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Journal Abstract Search

302 related items for PubMed ID: 12606941

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  • 3. Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.
    Selzer RR, Nyaga S, Tuo J, May A, Muftuoglu M, Christiansen M, Citterio E, Brosh RM, Bohr VA.
    Nucleic Acids Res; 2002 Feb 01; 30(3):782-93. PubMed ID: 11809892
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  • 4. Host cell reactivation of plasmids containing oxidative DNA lesions is defective in Cockayne syndrome but normal in UV-sensitive syndrome fibroblasts.
    Spivak G, Hanawalt PC.
    DNA Repair (Amst); 2006 Jan 05; 5(1):13-22. PubMed ID: 16129663
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  • 5. Cockayne syndrome group B (CSB) protein: at the crossroads of transcriptional networks.
    Vélez-Cruz R, Egly JM.
    Mech Ageing Dev; 2013 Jan 05; 134(5-6):234-42. PubMed ID: 23562425
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  • 7. Primary fibroblasts of Cockayne syndrome patients are defective in cellular repair of 8-hydroxyguanine and 8-hydroxyadenine resulting from oxidative stress.
    Tuo J, Jaruga P, Rodriguez H, Bohr VA, Dizdaroglu M.
    FASEB J; 2003 Apr 05; 17(6):668-74. PubMed ID: 12665480
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  • 8. The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair.
    Brosh RM, Balajee AS, Selzer RR, Sunesen M, Proietti De Santis L, Bohr VA.
    Mol Biol Cell; 1999 Nov 05; 10(11):3583-94. PubMed ID: 10564257
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  • 10. The Cockayne Syndrome group B gene product is involved in general genome base excision repair of 8-hydroxyguanine in DNA.
    Tuo J, Müftüoglu M, Chen C, Jaruga P, Selzer RR, Brosh RM, Rodriguez H, Dizdaroglu M, Bohr VA.
    J Biol Chem; 2001 Dec 07; 276(49):45772-9. PubMed ID: 11581270
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  • 11. The Cockayne syndrome B protein, involved in transcription-coupled DNA repair, resides in an RNA polymerase II-containing complex.
    van Gool AJ, Citterio E, Rademakers S, van Os R, Vermeulen W, Constantinou A, Egly JM, Bootsma D, Hoeijmakers JH.
    EMBO J; 1997 Oct 01; 16(19):5955-65. PubMed ID: 9312053
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  • 12. Impaired jun-NH2-terminal kinase activation by ultraviolet irradiation in fibroblasts of patients with Cockayne syndrome complementation group B.
    Dhar V, Adler V, Lehmann A, Ronai Z.
    Cell Growth Differ; 1996 Jun 01; 7(6):841-6. PubMed ID: 8780897
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  • 13. Mitochondrial repair of 8-oxoguanine is deficient in Cockayne syndrome group B.
    Stevnsner T, Nyaga S, de Souza-Pinto NC, van der Horst GT, Gorgels TG, Hogue BA, Thorslund T, Bohr VA.
    Oncogene; 2002 Dec 12; 21(57):8675-82. PubMed ID: 12483520
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  • 17. Transcription-coupled repair of 8-oxoguanine: requirement for XPG, TFIIH, and CSB and implications for Cockayne syndrome.
    Le Page F, Kwoh EE, Avrutskaya A, Gentil A, Leadon SA, Sarasin A, Cooper PK.
    Cell; 2000 Apr 14; 101(2):159-71. PubMed ID: 10786832
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  • 18. A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA base damage.
    Osterod M, Larsen E, Le Page F, Hengstler JG, Van Der Horst GT, Boiteux S, Klungland A, Epe B.
    Oncogene; 2002 Nov 28; 21(54):8232-9. PubMed ID: 12447686
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