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Journal Abstract Search

476 related items for PubMed ID: 12642586

  • 1. Minimal BH3 peptides promote cell death by antagonizing anti-apoptotic proteins.
    Moreau C, Cartron PF, Hunt A, Meflah K, Green DR, Evan G, Vallette FM, Juin P.
    J Biol Chem; 2003 May 23; 278(21):19426-35. PubMed ID: 12642586
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  • 2. The BH3 alpha-helical mimic BH3-M6 disrupts Bcl-X(L), Bcl-2, and MCL-1 protein-protein interactions with Bax, Bak, Bad, or Bim and induces apoptosis in a Bax- and Bim-dependent manner.
    Kazi A, Sun J, Doi K, Sung SS, Takahashi Y, Yin H, Rodriguez JM, Becerril J, Berndt N, Hamilton AD, Wang HG, Sebti SM.
    J Biol Chem; 2011 Mar 18; 286(11):9382-92. PubMed ID: 21148306
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  • 3. Bak BH3 peptides antagonize Bcl-xL function and induce apoptosis through cytochrome c-independent activation of caspases.
    Holinger EP, Chittenden T, Lutz RJ.
    J Biol Chem; 1999 May 07; 274(19):13298-304. PubMed ID: 10224090
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  • 4. Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax.
    Hockings C, Anwari K, Ninnis RL, Brouwer J, O'Hely M, Evangelista M, Hinds MG, Czabotar PE, Lee EF, Fairlie WD, Dewson G, Kluck RM.
    Cell Death Dis; 2015 Apr 23; 6(4):e1735. PubMed ID: 25906158
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  • 5. Bcl-XL protects BimEL-induced Bax conformational change and cytochrome C release independent of interacting with Bax or BimEL.
    Yamaguchi H, Wang HG.
    J Biol Chem; 2002 Nov 01; 277(44):41604-12. PubMed ID: 12198137
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  • 6. Induction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactions.
    Finnegan NM, Curtin JF, Prevost G, Morgan B, Cotter TG.
    Br J Cancer; 2001 Jul 06; 85(1):115-21. PubMed ID: 11437412
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  • 7. Peptides derived from BH3 domains of Bcl-2 family members: a comparative analysis of inhibition of Bcl-2, Bcl-x(L) and Bax oligomerization, induction of cytochrome c release, and activation of cell death.
    Shangary S, Johnson DE.
    Biochemistry; 2002 Jul 30; 41(30):9485-95. PubMed ID: 12135371
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  • 11. Structural biology of the Bcl-2 family of proteins.
    Petros AM, Olejniczak ET, Fesik SW.
    Biochim Biophys Acta; 2004 Mar 01; 1644(2-3):83-94. PubMed ID: 14996493
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  • 12. Molecular dynamics simulations of pro-apoptotic BH3 peptide helices in aqueous medium: relationship between helix stability and their binding affinities to the anti-apoptotic protein Bcl-X(L).
    Lama D, Sankararamakrishnan R.
    J Comput Aided Mol Des; 2011 May 01; 25(5):413-26. PubMed ID: 21523491
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  • 13. Sequence and helicity requirements for the proapoptotic activity of Bax BH3 peptides.
    Shangary S, Oliver CL, Tillman TS, Cascio M, Johnson DE.
    Mol Cancer Ther; 2004 Nov 01; 3(11):1343-54. PubMed ID: 15542773
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  • 14. Anti-apoptotic Bcl-XL protein in complex with BH3 peptides of pro-apoptotic Bak, Bad, and Bim proteins: comparative molecular dynamics simulations.
    Lama D, Sankararamakrishnan R.
    Proteins; 2008 Nov 01; 73(2):492-514. PubMed ID: 18452209
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  • 16. In non-transformed cells Bak activates upon loss of anti-apoptotic Bcl-XL and Mcl-1 but in the absence of active BH3-only proteins.
    Senft D, Weber A, Saathoff F, Berking C, Heppt MV, Kammerbauer C, Rothenfusser S, Kellner S, Kurgyis Z, Besch R, Häcker G.
    Cell Death Dis; 2015 Nov 26; 6(11):e1996. PubMed ID: 26610208
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  • 19. BH3-only proteins target BCL-xL/MCL-1, not BAX/BAK, to initiate apoptosis.
    Huang K, O'Neill KL, Li J, Zhou W, Han N, Pang X, Wu W, Struble L, Borgstahl G, Liu Z, Zhang L, Luo X.
    Cell Res; 2019 Nov 26; 29(11):942-952. PubMed ID: 31551537
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  • 20. Bax/Bak activation in the absence of Bid, Bim, Puma, and p53.
    Zhang J, Huang K, O'Neill KL, Pang X, Luo X.
    Cell Death Dis; 2016 Jun 16; 7(6):e2266. PubMed ID: 27310874
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