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88 related items for PubMed ID: 12754411
1. Arsenic trioxide-induced apoptosis is independent of stress-responsive signaling pathways but sensitive to inhibition of inducible nitric oxide synthase in HepG2 cells. Kang SH, Song JH, Kang HK, Kang JH, Kim SJ, Kang HW, Lee YK, Park DB. Exp Mol Med; 2003 Apr 30; 35(2):83-90. PubMed ID: 12754411 [Abstract] [Full Text] [Related]
8. Enhancement of esculetin on arsenic trioxide-provoked apoptosis in human leukemia U937 cells. Lin TH, Lu FJ, Yin YF, Tseng TH. Chem Biol Interact; 2009 Jun 15; 180(1):61-8. PubMed ID: 19428345 [Abstract] [Full Text] [Related]
12. Increased apoptotic efficacy of lonidamine plus arsenic trioxide combination in human leukemia cells. Reactive oxygen species generation and defensive protein kinase (MEK/ERK, Akt/mTOR) modulation. Calviño E, Estañ MC, Simón GP, Sancho P, Boyano-Adánez Mdel C, de Blas E, Bréard J, Aller P. Biochem Pharmacol; 2011 Dec 01; 82(11):1619-29. PubMed ID: 21889928 [Abstract] [Full Text] [Related]
14. Role of GSTP1-1 in mediating the effect of As2O3 in the Acute Promyelocytic Leukemia cell line NB4. Bernardini S, Nuccetelli M, Noguera NI, Bellincampi L, Lunghi P, Bonati A, Mann K, Miller WH, Federici G, Lo Coco F. Ann Hematol; 2006 Oct 01; 85(10):681-7. PubMed ID: 16733740 [Abstract] [Full Text] [Related]
15. Elimination of the differential chemoresistance between the murine B-cell lymphoma LY-ar and LY-as cell lines after arsenic (As2O3) exposure via the overexpression of gsto1 (p28). Giri U, Terry NH, Kala SV, Lieberman MW, Story MD. Cancer Chemother Pharmacol; 2005 Jun 01; 55(6):511-21. PubMed ID: 15761769 [Abstract] [Full Text] [Related]
16. Sustained activation of c-jun-terminal kinase (JNK) is closely related to arsenic trioxide-induced apoptosis in an acute myeloid leukemia (M2)-derived cell line, NKM-1. Kajiguchi T, Yamamoto K, Hossain K, Akhand AA, Nakashima I, Naoe T, Saito H, Emi N. Leukemia; 2003 Nov 01; 17(11):2189-95. PubMed ID: 12931215 [Abstract] [Full Text] [Related]
17. Overexpression of protein kinase C isoforms protects RAW 264.7 macrophages from nitric oxide-induced apoptosis: involvement of c-Jun N-terminal kinase/stress-activated protein kinase, p38 kinase, and CPP-32 protease pathways. Jun CD, Oh CD, Kwak HJ, Pae HO, Yoo JC, Choi BM, Chun JS, Park RK, Chung HT. J Immunol; 1999 Mar 15; 162(6):3395-401. PubMed ID: 10092794 [Abstract] [Full Text] [Related]