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319 related items for PubMed ID: 12615671
1. Changes in perlecan expression during vascular injury: role in the inhibition of smooth muscle cell proliferation in the late lesion. Kinsella MG, Tran PK, Weiser-Evans MC, Reidy M, Majack RA, Wight TN. Arterioscler Thromb Vasc Biol; 2003 Apr 01; 23(4):608-14. PubMed ID: 12615671 [Abstract] [Full Text] [Related]
2. Removal of heparan sulfate by heparinase treatment inhibits FGF-2-dependent smooth muscle cell proliferation in injured rat carotid arteries. Kinsella MG, Irvin C, Reidy MA, Wight TN. Atherosclerosis; 2004 Jul 01; 175(1):51-7. PubMed ID: 15186946 [Abstract] [Full Text] [Related]
11. Arterial heparan sulfate proteoglycans inhibit vascular smooth muscle cell proliferation and phenotype change in vitro and neointimal formation in vivo. Bingley JA, Hayward IP, Campbell JH, Campbell GR. J Vasc Surg; 1998 Aug 31; 28(2):308-18. PubMed ID: 9719326 [Abstract] [Full Text] [Related]
12. CREG promotes a mature smooth muscle cell phenotype and reduces neointimal formation in balloon-injured rat carotid artery. Han Y, Deng J, Guo L, Yan C, Liang M, Kang J, Liu H, Graham AM, Li S. Cardiovasc Res; 2008 Jun 01; 78(3):597-604. PubMed ID: 18267954 [Abstract] [Full Text] [Related]
19. Role of basic fibroblast growth factor and platelet-derived growth factor (B-chain) in neointima formation after arterial injury. Lindner V. Z Kardiol; 1995 Aug 10; 84 Suppl 4():137-44. PubMed ID: 8585266 [Abstract] [Full Text] [Related]
20. The effect of 2,3,4',5-tetrahydroxystilbene-2-0-β-D glucoside on neointima formation in a rat artery balloon injury model and its possible mechanisms. Xu XL, Ling DY, Zhu QY, Fan WJ, Zhang W. Eur J Pharmacol; 2013 Jan 05; 698(1-3):370-8. PubMed ID: 23178522 [Abstract] [Full Text] [Related] Page: [Next] [New Search]