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Journal Abstract Search

288 related items for PubMed ID: 12483520

  • 1. Mitochondrial repair of 8-oxoguanine is deficient in Cockayne syndrome group B.
    Stevnsner T, Nyaga S, de Souza-Pinto NC, van der Horst GT, Gorgels TG, Hogue BA, Thorslund T, Bohr VA.
    Oncogene; 2002 Dec 12; 21(57):8675-82. PubMed ID: 12483520
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  • 5. The basal levels of 8-oxoG and other oxidative modifications in intact mitochondrial DNA are low even in repair-deficient (Ogg1(-/-)/Csb(-/-)) mice.
    Trapp C, McCullough AK, Epe B.
    Mutat Res; 2007 Dec 01; 625(1-2):155-63. PubMed ID: 17675188
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  • 6. Repair of 8-oxoguanine in DNA is deficient in Cockayne syndrome group B cells.
    Dianov G, Bischoff C, Sunesen M, Bohr VA.
    Nucleic Acids Res; 1999 Mar 01; 27(5):1365-8. PubMed ID: 9973627
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  • 7. Transcription activities at 8-oxoG lesions in DNA.
    Larsen E, Kwon K, Coin F, Egly JM, Klungland A.
    DNA Repair (Amst); 2004 Nov 02; 3(11):1457-68. PubMed ID: 15380101
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  • 9. The peroxisome proliferator WY-14,643 promotes hepatocarcinogenesis caused by endogenously generated oxidative DNA base modifications in repair-deficient Csbm/m/Ogg1-/- mice.
    Trapp C, Schwarz M, Epe B.
    Cancer Res; 2007 Jun 01; 67(11):5156-61. PubMed ID: 17545594
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  • 10. Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.
    Selzer RR, Nyaga S, Tuo J, May A, Muftuoglu M, Christiansen M, Citterio E, Brosh RM, Bohr VA.
    Nucleic Acids Res; 2002 Feb 01; 30(3):782-93. PubMed ID: 11809892
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  • 11. Functional crosstalk between hOgg1 and the helicase domain of Cockayne syndrome group B protein.
    Tuo J, Chen C, Zeng X, Christiansen M, Bohr VA.
    DNA Repair (Amst); 2002 Nov 03; 1(11):913-27. PubMed ID: 12531019
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  • 12. Defective repair of 8-hydroxyguanine in mitochondria of MCF-7 and MDA-MB-468 human breast cancer cell lines.
    Mambo E, Nyaga SG, Bohr VA, Evans MK.
    Cancer Res; 2002 Mar 01; 62(5):1349-55. PubMed ID: 11888904
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  • 13. Early host cell reactivation of an oxidatively damaged adenovirus-encoded reporter gene requires the Cockayne syndrome proteins CSA and CSB.
    Leach DM, Rainbow AJ.
    Mutagenesis; 2011 Mar 01; 26(2):315-21. PubMed ID: 21059811
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  • 14. Accumulation of mitochondrial DNA damage and bioenergetic dysfunction in CSB defective cells.
    Osenbroch PØ, Auk-Emblem P, Halsne R, Strand J, Forstrøm RJ, van der Pluijm I, Eide L.
    FEBS J; 2009 May 01; 276(10):2811-21. PubMed ID: 19389114
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  • 15. The transcriptional response after oxidative stress is defective in Cockayne syndrome group B cells.
    Kyng KJ, May A, Brosh RM, Cheng WH, Chen C, Becker KG, Bohr VA.
    Oncogene; 2003 Feb 27; 22(8):1135-49. PubMed ID: 12606941
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  • 18. Deficiency of the Cockayne syndrome B (CSB) gene aggravates the genomic instability caused by endogenous oxidative DNA base damage in mice.
    Trapp C, Reite K, Klungland A, Epe B.
    Oncogene; 2007 Jun 07; 26(27):4044-8. PubMed ID: 17213818
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  • 19. Opposite base-dependent reactions of a human base excision repair enzyme on DNA containing 7,8-dihydro-8-oxoguanine and abasic sites.
    Bjorâs M, Luna L, Johnsen B, Hoff E, Haug T, Rognes T, Seeberg E.
    EMBO J; 1997 Oct 15; 16(20):6314-22. PubMed ID: 9321410
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  • 20. Primary fibroblasts of Cockayne syndrome patients are defective in cellular repair of 8-hydroxyguanine and 8-hydroxyadenine resulting from oxidative stress.
    Tuo J, Jaruga P, Rodriguez H, Bohr VA, Dizdaroglu M.
    FASEB J; 2003 Apr 15; 17(6):668-74. PubMed ID: 12665480
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