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Journal Abstract Search

74 related items for PubMed ID: 12811851

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  • 24. Anti-MOG antibodies are under polygenic regulation with the most significant control coming from the C-type lectin-like gene locus.
    Flytzani S, Stridh P, Guerreiro-Cacais AO, Marta M, Hedreul MT, Jagodic M, Olsson T.
    Genes Immun; 2013 Oct; 14(7):409-19. PubMed ID: 23784360
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  • 28. Myelin/oligodendrocyte glycoprotein-deficient (MOG-deficient) mice reveal lack of immune tolerance to MOG in wild-type mice.
    Delarasse C, Daubas P, Mars LT, Vizler C, Litzenburger T, Iglesias A, Bauer J, Della Gaspera B, Schubart A, Decker L, Dimitri D, Roussel G, Dierich A, Amor S, Dautigny A, Liblau R, Pham-Dinh D.
    J Clin Invest; 2003 Aug; 112(4):544-53. PubMed ID: 12925695
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  • 30. Autoantibodies recognizing native MOG are closely associated with active demyelination but not with neuroinflammation in chronic EAE.
    Ohtani S, Kohyama K, Matsumoto Y.
    Neuropathology; 2011 Apr; 31(2):101-11. PubMed ID: 20573031
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  • 31. MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated.
    Flytzani S, Guerreiro-Cacais AO, N'diaye M, Lindner M, Linington C, Meinl E, Stridh P, Jagodic M, Olsson T.
    J Neuroinflammation; 2015 Oct 29; 12():194. PubMed ID: 26511327
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  • 32. FTY720 ameliorates MOG-induced experimental autoimmune encephalomyelitis by suppressing both cellular and humoral immune responses.
    Papadopoulos D, Rundle J, Patel R, Marshall I, Stretton J, Eaton R, Richardson JC, Gonzalez MI, Philpott KL, Reynolds R.
    J Neurosci Res; 2010 Feb 01; 88(2):346-59. PubMed ID: 19658199
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  • 33. TNF production in macrophages is genetically determined and regulates inflammatory disease in rats.
    Gillett A, Marta M, Jin T, Tuncel J, Leclerc P, Nohra R, Lange S, Holmdahl R, Olsson T, Harris RA, Jagodic M.
    J Immunol; 2010 Jul 01; 185(1):442-50. PubMed ID: 20505148
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  • 34. Acceleration of experimental autoimmune encephalomyelitis in interleukin-10-deficient mice: roles of interleukin-10 in disease progression and recovery.
    Samoilova EB, Horton JL, Chen Y.
    Cell Immunol; 1998 Sep 15; 188(2):118-24. PubMed ID: 9756642
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  • 35. A comparative analysis of B cell-mediated myelin oligodendrocyte glycoprotein-experimental autoimmune encephalomyelitis pathogenesis in B cell-deficient mice reveals an effect on demyelination.
    Svensson L, Abdul-Majid KB, Bauer J, Lassmann H, Harris RA, Holmdahl R.
    Eur J Immunol; 2002 Jul 15; 32(7):1939-46. PubMed ID: 12115614
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  • 36. Detection of arthritis-susceptibility loci, including Ncf1, and variable effects of the major histocompatibility complex region depending on genetic background in rats.
    Rintisch C, Förster M, Holmdahl R.
    Arthritis Rheum; 2009 Feb 15; 60(2):419-27. PubMed ID: 19180494
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  • 37. Limiting-dilution analysis of the frequency of myelin basic protein-reactive T cells in Lewis, PVG/c and BN rats. Implication for susceptibility to autoimmune encephalomyelitis.
    Matsumoto Y, Kawai K, Tomita Y, Fujiwara M.
    Immunology; 1990 Feb 15; 69(2):215-21. PubMed ID: 1689693
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  • 39. Regulation of experimental autoimmune encephalomyelitis by CD4+, CD25+ and CD8+ T cells: analysis using depleting antibodies.
    Montero E, Nussbaum G, Kaye JF, Perez R, Lage A, Ben-Nun A, Cohen IR.
    J Autoimmun; 2004 Aug 15; 23(1):1-7. PubMed ID: 15236747
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  • 40. Genome-wide linkage analysis of chronic relapsing experimental autoimmune encephalomyelitis in the rat identifies a major susceptibility locus on chromosome 9.
    Dahlman I, Jacobsson L, Glaser A, Lorentzen JC, Andersson M, Luthman H, Olsson T.
    J Immunol; 1999 Mar 01; 162(5):2581-8. PubMed ID: 10072499
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