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1907 related items for PubMed ID: 12543789
1. Insulin-like growth factor I-mediated protection from rapamycin-induced apoptosis is independent of Ras-Erk1-Erk2 and phosphatidylinositol 3'-kinase-Akt signaling pathways. Thimmaiah KN, Easton J, Huang S, Veverka KA, Germain GS, Harwood FC, Houghton PJ. Cancer Res; 2003 Jan 15; 63(2):364-74. PubMed ID: 12543789 [Abstract] [Full Text] [Related]
2. Protection from rapamycin-induced apoptosis by insulin-like growth factor-I is partially dependent on protein kinase C signaling. Thimmaiah KN, Easton JB, Houghton PJ. Cancer Res; 2010 Mar 01; 70(5):2000-9. PubMed ID: 20179209 [Abstract] [Full Text] [Related]
3. In cardiomyocyte hypoxia, insulin-like growth factor-I-induced antiapoptotic signaling requires phosphatidylinositol-3-OH-kinase-dependent and mitogen-activated protein kinase-dependent activation of the transcription factor cAMP response element-binding protein. Mehrhof FB, Müller FU, Bergmann MW, Li P, Wang Y, Schmitz W, Dietz R, von Harsdorf R. Circulation; 2001 Oct 23; 104(17):2088-94. PubMed ID: 11673351 [Abstract] [Full Text] [Related]
4. Stimulation of insulin-like growth factor (IGF) binding protein-3 synthesis by IGF-I and transforming growth factor-alpha is mediated by both phosphatidylinositol-3 kinase and mitogen-activated protein kinase pathways in mammary epithelial cells. Sivaprasad U, Fleming J, Verma PS, Hogan KA, Desury G, Cohick WS. Endocrinology; 2004 Sep 23; 145(9):4213-21. PubMed ID: 15192040 [Abstract] [Full Text] [Related]
5. The lethal effects of pharmacological cyclin-dependent kinase inhibitors in human leukemia cells proceed through a phosphatidylinositol 3-kinase/Akt-dependent process. Yu C, Rahmani M, Dai Y, Conrad D, Krystal G, Dent P, Grant S. Cancer Res; 2003 Apr 15; 63(8):1822-33. PubMed ID: 12702569 [Abstract] [Full Text] [Related]
6. The cell survival signal Akt is differentially activated by PDGF-BB, EGF, and FGF-2 in osteoblastic cells. Chaudhary LR, Hruska KA. J Cell Biochem; 2001 Mar 26; 81(2):304-11. PubMed ID: 11241670 [Abstract] [Full Text] [Related]
7. Differential insulin-like growth factor I receptor signaling and function in estrogen receptor (ER)-positive MCF-7 and ER-negative MDA-MB-231 breast cancer cells. Bartucci M, Morelli C, Mauro L, Andò S, Surmacz E. Cancer Res; 2001 Sep 15; 61(18):6747-54. PubMed ID: 11559546 [Abstract] [Full Text] [Related]
10. Insulin-like growth factor-I-dependent signal transduction pathways leading to the induction of cell growth and differentiation of human neuroblastoma cell line SH-SY5Y: the roles of MAP kinase pathway and PI 3-kinase pathway. Kurihara S, Hakuno F, Takahashi S. Endocr J; 2000 Dec 15; 47(6):739-51. PubMed ID: 11228049 [Abstract] [Full Text] [Related]
11. Cell cycle progression and activation of Akt kinase are required for insulin-like growth factor I-mediated suppression of apoptosis in granulosa cells. Hu CL, Cowan RG, Harman RM, Quirk SM. Mol Endocrinol; 2004 Feb 15; 18(2):326-38. PubMed ID: 14593075 [Abstract] [Full Text] [Related]
12. The chimeric protein tyrosine kinase ETV6-NTRK3 requires both Ras-Erk1/2 and PI3-kinase-Akt signaling for fibroblast transformation. Tognon C, Garnett M, Kenward E, Kay R, Morrison K, Sorensen PH. Cancer Res; 2001 Dec 15; 61(24):8909-16. PubMed ID: 11751416 [Abstract] [Full Text] [Related]
13. Enhancement of survival by LPA via Erk1/Erk2 and PI 3-kinase/Akt pathways in a murine hepatocyte cell line. Sautin YY, Crawford JM, Svetlov SI. Am J Physiol Cell Physiol; 2001 Dec 15; 281(6):C2010-9. PubMed ID: 11698260 [Abstract] [Full Text] [Related]
15. Downstream effectors of oncogenic ras in multiple myeloma cells. Hu L, Shi Y, Hsu JH, Gera J, Van Ness B, Lichtenstein A. Blood; 2003 Apr 15; 101(8):3126-35. PubMed ID: 12515720 [Abstract] [Full Text] [Related]
16. Glucose-potentiated chemotaxis in human vascular smooth muscle is dependent on cross-talk between the PI3K and MAPK signaling pathways. Campbell M, Allen WE, Sawyer C, Vanhaesebroeck B, Trimble ER. Circ Res; 2004 Aug 20; 95(4):380-8. PubMed ID: 15242975 [Abstract] [Full Text] [Related]
17. Concomitant activation of the PI3K-Akt and the Ras-ERK signaling pathways is essential for transformation by the V-SEA tyrosine kinase oncogene. Agazie Y, Ischenko I, Hayman M. Oncogene; 2002 Jan 24; 21(5):697-707. PubMed ID: 11850798 [Abstract] [Full Text] [Related]
18. Differential regulation of the phosphoinositide 3-kinase and MAP kinase pathways by hepatocyte growth factor vs. insulin-like growth factor-I in myogenic cells. Halevy O, Cantley LC. Exp Cell Res; 2004 Jul 01; 297(1):224-34. PubMed ID: 15194438 [Abstract] [Full Text] [Related]
19. Synthetic lethal interaction between PI3K/Akt/mTOR and Ras/MEK/ERK pathway inhibition in rhabdomyosarcoma. Guenther MK, Graab U, Fulda S. Cancer Lett; 2013 Sep 01; 337(2):200-9. PubMed ID: 23684925 [Abstract] [Full Text] [Related]
20. Up-regulation of urokinase-type plasminogen activator by insulin-like growth factor-I depends upon phosphatidylinositol-3 kinase and mitogen-activated protein kinase kinase. Dunn SE, Torres JV, Oh JS, Cykert DM, Barrett JC. Cancer Res; 2001 Feb 15; 61(4):1367-74. PubMed ID: 11245436 [Abstract] [Full Text] [Related] Page: [Next] [New Search]