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573 related items for PubMed ID: 12805111

  • 1. Changes in the expression and localization of the paranodal protein Caspr on axons in chronic multiple sclerosis.
    Wolswijk G, Balesar R.
    Brain; 2003 Jul; 126(Pt 7):1638-49. PubMed ID: 12805111
    [Abstract] [Full Text] [Related]

  • 2. Nodal, paranodal and juxtaparanodal axonal proteins during demyelination and remyelination in multiple sclerosis.
    Coman I, Aigrot MS, Seilhean D, Reynolds R, Girault JA, Zalc B, Lubetzki C.
    Brain; 2006 Dec; 129(Pt 12):3186-95. PubMed ID: 16766541
    [Abstract] [Full Text] [Related]

  • 3. A novel Caspr mutation causes the shambling mouse phenotype by disrupting axoglial interactions of myelinated nerves.
    Sun XY, Takagishi Y, Okabe E, Chishima Y, Kanou Y, Murase S, Mizumura K, Inaba M, Komatsu Y, Hayashi Y, Peles E, Oda S, Murata Y.
    J Neuropathol Exp Neurol; 2009 Nov; 68(11):1207-18. PubMed ID: 19816196
    [Abstract] [Full Text] [Related]

  • 4. Disposition of axonal caspr with respect to glial cell membranes: Implications for the process of myelination.
    Pedraza L, Huang JK, Colman D.
    J Neurosci Res; 2009 Nov 15; 87(15):3480-91. PubMed ID: 19170162
    [Abstract] [Full Text] [Related]

  • 5. Schwann cell remyelination of demyelinated axons in spinal cord multiple sclerosis lesions.
    Itoyama Y, Webster HD, Richardson EP, Trapp BD.
    Ann Neurol; 1983 Sep 15; 14(3):339-46. PubMed ID: 6195956
    [Abstract] [Full Text] [Related]

  • 6. Demyelination in spinal cord injury and multiple sclerosis: what can we do to enhance functional recovery?
    Waxman SG.
    J Neurotrauma; 1992 Mar 15; 9 Suppl 1():S105-17. PubMed ID: 1588601
    [Abstract] [Full Text] [Related]

  • 7. Altered expression patterns of group I and II metabotropic glutamate receptors in multiple sclerosis.
    Geurts JJ, Wolswijk G, Bö L, van der Valk P, Polman CH, Troost D, Aronica E.
    Brain; 2003 Aug 15; 126(Pt 8):1755-66. PubMed ID: 12805104
    [Abstract] [Full Text] [Related]

  • 8. Immunocytochemical observations on the distribution of myelin-associated glycoprotein and myelin basic protein in multiple sclerosis lesions.
    Itoyama Y, Sternberger NH, Webster HD, Quarles RH, Cohen SR, Richardson EP.
    Ann Neurol; 1980 Feb 15; 7(2):167-77. PubMed ID: 6154440
    [Abstract] [Full Text] [Related]

  • 9. Ultrastructural analysis of the paranodal junction of myelinated fibers in 31-month-old-rats.
    Sugiyama I, Tanaka K, Akita M, Yoshida K, Kawase T, Asou H.
    J Neurosci Res; 2002 Nov 01; 70(3):309-17. PubMed ID: 12391590
    [Abstract] [Full Text] [Related]

  • 10. Axonal transection in the lesions of multiple sclerosis.
    Trapp BD, Peterson J, Ransohoff RM, Rudick R, Mörk S, Bö L.
    N Engl J Med; 1998 Jan 29; 338(5):278-85. PubMed ID: 9445407
    [Abstract] [Full Text] [Related]

  • 11. Reduced raft-association of NF155 in active MS-lesions is accompanied by the disruption of the paranodal junction.
    Maier O, Baron W, Hoekstra D.
    Glia; 2007 Jun 29; 55(8):885-95. PubMed ID: 17405145
    [Abstract] [Full Text] [Related]

  • 12. The axonal membrane protein Caspr, a homologue of neurexin IV, is a component of the septate-like paranodal junctions that assemble during myelination.
    Einheber S, Zanazzi G, Ching W, Scherer S, Milner TA, Peles E, Salzer JL.
    J Cell Biol; 1997 Dec 15; 139(6):1495-506. PubMed ID: 9396755
    [Abstract] [Full Text] [Related]

  • 13. The cytoskeletal adaptor protein band 4.1B is required for the maintenance of paranodal axoglial septate junctions in myelinated axons.
    Buttermore ED, Dupree JL, Cheng J, An X, Tessarollo L, Bhat MA.
    J Neurosci; 2011 Jun 01; 31(22):8013-24. PubMed ID: 21632923
    [Abstract] [Full Text] [Related]

  • 14. Functional changes in genetically dysmyelinated spinal cord axons of shiverer mice: role of juxtaparanodal Kv1 family K+ channels.
    Sinha K, Karimi-Abdolrezaee S, Velumian AA, Fehlings MG.
    J Neurophysiol; 2006 Mar 01; 95(3):1683-95. PubMed ID: 16319208
    [Abstract] [Full Text] [Related]

  • 15. Enhanced number and activity of mitochondria in multiple sclerosis lesions.
    Witte ME, Bø L, Rodenburg RJ, Belien JA, Musters R, Hazes T, Wintjes LT, Smeitink JA, Geurts JJ, De Vries HE, van der Valk P, van Horssen J.
    J Pathol; 2009 Oct 01; 219(2):193-204. PubMed ID: 19591199
    [Abstract] [Full Text] [Related]

  • 16. Premyelinating oligodendrocytes in chronic lesions of multiple sclerosis.
    Chang A, Tourtellotte WW, Rudick R, Trapp BD.
    N Engl J Med; 2002 Jan 17; 346(3):165-73. PubMed ID: 11796850
    [Abstract] [Full Text] [Related]

  • 17. Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis.
    Misu T, Fujihara K, Kakita A, Konno H, Nakamura M, Watanabe S, Takahashi T, Nakashima I, Takahashi H, Itoyama Y.
    Brain; 2007 May 17; 130(Pt 5):1224-34. PubMed ID: 17405762
    [Abstract] [Full Text] [Related]

  • 18. Pattern of axonal injury in murine myelin oligodendrocyte glycoprotein induced experimental autoimmune encephalomyelitis: implications for multiple sclerosis.
    Herrero-Herranz E, Pardo LA, Gold R, Linker RA.
    Neurobiol Dis; 2008 May 17; 30(2):162-73. PubMed ID: 18342527
    [Abstract] [Full Text] [Related]

  • 19. No effect of genetic deletion of contactin-associated protein (CASPR) on axonal orientation and synaptic plasticity.
    Pillai AM, Garcia-Fresco GP, Sousa AD, Dupree JL, Philpot BD, Bhat MA.
    J Neurosci Res; 2007 Aug 15; 85(11):2318-31. PubMed ID: 17549747
    [Abstract] [Full Text] [Related]

  • 20. Neurological disability correlates with spinal cord axonal loss and reduced N-acetyl aspartate in chronic multiple sclerosis patients.
    Bjartmar C, Kidd G, Mörk S, Rudick R, Trapp BD.
    Ann Neurol; 2000 Dec 15; 48(6):893-901. PubMed ID: 11117546
    [Abstract] [Full Text] [Related]

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