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307 related items for PubMed ID: 12414630

  • 1. Mesothelial cell transformation requires increased AP-1 binding activity and ERK-dependent Fra-1 expression.
    Ramos-Nino ME, Timblin CR, Mossman BT.
    Cancer Res; 2002 Nov 01; 62(21):6065-9. PubMed ID: 12414630
    [Abstract] [Full Text] [Related]

  • 2. Patterns of c-fos and c-jun proto-oncogene expression, apoptosis, and proliferation in rat pleural mesothelial cells exposed to erionite or asbestos fibers.
    Timblin CR, Guthrie GD, Janssen YW, Walsh ES, Vacek P, Mossman BT.
    Toxicol Appl Pharmacol; 1998 Jul 01; 151(1):88-97. PubMed ID: 9705890
    [Abstract] [Full Text] [Related]

  • 3. Silica-induced activation of c-Jun-NH2-terminal amino kinases, protracted expression of the activator protein-1 proto-oncogene, fra-1, and S-phase alterations are mediated via oxidative stress.
    Shukla A, Timblin CR, Hubbard AK, Bravman J, Mossman BT.
    Cancer Res; 2001 Mar 01; 61(5):1791-5. PubMed ID: 11280724
    [Abstract] [Full Text] [Related]

  • 4. High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model.
    Zoumpourlis V, Papassava P, Linardopoulos S, Gillespie D, Balmain A, Pintzas A.
    Oncogene; 2000 Aug 17; 19(35):4011-21. PubMed ID: 10962557
    [Abstract] [Full Text] [Related]

  • 5. Expression of dominant negative Erk2 inhibits AP-1 transactivation and neoplastic transformation.
    Watts RG, Huang C, Young MR, Li JJ, Dong Z, Pennie WD, Colburn NH.
    Oncogene; 1998 Dec 31; 17(26):3493-8. PubMed ID: 10030673
    [Abstract] [Full Text] [Related]

  • 6. Asbestos causes stimulation of the extracellular signal-regulated kinase 1 mitogen-activated protein kinase cascade after phosphorylation of the epidermal growth factor receptor.
    Zanella CL, Posada J, Tritton TR, Mossman BT.
    Cancer Res; 1996 Dec 01; 56(23):5334-8. PubMed ID: 8968079
    [Abstract] [Full Text] [Related]

  • 7. Mitogen activated protein kinase-dependent activation of c-Jun and c-Fos is required for neuronal differentiation but not for growth and stress response in PC12 cells.
    Eriksson M, Taskinen M, Leppä S.
    J Cell Physiol; 2007 Feb 01; 210(2):538-48. PubMed ID: 17111371
    [Abstract] [Full Text] [Related]

  • 8. Phosphorylation and high level expression of Fra-2 in v-src transformed cells: a pathway of activation of endogenous AP-1.
    Murakami M, Sonobe MH, Ui M, Kabuyama Y, Watanabe H, Wada T, Handa H, Iba H.
    Oncogene; 1997 May 22; 14(20):2435-44. PubMed ID: 9188858
    [Abstract] [Full Text] [Related]

  • 9. Age-related differences in MAP kinase activity in VSMC in response to glucose or TNF-alpha.
    Li M, Mossman BT, Kolpa E, Timblin CR, Shukla A, Taatjes DJ, Fukagawa NK.
    J Cell Physiol; 2003 Dec 22; 197(3):418-25. PubMed ID: 14566971
    [Abstract] [Full Text] [Related]

  • 10. Elevated ERK-MAP kinase activity protects the FOS family member FRA-1 against proteasomal degradation in colon carcinoma cells.
    Vial E, Marshall CJ.
    J Cell Sci; 2003 Dec 15; 116(Pt 24):4957-63. PubMed ID: 14625389
    [Abstract] [Full Text] [Related]

  • 11. AP-1 regulates alpha2beta1 integrin expression by ERK-dependent signals during megakaryocytic differentiation of K562 cells.
    Eriksson M, Arminen L, Karjalainen-Lindsberg ML, Leppä S.
    Exp Cell Res; 2005 Mar 10; 304(1):175-86. PubMed ID: 15707584
    [Abstract] [Full Text] [Related]

  • 12. PACAP and C2-ceramide generate different AP-1 complexes through a MAP-kinase-dependent pathway: involvement of c-Fos in PACAP-induced Bcl-2 expression.
    Aubert N, Falluel-Morel A, Vaudry D, Xifro X, Rodriguez-Alvarez J, Fisch C, de Jouffrey S, Lebigot JF, Fournier A, Vaudry H, Gonzalez BJ.
    J Neurochem; 2006 Nov 10; 99(4):1237-50. PubMed ID: 17026529
    [Abstract] [Full Text] [Related]

  • 13. Microarray analysis and RNA silencing link fra-1 to cd44 and c-met expression in mesothelioma.
    Ramos-Nino ME, Scapoli L, Martinelli M, Land S, Mossman BT.
    Cancer Res; 2003 Jul 01; 63(13):3539-45. PubMed ID: 12839939
    [Abstract] [Full Text] [Related]

  • 14. Transition from SCLC to NSCLC phenotype is accompanied by an increased TRE-binding activity and recruitment of specific AP-1 proteins.
    Risse-Hackl G, Adamkiewicz J, Wimmel A, Schuermann M.
    Oncogene; 1998 Jun 11; 16(23):3057-68. PubMed ID: 9662339
    [Abstract] [Full Text] [Related]

  • 15. Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence.
    Unni E, Sun S, Nan B, McPhaul MJ, Cheskis B, Mancini MA, Marcelli M.
    Cancer Res; 2004 Oct 01; 64(19):7156-68. PubMed ID: 15466214
    [Abstract] [Full Text] [Related]

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  • 17. The gamma-glutamylcysteine synthetase and glutathione regulate asbestos-induced expression of activator protein-1 family members and activity.
    Shukla A, Flanders T, Lounsbury KM, Mossman BT.
    Cancer Res; 2004 Nov 01; 64(21):7780-6. PubMed ID: 15520183
    [Abstract] [Full Text] [Related]

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  • 19. Celecoxib inhibits phorbol ester-induced expression of COX-2 and activation of AP-1 and p38 MAP kinase in mouse skin.
    Chun KS, Kim SH, Song YS, Surh YJ.
    Carcinogenesis; 2004 May 01; 25(5):713-22. PubMed ID: 14729583
    [Abstract] [Full Text] [Related]

  • 20. AP-1 transrepressing retinoic acid does not deplete coactivators or AP-1 monomers but may target specific Jun or Fos containing dimers.
    Suzukawa K, Colburn NH.
    Oncogene; 2002 Mar 28; 21(14):2181-90. PubMed ID: 11948401
    [Abstract] [Full Text] [Related]

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