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Journal Abstract Search

573 related items for PubMed ID: 12805111

  • 1. Changes in the expression and localization of the paranodal protein Caspr on axons in chronic multiple sclerosis.
    Wolswijk G, Balesar R.
    Brain; 2003 Jul; 126(Pt 7):1638-49. PubMed ID: 12805111
    [Abstract] [Full Text] [Related]

  • 2. Nodal, paranodal and juxtaparanodal axonal proteins during demyelination and remyelination in multiple sclerosis.
    Coman I, Aigrot MS, Seilhean D, Reynolds R, Girault JA, Zalc B, Lubetzki C.
    Brain; 2006 Dec; 129(Pt 12):3186-95. PubMed ID: 16766541
    [Abstract] [Full Text] [Related]

  • 3. A novel Caspr mutation causes the shambling mouse phenotype by disrupting axoglial interactions of myelinated nerves.
    Sun XY, Takagishi Y, Okabe E, Chishima Y, Kanou Y, Murase S, Mizumura K, Inaba M, Komatsu Y, Hayashi Y, Peles E, Oda S, Murata Y.
    J Neuropathol Exp Neurol; 2009 Nov; 68(11):1207-18. PubMed ID: 19816196
    [Abstract] [Full Text] [Related]

  • 4. Disposition of axonal caspr with respect to glial cell membranes: Implications for the process of myelination.
    Pedraza L, Huang JK, Colman D.
    J Neurosci Res; 2009 Nov 15; 87(15):3480-91. PubMed ID: 19170162
    [Abstract] [Full Text] [Related]

  • 5. Schwann cell remyelination of demyelinated axons in spinal cord multiple sclerosis lesions.
    Itoyama Y, Webster HD, Richardson EP, Trapp BD.
    Ann Neurol; 1983 Sep 15; 14(3):339-46. PubMed ID: 6195956
    [Abstract] [Full Text] [Related]

  • 6. Demyelination in spinal cord injury and multiple sclerosis: what can we do to enhance functional recovery?
    Waxman SG.
    J Neurotrauma; 1992 Mar 15; 9 Suppl 1():S105-17. PubMed ID: 1588601
    [Abstract] [Full Text] [Related]

  • 7. Altered expression patterns of group I and II metabotropic glutamate receptors in multiple sclerosis.
    Geurts JJ, Wolswijk G, Bö L, van der Valk P, Polman CH, Troost D, Aronica E.
    Brain; 2003 Aug 15; 126(Pt 8):1755-66. PubMed ID: 12805104
    [Abstract] [Full Text] [Related]

  • 8. Immunocytochemical observations on the distribution of myelin-associated glycoprotein and myelin basic protein in multiple sclerosis lesions.
    Itoyama Y, Sternberger NH, Webster HD, Quarles RH, Cohen SR, Richardson EP.
    Ann Neurol; 1980 Feb 15; 7(2):167-77. PubMed ID: 6154440
    [Abstract] [Full Text] [Related]

  • 9. Ultrastructural analysis of the paranodal junction of myelinated fibers in 31-month-old-rats.
    Sugiyama I, Tanaka K, Akita M, Yoshida K, Kawase T, Asou H.
    J Neurosci Res; 2002 Nov 01; 70(3):309-17. PubMed ID: 12391590
    [Abstract] [Full Text] [Related]

  • 10. Axonal transection in the lesions of multiple sclerosis.
    Trapp BD, Peterson J, Ransohoff RM, Rudick R, Mörk S, Bö L.
    N Engl J Med; 1998 Jan 29; 338(5):278-85. PubMed ID: 9445407
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  • 18. Pattern of axonal injury in murine myelin oligodendrocyte glycoprotein induced experimental autoimmune encephalomyelitis: implications for multiple sclerosis.
    Herrero-Herranz E, Pardo LA, Gold R, Linker RA.
    Neurobiol Dis; 2008 May 29; 30(2):162-73. PubMed ID: 18342527
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  • 19. No effect of genetic deletion of contactin-associated protein (CASPR) on axonal orientation and synaptic plasticity.
    Pillai AM, Garcia-Fresco GP, Sousa AD, Dupree JL, Philpot BD, Bhat MA.
    J Neurosci Res; 2007 Aug 15; 85(11):2318-31. PubMed ID: 17549747
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  • 20. Neurological disability correlates with spinal cord axonal loss and reduced N-acetyl aspartate in chronic multiple sclerosis patients.
    Bjartmar C, Kidd G, Mörk S, Rudick R, Trapp BD.
    Ann Neurol; 2000 Dec 15; 48(6):893-901. PubMed ID: 11117546
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