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Journal Abstract Search

126 related items for PubMed ID: 12548020

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  • 5. p21(WAF1/CIP1) is upregulated by the geranylgeranyltransferase I inhibitor GGTI-298 through a transforming growth factor beta- and Sp1-responsive element: involvement of the small GTPase rhoA.
    Adnane J, Bizouarn FA, Qian Y, Hamilton AD, Sebti SM.
    Mol Cell Biol; 1998 Dec; 18(12):6962-70. PubMed ID: 9819384
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  • 7. RhoA prenylation is required for promotion of cell growth and transformation and cytoskeleton organization but not for induction of serum response element transcription.
    Allal C, Favre G, Couderc B, Salicio S, Sixou S, Hamilton AD, Sebti SM, Lajoie-Mazenc I, Pradines A.
    J Biol Chem; 2000 Oct 06; 275(40):31001-8. PubMed ID: 10896672
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  • 8. RhoA biological activity is dependent on prenylation but independent of specific isoprenoid modification.
    Solski PA, Helms W, Keely PJ, Su L, Der CJ.
    Cell Growth Differ; 2002 Aug 06; 13(8):363-73. PubMed ID: 12193475
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  • 9. Geranylgeranyltransferase I inhibitors target RalB to inhibit anchorage-dependent growth and induce apoptosis and RalA to inhibit anchorage-independent growth.
    Falsetti SC, Wang DA, Peng H, Carrico D, Cox AD, Der CJ, Hamilton AD, Sebti SM.
    Mol Cell Biol; 2007 Nov 06; 27(22):8003-14. PubMed ID: 17875936
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  • 10. Protein geranylgeranylation, not farnesylation, is required for the G1 to S phase transition in mouse fibroblasts.
    Vogt A, Qian Y, McGuire TF, Hamilton AD, Sebti SM.
    Oncogene; 1996 Nov 07; 13(9):1991-9. PubMed ID: 8934546
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  • 11. Geranylgeranylated, but not farnesylated, RhoB suppresses Ras transformation of NIH-3T3 cells.
    Mazières J, Tillement V, Allal C, Clanet C, Bobin L, Chen Z, Sebti SM, Favre G, Pradines A.
    Exp Cell Res; 2005 Apr 01; 304(2):354-64. PubMed ID: 15748883
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  • 13. Both farnesyltransferase and geranylgeranyltransferase I inhibitors are required for inhibition of oncogenic K-Ras prenylation but each alone is sufficient to suppress human tumor growth in nude mouse xenografts.
    Sun J, Qian Y, Hamilton AD, Sebti SM.
    Oncogene; 1998 Mar 01; 16(11):1467-73. PubMed ID: 9525745
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  • 14. Inhibition of GGTase-I and FTase disrupts cytoskeletal organization of human PC-3 prostate cancer cells.
    Virtanen SS, Sandholm J, Yegutkin G, Kalervo Väänänen H, Härkönen PL.
    Cell Biol Int; 2010 Aug 01; 34(8):815-26. PubMed ID: 20446922
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  • 16. Inhibition of geranylgeranyltransferase inhibits bronchial smooth muscle hyperresponsiveness in mice.
    Chiba Y, Sato S, Hanazaki M, Sakai H, Misawa M.
    Am J Physiol Lung Cell Mol Physiol; 2009 Nov 01; 297(5):L984-91. PubMed ID: 19717551
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  • 17. The geranylgeranyltransferase I inhibitor GGTI-298 induces hypophosphorylation of retinoblastoma and partner switching of cyclin-dependent kinase inhibitors. A potential mechanism for GGTI-298 antitumor activity.
    Sun J, Qian Y, Chen Z, Marfurt J, Hamilton AD, Sebti SM.
    J Biol Chem; 1999 Mar 12; 274(11):6930-4. PubMed ID: 10066746
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  • 18. Geranylgeranylated RhoB mediates suppression of human tumor cell growth by farnesyltransferase inhibitors.
    Du W, Prendergast GC.
    Cancer Res; 1999 Nov 01; 59(21):5492-6. PubMed ID: 10554025
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  • 19. Farnesyltransferase inhibitors disrupt EGF receptor traffic through modulation of the RhoB GTPase.
    Wherlock M, Gampel A, Futter C, Mellor H.
    J Cell Sci; 2004 Jul 01; 117(Pt 15):3221-31. PubMed ID: 15226397
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  • 20. Involvement of RhoA and RalB in geranylgeranyltransferase I inhibitor-mediated inhibition of proliferation and migration of human oral squamous cell carcinoma cells.
    Hamada M, Miki T, Iwai S, Shimizu H, Yura Y.
    Cancer Chemother Pharmacol; 2011 Sep 01; 68(3):559-69. PubMed ID: 21107573
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