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Journal Abstract Search

442 related items for PubMed ID: 1281165

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  • 3. Suppression of experimental autoimmune encephalomyelitis by oral administration of myelin basic protein. VI. Suppression of adoptively transferred disease and differential effects of oral vs. intravenous tolerization.
    Miller A, Zhang ZJ, Sobel RA, al-Sabbagh A, Weiner HL.
    J Neuroimmunol; 1993 Jul; 46(1-2):73-82. PubMed ID: 7689596
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  • 4. Inactivation of T cell receptor peptide-specific CD4 regulatory T cells induces chronic experimental autoimmune encephalomyelitis (EAE).
    Kumar V, Stellrecht K, Sercarz E.
    J Exp Med; 1996 Nov 01; 184(5):1609-17. PubMed ID: 8920851
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  • 5. Oral tolerance in myelin basic protein T-cell receptor transgenic mice: suppression of autoimmune encephalomyelitis and dose-dependent induction of regulatory cells.
    Chen Y, Inobe J, Kuchroo VK, Baron JL, Janeway CA, Weiner HL.
    Proc Natl Acad Sci U S A; 1996 Jan 09; 93(1):388-91. PubMed ID: 8552644
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  • 7. Adoptive transfer of experimental allergic encephalomyelitis: recipient response to myelin basic protein-reactive lymphocytes.
    Bouwer HG, Hinrichs DJ.
    J Neuroimmunol; 1994 Oct 09; 54(1-2):87-98. PubMed ID: 7523447
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  • 10. Overcoming unresponsiveness in experimental autoimmune encephalomyelitis (EAE) resistant mouse strains by adoptive transfer and antigenic challenge.
    Shaw MK, Zhao XQ, Tse HY.
    J Vis Exp; 2012 Apr 09; (62):e3778. PubMed ID: 22507967
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  • 11. Pathogenic and non-pathogenic T lymphocytes specific for the encephalitogenic epitope of myelin basic protein: functional characteristics and vaccination properties.
    Beraud E, Balzano C, Zamora AJ, Varriale S, Bernard D, Ben-Nun A.
    J Neuroimmunol; 1993 Aug 09; 47(1):41-53. PubMed ID: 7690770
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  • 13. The kinetics of myelin antigen uptake by myeloid cells in the central nervous system during experimental autoimmune encephalomyelitis.
    Sosa RA, Murphey C, Ji N, Cardona AE, Forsthuber TG.
    J Immunol; 2013 Dec 15; 191(12):5848-57. PubMed ID: 24227784
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  • 14. Epitope spreading occurs in active but not passive EAE induced by myelin basic protein.
    Voskuhl RR, Farris RW, Nagasato K, McFarland HF, Dalcq MD.
    J Neuroimmunol; 1996 Nov 15; 70(2):103-11. PubMed ID: 8898718
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  • 15. The immune response to a subdominant epitope in myelin basic protein exon-2 results in immunity to intra- and intermolecular dominant epitopes.
    Zhao ML, Fritz RB.
    J Neuroimmunol; 1995 Sep 15; 61(2):179-84. PubMed ID: 7593553
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  • 16. Lymph node-derived donor encephalitogenic CD4+ T cells in C57BL/6 mice adoptive transfer experimental autoimmune encephalomyelitis highly express GM-CSF and T-bet.
    Cravens PD, Hussain RZ, Zacharias TE, Ben LH, Herndon E, Vinnakota R, Lambracht-Washington D, Nessler S, Zamvil SS, Eagar TN, Stüve O.
    J Neuroinflammation; 2011 Jun 24; 8():73. PubMed ID: 21702922
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  • 18. The complement system contributes to the pathology of experimental autoimmune encephalomyelitis by triggering demyelination and modifying the antigen-specific T and B cell response.
    Hundgeburth LC, Wunsch M, Rovituso D, Recks MS, Addicks K, Lehmann PV, Kuerten S.
    Clin Immunol; 2013 Mar 24; 146(3):155-64. PubMed ID: 23352967
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  • 19. Experimental autoimmune encephalomyelitis-resistant mice have highly encephalitogenic myelin basic protein (MBP)-specific T cell clones that recognize a MBP peptide with high affinity for MHC class II.
    Abromson-Leeman S, Alexander J, Bronson R, Carroll J, Southwood S, Dorf M.
    J Immunol; 1995 Jan 01; 154(1):388-98. PubMed ID: 7527816
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  • 20. Prevention of experimental allergic encephalomyelitis in rats by targeting autoantigen to B cells: evidence that the protective mechanism depends on changes in the cytokine response and migratory properties of the autoantigen-specific T cells.
    Saoudi A, Simmonds S, Huitinga I, Mason D.
    J Exp Med; 1995 Aug 01; 182(2):335-44. PubMed ID: 7543135
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