These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
278 related items for PubMed ID: 12447686
1. A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA base damage. Osterod M, Larsen E, Le Page F, Hengstler JG, Van Der Horst GT, Boiteux S, Klungland A, Epe B. Oncogene; 2002 Nov 28; 21(54):8232-9. PubMed ID: 12447686 [Abstract] [Full Text] [Related]
2. Deficiency of the Cockayne syndrome B (CSB) gene aggravates the genomic instability caused by endogenous oxidative DNA base damage in mice. Trapp C, Reite K, Klungland A, Epe B. Oncogene; 2007 Jun 07; 26(27):4044-8. PubMed ID: 17213818 [Abstract] [Full Text] [Related]
3. The basal levels of 8-oxoG and other oxidative modifications in intact mitochondrial DNA are low even in repair-deficient (Ogg1(-/-)/Csb(-/-)) mice. Trapp C, McCullough AK, Epe B. Mutat Res; 2007 Dec 01; 625(1-2):155-63. PubMed ID: 17675188 [Abstract] [Full Text] [Related]
4. The peroxisome proliferator WY-14,643 promotes hepatocarcinogenesis caused by endogenously generated oxidative DNA base modifications in repair-deficient Csbm/m/Ogg1-/- mice. Trapp C, Schwarz M, Epe B. Cancer Res; 2007 Jun 01; 67(11):5156-61. PubMed ID: 17545594 [Abstract] [Full Text] [Related]
7. Mitochondrial repair of 8-oxoguanine is deficient in Cockayne syndrome group B. Stevnsner T, Nyaga S, de Souza-Pinto NC, van der Horst GT, Gorgels TG, Hogue BA, Thorslund T, Bohr VA. Oncogene; 2002 Dec 12; 21(57):8675-82. PubMed ID: 12483520 [Abstract] [Full Text] [Related]
11. Global genome repair of 8-oxoG in hamster cells requires a functional CSB gene product. Sunesen M, Stevnsner T, Brosh RM, Dianov GL, Bohr VA. Oncogene; 2002 May 16; 21(22):3571-8. PubMed ID: 12032859 [Abstract] [Full Text] [Related]
12. Spontaneous mutation, oxidative DNA damage, and the roles of base and nucleotide excision repair in the yeast Saccharomyces cerevisiae. Scott AD, Neishabury M, Jones DH, Reed SH, Boiteux S, Waters R. Yeast; 1999 Feb 16; 15(3):205-18. PubMed ID: 10077187 [Abstract] [Full Text] [Related]
14. The relationship between benzo[a]pyrene-induced mutagenesis and carcinogenesis in repair-deficient Cockayne syndrome group B mice. Wijnhoven SW, Kool HJ, van Oostrom CT, Beems RB, Mullenders LH, van Zeeland AA, van der Horst GT, Vrieling H, van Steeg H. Cancer Res; 2000 Oct 15; 60(20):5681-7. PubMed ID: 11059760 [Abstract] [Full Text] [Related]
15. Age-related and tissue-specific accumulation of oxidative DNA base damage in 7,8-dihydro-8-oxoguanine-DNA glycosylase (Ogg1) deficient mice. Osterod M, Hollenbach S, Hengstler JG, Barnes DE, Lindahl T, Epe B. Carcinogenesis; 2001 Sep 15; 22(9):1459-63. PubMed ID: 11532868 [Abstract] [Full Text] [Related]
16. 8-Oxoguanine DNA glycosylase (Ogg1) causes a transcriptional inactivation of damaged DNA in the absence of functional Cockayne syndrome B (Csb) protein. Khobta A, Kitsera N, Speckmann B, Epe B. DNA Repair (Amst); 2009 Mar 01; 8(3):309-17. PubMed ID: 19061977 [Abstract] [Full Text] [Related]
18. Functional cooperation of Ogg1 and Mutyh in preventing G: C-->T: a transversions in mice. Isogawa A. Fukuoka Igaku Zasshi; 2004 Jan 01; 95(1):17-30. PubMed ID: 15031996 [Abstract] [Full Text] [Related]