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Journal Abstract Search
164 related items for PubMed ID: 12672777
1. Inhibition of mitochondrial Ca2+ uptake affects phasic release from motor terminals differently depending on external [Ca2+]. Talbot JD, David G, Barrett EF. J Neurophysiol; 2003 Jul; 90(1):491-502. PubMed ID: 12672777 [Abstract] [Full Text] [Related]
2. Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals. David G, Barrett EF. J Physiol; 2003 Apr 15; 548(Pt 2):425-38. PubMed ID: 12588898 [Abstract] [Full Text] [Related]
4. The role of extracellular calcium in exo- and endocytosis of synaptic vesicles at the frog motor nerve terminals. Zefirov AL, Abdrakhmanov MM, Mukhamedyarov MA, Grigoryev PN. Neuroscience; 2006 Dec 28; 143(4):905-10. PubMed ID: 17000054 [Abstract] [Full Text] [Related]
8. Extrusion of Ca2+ from mouse motor terminal mitochondria via a Na+-Ca2+ exchanger increases post-tetanic evoked release. García-Chacón LE, Nguyen KT, David G, Barrett EF. J Physiol; 2006 Aug 01; 574(Pt 3):663-75. PubMed ID: 16613870 [Abstract] [Full Text] [Related]
9. Rapid, stimulation-induced reduction of C12-resorufin in motor nerve terminals: linkage to mitochondrial metabolism. Talbot JD, Barrett JN, Barrett EF, David G. J Neurochem; 2008 May 01; 105(3):807-19. PubMed ID: 18205748 [Abstract] [Full Text] [Related]
14. Role of mitochondrial dysfunction in the Ca2+-induced decline of transmitter release at K+-depolarized motor neuron terminals. Calupca MA, Hendricks GM, Hardwick JC, Parsons RL. J Neurophysiol; 1999 Feb 01; 81(2):498-506. PubMed ID: 10036254 [Abstract] [Full Text] [Related]