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Journal Abstract Search


195 related items for PubMed ID: 12626348

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  • 3. Differential effect of imipenem treatment on injury caused by cecal ligation and puncture in wild-type and NK cell-deficient beta(2)-microgloblin knockout mice.
    Enoh VT, Lin CY, Varma TK, Sherwood ER.
    Am J Physiol Gastrointest Liver Physiol; 2006 Feb; 290(2):G277-84. PubMed ID: 16166341
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  • 5. Differential effect of imipenem treatment on wild-type and NK cell-deficient CD8 knockout mice during acute intra-abdominal injury.
    Enoh VT, Fairchild CD, Lin CY, Varma TK, Sherwood ER.
    Am J Physiol Regul Integr Comp Physiol; 2006 Mar; 290(3):R685-93. PubMed ID: 16269570
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  • 6. CD4+ T-cell depletion is not associated with alterations in survival, bacterial clearance, and inflammation after cecal ligation and puncture.
    Enoh VT, Lin SH, Etogo A, Lin CY, Sherwood ER.
    Shock; 2008 Jan; 29(1):56-64. PubMed ID: 17693926
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  • 7. Are alterations of lymphocyte subpopulations in polymicrobial sepsis and DHEA treatment mediated by the tumour necrosis factor (TNF)-alpha receptor (TNF-RI)? A study in TNF-RI (TNF-RI(-/-)) knock-out rodents.
    Hildebrand F, Pape HC, Harwood P, Wittwer T, Krettek C, van Griensven M.
    Clin Exp Immunol; 2004 Nov; 138(2):221-9. PubMed ID: 15498030
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  • 8. Beta2-microglobulin knockout mice treated with anti-asialoGM1 exhibit improved hemodynamics and cardiac contractile function during acute intra-abdominal sepsis.
    Tao W, Sherwood ER.
    Am J Physiol Regul Integr Comp Physiol; 2004 Mar; 286(3):R569-75. PubMed ID: 14630624
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  • 9. A1 adenosine receptor knockout mice exhibit increased mortality, renal dysfunction, and hepatic injury in murine septic peritonitis.
    Gallos G, Ruyle TD, Emala CW, Lee HT.
    Am J Physiol Renal Physiol; 2005 Aug; 289(2):F369-76. PubMed ID: 15784841
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  • 11. Requirement of TNF and TNF receptor type 2 for LPS-induced protection from lethal septic peritonitis.
    Echtenacher B, Männel DN.
    J Endotoxin Res; 2002 Aug; 8(5):365-9. PubMed ID: 12537695
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  • 12. AsialoGM1+CD8+ central memory-type T cells in unimmunized mice as novel immunomodulator of IFN-gamma-dependent type 1 immunity.
    Kosaka A, Wakita D, Matsubara N, Togashi Y, Nishimura S, Kitamura H, Nishimura T.
    Int Immunol; 2007 Mar; 19(3):249-56. PubMed ID: 17229818
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  • 13. CD4 and CD8 T Cell Memory Interactions Alter Innate Immunity and Organ Injury in the CLP Sepsis Model.
    Taylor MD, Fernandes TD, Kelly AP, Abraham MN, Deutschman CS.
    Front Immunol; 2020 Mar; 11():563402. PubMed ID: 33329524
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  • 14. MHC class I-dependent presentation of exoerythrocytic antigens to CD8+ T lymphocytes is required for protective immunity against Plasmodium berghei.
    White KL, Snyder HL, Krzych U.
    J Immunol; 1996 May 01; 156(9):3374-81. PubMed ID: 8617963
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  • 15. Beta 2-microglobulin-deficient background ameliorates lethal phenotype of the TGF-beta 1 null mouse.
    Kobayashi S, Yoshida K, Ward JM, Letterio JJ, Longenecker G, Yaswen L, Mittleman B, Mozes E, Roberts AB, Karlsson S, Kulkarni AB.
    J Immunol; 1999 Oct 01; 163(7):4013-9. PubMed ID: 10491004
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  • 16. STAT4 is required for antibacterial defense but enhances mortality during polymicrobial sepsis.
    Godshall CJ, Lentsch AB, Peyton JC, Scott MJ, Cheadle WG.
    Clin Diagn Lab Immunol; 2001 Nov 01; 8(6):1044-8. PubMed ID: 11687437
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  • 17. Inhibition of NF-kappaB activity prevents downregulation of alpha1-adrenergic receptors and circulatory failure during CLP-induced sepsis.
    Schmidt C, Kurt B, Höcherl K, Bucher M.
    Shock; 2009 Sep 01; 32(3):239-46. PubMed ID: 19106815
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  • 18. Endogenous IL-10 protects mice from death during septic peritonitis.
    van der Poll T, Marchant A, Buurman WA, Berman L, Keogh CV, Lazarus DD, Nguyen L, Goldman M, Moldawer LL, Lowry SF.
    J Immunol; 1995 Dec 01; 155(11):5397-401. PubMed ID: 7594556
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  • 19. Critical role of CD14 for production of proinflammatory cytokines and cytokine inhibitors during sepsis with failure to alter morbidity or mortality.
    Ebong SJ, Goyert SM, Nemzek JA, Kim J, Bolgos GL, Remick DG.
    Infect Immun; 2001 Apr 01; 69(4):2099-106. PubMed ID: 11254563
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  • 20. Sepsis leads to a reduced antigen-specific primary antibody response.
    Mohr A, Polz J, Martin EM, Griessl S, Kammler A, Pötschke C, Lechner A, Bröker BM, Mostböck S, Männel DN.
    Eur J Immunol; 2012 Feb 01; 42(2):341-52. PubMed ID: 22105154
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