 24; 21(5):719-30. PubMed ID: 11850800
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  • 3. Transient expression of E1A and Ras oncogenes causes downregulation of c-fos gene transcription in nontransformed REF52 cells.
    Usenko T, Kukushkin A, Pospelova T, Pospelov V.
    Oncogene; 2003 Oct 23; 22(48):7661-6. PubMed ID: 14576829
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  • 6. [Constitutive activity of MAP kinase cascades in REF cells transformed by E1A and cHa-ras oncogenes].
    Svetlikova SB, Abramova MV, Kukushkin AN, Darieva ZA, Pospelova TV, Pospelov VA.
    Tsitologiia; 2001 Oct 23; 43(10):961-8. PubMed ID: 11769129
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  • 7. Deregulation of p53/p21Cip1/Waf1 pathway contributes to polyploidy and apoptosis of E1A+cHa-ras transformed cells after gamma-irradiation.
    Bulavin DV, Tararova ND, Aksenov ND, Pospelov VA, Pospelova TV.
    Oncogene; 1999 Oct 07; 18(41):5611-9. PubMed ID: 10523840
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  • 8. Selective repression of c-fos gene transcription in rat embryo fibroblasts transformed by oncogenes E1A and cHa-ras.
    Abramova MV, Kukushkin AN, Svetlikova SB, Pospelova TV, Pospelov VA.
    Biochem Biophys Res Commun; 2003 Jun 27; 306(2):483-7. PubMed ID: 12804589
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  • 9. E1A + cHa-ras transformed rat embryo fibroblast cells are characterized by high and constitutive DNA binding activities of AP-1 dimers with significantly altered composition.
    Pospelova TV, Medvedev AV, Kukushkin AN, Svetlikova SB, van der Eb AJ, Dorsman JC, Pospelov VA.
    Gene Expr; 1999 Jun 27; 8(1):19-32. PubMed ID: 10543728
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  • 10. [Effect of histone deacetylases inhibitor sodium butyrate (NaB) on transformants E1A + cHa-Ras expressing wild type p53 with supressed transactivation function].
    Bukreeva EI, Aksenov ND, Bardin AA, Pospelov VA, Pospelova TV.
    Tsitologiia; 2009 Jun 27; 51(8):697-705. PubMed ID: 19799355
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  • 11. Phosphorylation of elk-1 by MEK/ERK pathway is necessary for c-fos gene activation during cardiac myocyte hypertrophy.
    Babu GJ, Lalli MJ, Sussman MA, Sadoshima J, Periasamy M.
    J Mol Cell Cardiol; 2000 Aug 27; 32(8):1447-57. PubMed ID: 10900171
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  • 12. [Histone deacetylase inhibitor blocks proliferation of cells transformed with oncogenes E1A and cHa-ras].
    Abramova MV, Svetlikova SB, Aksenov ND, Pospelova TV, Pospelov VA.
    Tsitologiia; 2003 Aug 27; 45(11):1100-8. PubMed ID: 14989149
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  • 14. [Rat embryo fibroblasts transformed by complementation with oncogenes E1A+E1B-19 and E1A+cHa-ras differ in the ability to realize the G1/S block in serum free media].
    Brichkina AI, Tararova ND, Aksenov ND, Pospelov VA, Pospelova TV.
    Tsitologiia; 2001 Aug 27; 43(11):1024-30. PubMed ID: 11840777
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  • 15. Activation of Elk-1, an Ets transcription factor, by glucose and EGF treatment of insulinoma cells.
    Bernal-Mizrachi E, Wen W, Srinivasan S, Klenk A, Cohen D, Permutt MA.
    Am J Physiol Endocrinol Metab; 2001 Dec 27; 281(6):E1286-99. PubMed ID: 11701445
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  • 17. [Protein phosphatase MKP-1 participates in c-fos gene derepression under the action of stress factors on fibroblasts transformed with E1A and cHA-ras oncogenes].
    Kukushkin AN, Svetlikova SB, Pospelov VA.
    Tsitologiia; 2013 Dec 27; 55(12):861-7. PubMed ID: 25474904
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  • 18. Temporal recruitment of the mSin3A-histone deacetylase corepressor complex to the ETS domain transcription factor Elk-1.
    Yang SH, Vickers E, Brehm A, Kouzarides T, Sharrocks AD.
    Mol Cell Biol; 2001 Apr 27; 21(8):2802-14. PubMed ID: 11283259
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  • 19. G1/S arrest induced by histone deacetylase inhibitor sodium butyrate in E1A + Ras-transformed cells is mediated through down-regulation of E2F activity and stabilization of beta-catenin.
    Abramova MV, Pospelova TV, Nikulenkov FP, Hollander CM, Fornace AJ, Pospelov VA.
    J Biol Chem; 2006 Jul 28; 281(30):21040-21051. PubMed ID: 16717102
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  • 20. N-Methyl-D-aspartate receptors and p38 mitogen-activated protein kinase are required for cAMP-dependent cyclase response element binding protein and Elk-1 phosphorylation in the striatum.
    Choe ES, McGinty JF.
    Neuroscience; 2000 Jul 28; 101(3):607-17. PubMed ID: 11113310
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