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Journal Abstract Search

186 related items for PubMed ID: 12612945

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  • 7. [Effects of lovastatin on renal function and expression of phosphorylating-p38 mitogen-activated protein kinase in experimental diabetic nephropathy in rats].
    Wang LH, Duan HJ, Shi YH, Liu QJ, He N, Liu SX.
    Zhongguo Wei Zhong Bing Ji Jiu Yi Xue; 2004 Dec; 16(12):734-7. PubMed ID: 15585148
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  • 8. Kidney involvement and disease in patients with diabetes.
    Chiarelli F, Trotta D, Verrotti A, Mohn A.
    Panminerva Med; 2003 Mar; 45(1):23-41. PubMed ID: 12682618
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  • 9. RAGE control of diabetic nephropathy in a mouse model: effects of RAGE gene disruption and administration of low-molecular weight heparin.
    Myint KM, Yamamoto Y, Doi T, Kato I, Harashima A, Yonekura H, Watanabe T, Shinohara H, Takeuchi M, Tsuneyama K, Hashimoto N, Asano M, Takasawa S, Okamoto H, Yamamoto H.
    Diabetes; 2006 Sep; 55(9):2510-22. PubMed ID: 16936199
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  • 10. Oxidative stress mediates protein kinase C activation and advanced glycation end product formation in a mesangial cell model of diabetes and high protein diet.
    Tuttle KR, Anderberg RJ, Cooney SK, Meek RL.
    Am J Nephrol; 2009 Sep; 29(3):171-80. PubMed ID: 18781061
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  • 11. RhoA/Rho-kinase contribute to the pathogenesis of diabetic renal disease.
    Peng F, Wu D, Gao B, Ingram AJ, Zhang B, Chorneyko K, McKenzie R, Krepinsky JC.
    Diabetes; 2008 Jun; 57(6):1683-92. PubMed ID: 18356410
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  • 13. An inhibitor of advanced glycation end product formation reduces N epsilon-(carboxymethyl)lysine accumulation in glomeruli of diabetic rats.
    Nakamura S, Tachikawa T, Tobita K, Aoyama I, Takayama F, Enomoto A, Niwa T.
    Am J Kidney Dis; 2003 Mar; 41(3 Suppl 1):S68-71. PubMed ID: 12612956
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  • 15. Angiotensin II type 1 receptor expression is increased via 12-lipoxygenase in high glucose-stimulated glomerular cells and type 2 diabetic glomeruli.
    Xu ZG, Miao LN, Cui YC, Jia Y, Yuan H, Wu M.
    Nephrol Dial Transplant; 2009 Jun; 24(6):1744-52. PubMed ID: 19103735
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  • 16. Glomerular changes in the KK-Ay/Ta mouse: a possible model for human type 2 diabetic nephropathy.
    Ito T, Tanimoto M, Yamada K, Kaneko S, Matsumoto M, Obayashi K, Hagiwara S, Murakoshi M, Aoki T, Wakabayashi M, Gohda T, Funabiki K, Maeda K, Horikoshi S, Tomino Y.
    Nephrology (Carlton); 2006 Feb; 11(1):29-35. PubMed ID: 16509929
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  • 17. Eicosapentaenoic acid ameliorates diabetic nephropathy of type 2 diabetic KKAy/Ta mice: involvement of MCP-1 suppression and decreased ERK1/2 and p38 phosphorylation.
    Hagiwara S, Makita Y, Gu L, Tanimoto M, Zhang M, Nakamura S, Kaneko S, Itoh T, Gohda T, Horikoshi S, Tomino Y.
    Nephrol Dial Transplant; 2006 Mar; 21(3):605-15. PubMed ID: 16282336
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  • 18. Induction of reactive oxygen species from isolated rat glomeruli by protein kinase C activation and TNF-alpha stimulation, and effects of a phosphodiesterase inhibitor.
    Koike N, Takamura T, Kaneko S.
    Life Sci; 2007 Apr 10; 80(18):1721-8. PubMed ID: 17346751
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  • 19. Tranilast prevents the progression of experimental diabetic nephropathy through suppression of enhanced extracellular matrix gene expression.
    Akahori H, Ota T, Torita M, Ando H, Kaneko S, Takamura T.
    J Pharmacol Exp Ther; 2005 Aug 10; 314(2):514-21. PubMed ID: 15857946
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  • 20. Diabetic endothelial nitric oxide synthase knockout mice develop advanced diabetic nephropathy.
    Nakagawa T, Sato W, Glushakova O, Heinig M, Clarke T, Campbell-Thompson M, Yuzawa Y, Atkinson MA, Johnson RJ, Croker B.
    J Am Soc Nephrol; 2007 Feb 10; 18(2):539-50. PubMed ID: 17202420
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