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162 related items for PubMed ID: 12416391

  • 1. How do neurons degenerate in prion diseases or transmissible spongiform encephalopathies (TSEs): neuronal autophagy revisited.
    Liberski PP, Gajdusek DC, Brown P.
    Acta Neurobiol Exp (Wars); 2002; 62(3):141-7. PubMed ID: 12416391
    [Abstract] [Full Text] [Related]

  • 2. Neuronal cell death in transmissible spongiform encephalopathies (prion diseases) revisited: from apoptosis to autophagy.
    Liberski PP, Sikorska B, Bratosiewicz-Wasik J, Gajdusek DC, Brown P.
    Int J Biochem Cell Biol; 2004 Dec; 36(12):2473-90. PubMed ID: 15325586
    [Abstract] [Full Text] [Related]

  • 3. Cell death and autophagy in prion diseases (transmissible spongiform encephalopathies).
    Liberski PP, Brown DR, Sikorska B, Caughey B, Brown P.
    Folia Neuropathol; 2008 Dec; 46(1):1-25. PubMed ID: 18368623
    [Abstract] [Full Text] [Related]

  • 4. Neuronal autophagy and aggresomes constitute a consistent part of neurodegeneration in experimental scrapie.
    Sikorska B, Liberski PP, Brown P.
    Folia Neuropathol; 2007 Dec; 45(4):170-8. PubMed ID: 18176890
    [Abstract] [Full Text] [Related]

  • 5. Echigo-1: a panencephalopathic strain of Creutzfeldt-Jakob disease. II. Ultrastructural studies in hamsters.
    Sikorska B, Hainfellner JA, Mori S, Bratosiewiczi J, Liberski PP, Budka H.
    Folia Neuropathol; 2004 Dec; 42 Suppl B():167-75. PubMed ID: 16903151
    [Abstract] [Full Text] [Related]

  • 6. How do neurons degenerate in transmissible spongiform encephalopathies?
    Liberski PP.
    Mol Chem Neuropathol; 1996 Dec; 28(1-3):245-9. PubMed ID: 8871966
    [Abstract] [Full Text] [Related]

  • 7. A special report I. Prion protein (PrP)--amyloid plaques in the transmissible spongiform encephalopathies, or prion diseases revisited.
    Liberski PP, Bratosiewicz J, Waliś A, Kordek R, Jeffrey M, Brown P.
    Folia Neuropathol; 2001 Dec; 39(4):217-35. PubMed ID: 11928893
    [Abstract] [Full Text] [Related]

  • 8. Ultrastructural changes in the optic nerves of rodents with experimental Creutzfeldt-Jakob Disease (CJD), Gerstmann-Sträussler-Scheinker disease (GSS) or scrapie.
    Waliś A, Bratosiewicz J, Sikorska B, Brown P, Gajdusek DC, Liberski PP.
    J Comp Pathol; 2003 Dec; 129(2-3):213-25. PubMed ID: 12921728
    [Abstract] [Full Text] [Related]

  • 9. Ultrastructural studies of experimental scrapie and Creutzfeldt-Jakob disease in hamsters. I. Alterations of myelinated axons.
    Liberski PP, Bratosiewicz-Wasik J, Gajdusek DC, Brown P.
    Acta Neurobiol Exp (Wars); 2002 Dec; 62(3):121-9. PubMed ID: 12416389
    [Abstract] [Full Text] [Related]

  • 10. Neuronal autophagic vacuoles in experimental scrapie and Creutzfeldt-Jakob disease.
    Liberski PP, Yanagihara R, Gibbs CJ, Gajdusek DC.
    Acta Neuropathol; 1992 Dec; 83(2):134-9. PubMed ID: 1557945
    [Abstract] [Full Text] [Related]

  • 11. Ultrastructural studies of experimental scrapie and Creutzfeldt-Jakob disease in hamsters. II. Astrocytic and macrophage reaction towards axonal destruction.
    Liberski PP, Bratosiewicz-Wasik J, Gajdusek DC, Brown P.
    Acta Neurobiol Exp (Wars); 2002 Dec; 62(3):131-9. PubMed ID: 12416390
    [Abstract] [Full Text] [Related]

  • 12. Tubulovesicular structures are present in brains of hamsters infected with the Echigo-1 strain of Creutzfeldt-Jakob disease agent.
    Liberski P.
    Acta Neurobiol Exp (Wars); 2008 Dec; 68(1):39-42. PubMed ID: 18389013
    [Abstract] [Full Text] [Related]

  • 13. Scrg1 is induced in TSE and brain injuries, and associated with autophagy.
    Dron M, Bailly Y, Beringue V, Haeberlé AM, Griffond B, Risold PY, Tovey MG, Laude H, Dandoy-Dron F.
    Eur J Neurosci; 2005 Jul; 22(1):133-46. PubMed ID: 16029203
    [Abstract] [Full Text] [Related]

  • 14. Axonal changes in experimental prion diseases recapitulate those following constriction of postganglionic branches of the superior cervical ganglion: a comparison 40 years later.
    Liberski PP.
    Prion; 2019 Jan; 13(1):83-93. PubMed ID: 30966865
    [Abstract] [Full Text] [Related]

  • 15. Human transmissible spongiform encephalopathies: historic view.
    Asher DM, Gregori L.
    Handb Clin Neurol; 2018 Jan; 153():1-17. PubMed ID: 29887130
    [Abstract] [Full Text] [Related]

  • 16. Autophagy is a part of ultrastructural synaptic pathology in Creutzfeldt-Jakob disease: a brain biopsy study.
    Sikorska B, Liberski PP, Giraud P, Kopp N, Brown P.
    Int J Biochem Cell Biol; 2004 Dec; 36(12):2563-73. PubMed ID: 15325593
    [Abstract] [Full Text] [Related]

  • 17. [Creutzfeldt-Jakob disease and other human transmissible spongiform encephalopathies. Part II].
    Zaborowski A.
    Psychiatr Pol; 2004 Dec; 38(2):297-309. PubMed ID: 15307294
    [Abstract] [Full Text] [Related]

  • 18. Reduction of protein kinase MARK4 in the brains of experimental scrapie rodents and human prion disease correlates with deposits of PrP(Sc).
    Gong HS, Guo Y, Tian C, Xie WL, Shi Q, Zhang J, Xu Y, Wang SB, Zhang BY, Chen C, Liu Y, Dong XP.
    Int J Mol Med; 2012 Sep; 30(3):569-78. PubMed ID: 22692785
    [Abstract] [Full Text] [Related]

  • 19. Neuropathological diagnostic criteria for Creutzfeldt-Jakob disease (CJD) and other human spongiform encephalopathies (prion diseases).
    Budka H, Aguzzi A, Brown P, Brucher JM, Bugiani O, Gullotta F, Haltia M, Hauw JJ, Ironside JW, Jellinger K.
    Brain Pathol; 1995 Oct; 5(4):459-66. PubMed ID: 8974629
    [Abstract] [Full Text] [Related]

  • 20. Disease-specific particles without prion protein in prion diseases - phenomenon or epiphenomenon?
    Liberski PP, Brown P.
    Neuropathol Appl Neurobiol; 2007 Aug; 33(4):395-7. PubMed ID: 17617871
    [Abstract] [Full Text] [Related]

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