These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

521 related items for PubMed ID: 12739027

  • 1. Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.
    Chang TI, Horal M, Jain SK, Wang F, Patel R, Loeken MR.
    Diabetologia; 2003 Apr; 46(4):538-45. PubMed ID: 12739027
    [Abstract] [Full Text] [Related]

  • 2. Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis.
    Phelan SA, Ito M, Loeken MR.
    Diabetes; 1997 Jul; 46(7):1189-97. PubMed ID: 9200655
    [Abstract] [Full Text] [Related]

  • 3. Evidence that elevated glucose causes altered gene expression, apoptosis, and neural tube defects in a mouse model of diabetic pregnancy.
    Fine EL, Horal M, Chang TI, Fortin G, Loeken MR.
    Diabetes; 1999 Dec; 48(12):2454-62. PubMed ID: 10580436
    [Abstract] [Full Text] [Related]

  • 4. AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy.
    Wu Y, Viana M, Thirumangalathu S, Loeken MR.
    Diabetologia; 2012 Jan; 55(1):245-54. PubMed ID: 21993711
    [Abstract] [Full Text] [Related]

  • 5.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 6. Polymorphic susceptibility to the molecular causes of neural tube defects during diabetic embryopathy.
    Pani L, Horal M, Loeken MR.
    Diabetes; 2002 Sep; 51(9):2871-4. PubMed ID: 12196484
    [Abstract] [Full Text] [Related]

  • 7. Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress.
    Li R, Chase M, Jung SK, Smith PJ, Loeken MR.
    Am J Physiol Endocrinol Metab; 2005 Oct; 289(4):E591-9. PubMed ID: 15928021
    [Abstract] [Full Text] [Related]

  • 8. Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy.
    Loeken MR.
    Am J Med Genet C Semin Med Genet; 2005 May 15; 135C(1):77-87. PubMed ID: 15800853
    [Abstract] [Full Text] [Related]

  • 9.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 10. Lack of metformin effect on mouse embryo AMPK activity: implications for metformin treatment during pregnancy.
    Lee HY, Wei D, Loeken MR.
    Diabetes Metab Res Rev; 2014 Jan 15; 30(1):23-30. PubMed ID: 23983188
    [Abstract] [Full Text] [Related]

  • 11.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 12. Modulation of nuclear factor-κB signaling and reduction of neural tube defects by quercetin-3-glucoside in embryos of diabetic mice.
    Tan C, Meng F, Reece EA, Zhao Z.
    Am J Obstet Gynecol; 2018 Aug 15; 219(2):197.e1-197.e8. PubMed ID: 29733843
    [Abstract] [Full Text] [Related]

  • 13. Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.
    Morgan SC, Relaix F, Sandell LL, Loeken MR.
    Birth Defects Res A Clin Mol Teratol; 2008 Jun 15; 82(6):453-63. PubMed ID: 18435457
    [Abstract] [Full Text] [Related]

  • 14. High glucose-induced oxidative stress represses sirtuin deacetylase expression and increases histone acetylation leading to neural tube defects.
    Yu J, Wu Y, Yang P.
    J Neurochem; 2016 May 15; 137(3):371-83. PubMed ID: 26896748
    [Abstract] [Full Text] [Related]

  • 15. Reduced expression of pax-3 is associated with overexpression of cdc46 in the mouse embryo.
    Hill AL, Phelan SA, Loeken MR.
    Dev Genes Evol; 1998 May 15; 208(3):128-34. PubMed ID: 9601985
    [Abstract] [Full Text] [Related]

  • 16. Altered gene expression in rat cranial neural crest cells exposed to a teratogenic glucose concentration in vitro: paradoxical downregulation of antioxidative defense genes.
    Wentzel P, Eriksson UJ.
    Birth Defects Res B Dev Reprod Toxicol; 2011 Oct 15; 92(5):487-97. PubMed ID: 21818840
    [Abstract] [Full Text] [Related]

  • 17. Expression of the gene encoding the high-Km glucose transporter 2 by the early postimplantation mouse embryo is essential for neural tube defects associated with diabetic embryopathy.
    Li R, Thorens B, Loeken MR.
    Diabetologia; 2007 Mar 15; 50(3):682-9. PubMed ID: 17235524
    [Abstract] [Full Text] [Related]

  • 18.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 19. Deficiency of the oxidative stress-responsive kinase p70S6K1 restores autophagy and ameliorates neural tube defects in diabetic embryopathy.
    Cao S, Shen WB, Reece EA, Yang P.
    Am J Obstet Gynecol; 2020 Nov 15; 223(5):753.e1-753.e14. PubMed ID: 32416155
    [Abstract] [Full Text] [Related]

  • 20. Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: involvement in diabetic teratogenesis.
    Horal M, Zhang Z, Stanton R, Virkamäki A, Loeken MR.
    Birth Defects Res A Clin Mol Teratol; 2004 Aug 15; 70(8):519-27. PubMed ID: 15329829
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 27.