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153 related items for PubMed ID: 12801805

  • 1. The use of the murine model of infection with Leishmania major to reveal the antagonistic effects that IL-4 can exert on T helper cell development and demonstrate that these opposite effects depend upon the nature of the cells targeted for IL-4 signaling.
    Louis JA, Gumy A, Voigt H, Launois P, Rocken M.
    Pathol Biol (Paris); 2003 Mar; 51(2):71-3. PubMed ID: 12801805
    [Abstract] [Full Text] [Related]

  • 2. IL-4 instructs TH1 responses and resistance to Leishmania major in susceptible BALB/c mice.
    Biedermann T, Zimmermann S, Himmelrich H, Gumy A, Egeter O, Sakrauski AK, Seegmüller I, Voigt H, Launois P, Levine AD, Wagner H, Heeg K, Louis JA, Röcken M.
    Nat Immunol; 2001 Nov; 2(11):1054-60. PubMed ID: 11600887
    [Abstract] [Full Text] [Related]

  • 3. Functional plasticity of the LACK-reactive Vbeta4-Valpha8 CD4(+) T cells normally producing the early IL-4 instructing Th2 cell development and susceptibility to Leishmania major in BALB / c mice.
    Maillard I, Launois P, Himmelrich H, Acha-Orbea H, Diggelmann H, Locksley RM, Louis JA.
    Eur J Immunol; 2001 Apr; 31(4):1288-96. PubMed ID: 11298356
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  • 4. IL-4 rapidly produced by V beta 4 V alpha 8 CD4+ T cells instructs Th2 development and susceptibility to Leishmania major in BALB/c mice.
    Launois P, Maillard I, Pingel S, Swihart KG, Xénarios I, Acha-Orbea H, Diggelmann H, Locksley RM, MacDonald HR, Louis JA.
    Immunity; 1997 May; 6(5):541-9. PubMed ID: 9175832
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  • 5. Altered ligands reveal limited plasticity in the T cell response to a pathogenic epitope.
    Pingel S, Launois P, Fowell DJ, Turck CW, Southwood S, Sette A, Glaichenhaus N, Louis JA, Locksley RM.
    J Exp Med; 1999 Apr 05; 189(7):1111-20. PubMed ID: 10190902
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  • 6. In BALB/c mice, IL-4 production during the initial phase of infection with Leishmania major is necessary and sufficient to instruct Th2 cell development resulting in progressive disease.
    Himmelrich H, Launois P, Maillard I, Biedermann T, Tacchini-Cottier F, Locksley RM, Röcken M, Louis JA.
    J Immunol; 2000 May 01; 164(9):4819-25. PubMed ID: 10779790
    [Abstract] [Full Text] [Related]

  • 7. Rapid IL-4 production by Leishmania homolog of mammalian RACK1-reactive CD4(+) T cells in resistant mice treated once with anti-IL-12 or -IFN-gamma antibodies at the onset of infection with Leishmania major instructs Th2 cell development, resulting in nonhealing lesions.
    Launois P, Gumy A, Himmelrich H, Locksley RM, Röcken M, Louis JA.
    J Immunol; 2002 May 01; 168(9):4628-35. PubMed ID: 11971011
    [Abstract] [Full Text] [Related]

  • 8. Anti-leishmania effector functions of CD4+ Th1 cells and early events instructing Th2 cell development and susceptibility to Leishmania major in BALB/c mice.
    Louis JA, Conceiçao-Silva F, Himmelrich H, Tacchini-Cottier F, Launois P.
    Adv Exp Med Biol; 1998 May 01; 452():53-60. PubMed ID: 9889959
    [Abstract] [Full Text] [Related]

  • 9. Genetic susceptibility to Leishmania: IL-12 responsiveness in TH1 cell development.
    Güler ML, Gorham JD, Hsieh CS, Mackey AJ, Steen RG, Dietrich WF, Murphy KM.
    Science; 1996 Feb 16; 271(5251):984-7. PubMed ID: 8584935
    [Abstract] [Full Text] [Related]

  • 10. Deletion of IL-4Ralpha on CD4 T cells renders BALB/c mice resistant to Leishmania major infection.
    Radwanska M, Cutler AJ, Hoving JC, Magez S, Holscher C, Bohms A, Arendse B, Kirsch R, Hunig T, Alexander J, Kaye P, Brombacher F.
    PLoS Pathog; 2007 May 11; 3(5):e68. PubMed ID: 17500591
    [Abstract] [Full Text] [Related]

  • 11. KM(+), a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection.
    Panunto-Castelo A, Souza MA, Roque-Barreira MC, Silva JS.
    Glycobiology; 2001 Dec 11; 11(12):1035-42. PubMed ID: 11805076
    [Abstract] [Full Text] [Related]

  • 12. Setting in motion the immune mechanisms underlying genetically determined resistance and susceptibility to infection with Leishmania major.
    Launois P, Conceiçao-Silva F, Himmerlich H, Parra-Lopez C, Tacchini-Cottier F, Louis JA.
    Parasite Immunol; 1998 May 11; 20(5):223-30. PubMed ID: 9651923
    [Abstract] [Full Text] [Related]

  • 13. Induction of a Th2 population from a polarized Leishmania-specific Th1 population by in vitro culture with IL-4.
    Mocci S, Coffman RL.
    J Immunol; 1995 Apr 15; 154(8):3779-87. PubMed ID: 7706719
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  • 17. The IL-4 rapidly produced in BALB/c mice after infection with Leishmania major down-regulates IL-12 receptor beta 2-chain expression on CD4+ T cells resulting in a state of unresponsiveness to IL-12.
    Himmelrich H, Parra-Lopez C, Tacchini-Cottier F, Louis JA, Launois P.
    J Immunol; 1998 Dec 01; 161(11):6156-63. PubMed ID: 9834101
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  • 19. Genetically resistant mice lacking MyD88-adapter protein display a high susceptibility to Leishmania major infection associated with a polarized Th2 response.
    Muraille E, De Trez C, Brait M, De Baetselier P, Leo O, Carlier Y.
    J Immunol; 2003 Apr 15; 170(8):4237-41. PubMed ID: 12682257
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