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Journal Abstract Search


185 related items for PubMed ID: 12826020

  • 1. Baseline blood flow and bradykinin-induced vasodilator responses in the human forearm are insensitive to the cytochrome P450 2C9 (CYP2C9) inhibitor sulphaphenazole.
    Passauer J, Büssemaker E, Lässig G, Pistrosch F, Fauler J, Gross P, Fleming I.
    Clin Sci (Lond); 2003 Oct; 105(4):513-8. PubMed ID: 12826020
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  • 3. Identification of a cytochrome P450 2C9-derived endothelium-derived hyperpolarizing factor in essential hypertensive patients.
    Taddei S, Versari D, Cipriano A, Ghiadoni L, Galetta F, Franzoni F, Magagna A, Virdis A, Salvetti A.
    J Am Coll Cardiol; 2006 Aug 01; 48(3):508-15. PubMed ID: 16875977
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  • 4. Effect of sulfaphenazole on tissue plasminogen activator release in normotensive subjects and hypertensive patients.
    Giannarelli C, Virdis A, De Negri F, Magagna A, Duranti E, Salvetti A, Taddei S.
    Circulation; 2009 Mar 31; 119(12):1625-33. PubMed ID: 19289643
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  • 5. Direct biochemical evidence for eNOS stimulation by bradykinin in the human forearm vasculature.
    Lauer T, Kleinbongard P, Preik M, Rauch BH, Deussen A, Feelisch M, Strauer BE, Kelm M.
    Basic Res Cardiol; 2003 Mar 31; 98(2):84-9. PubMed ID: 12607129
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  • 6. The role of nitric oxide- and prostacyclin-independent vasodilatation in the human cutaneous microcirculation: effect of cytochrome P450 2C9 inhibition.
    Lenasi H.
    Clin Physiol Funct Imaging; 2009 Jul 31; 29(4):263-70. PubMed ID: 19291087
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  • 8. The contribution of nitric oxide and vasodilatory prostanoids to bradykinin-mediated vasodilation in Type 1 diabetes.
    Wotherspoon F, Browne DL, Meeking DR, Allard SE, Munday LJ, Shaw KM, Cummings MH.
    Diabet Med; 2005 Jun 31; 22(6):697-702. PubMed ID: 15910619
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  • 9. Block of inward rectifying K+ channels (KIR) inhibits bradykinin-induced vasodilatation in human forearm resistance vasculature.
    Dwivedi R, Saha S, Chowienczyk PJ, Ritter JM.
    Arterioscler Thromb Vasc Biol; 2005 Feb 31; 25(2):e7-9. PubMed ID: 15591222
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  • 11. Cytochrome P-450 2C9 signaling does not contribute to age-associated vascular endothelial dysfunction in humans.
    Donato AJ, Eskurza I, Jablonski KL, Gano LB, Pierce GL, Seals DR.
    J Appl Physiol (1985); 2008 Oct 31; 105(4):1359-63. PubMed ID: 18669935
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  • 12. [Vasodilator effects of bradykinin on the resistive circulation of the forearm of coronary patients].
    Benacerraf S, Carville C, Sediame S, Belhassen L, Adnot S, Dubois-Randé JL.
    Arch Mal Coeur Vaiss; 2000 Sep 31; 93(9):1103-8. PubMed ID: 11055001
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  • 13. Basal production of nitric oxide (NO) and non-NO vasodilators in the forearm microcirculation in Type 2 diabetes: associations with blood pressure and HDL cholesterol.
    Woodman RJ, Playford DA, Watts GF.
    Diabetes Res Clin Pract; 2006 Jan 31; 71(1):59-67. PubMed ID: 16029909
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  • 14. Vasodilator prostanoids, but not nitric oxide, may account for skeletal muscle hyperaemia in Type I diabetes mellitus.
    Skyrme-Jones RA, O'Brien RC, Meredith IT.
    Clin Sci (Lond); 2000 Nov 31; 99(5):383-92. PubMed ID: 11052918
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  • 15. Effects of arginine vasopressin and 1-desamino-8-D arginine vasopressin on forearm vasculature of healthy subjects and patients with a V2 receptor defect.
    van Lieburg AF, Knoers NV, Monnens LA, Smits P.
    J Hypertens; 1995 Dec 31; 13(12 Pt 2):1695-700. PubMed ID: 8903635
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  • 16. Contribution of nitric oxide and prostaglandins to reactive hyperemia in human forearm.
    Engelke KA, Halliwill JR, Proctor DN, Dietz NM, Joyner MJ.
    J Appl Physiol (1985); 1996 Oct 31; 81(4):1807-14. PubMed ID: 8904603
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  • 17. Forearm vasoconstriction in response to noradrenaline and NG-monomethyl-L-arginine in essential hypertension.
    Kneale BJ, Chowienczyk PJ, Brett SE, Cockcroft JR, Ritter JM.
    Clin Sci (Lond); 1999 Sep 31; 97(3):277-82. PubMed ID: 10464052
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  • 18. Ageing is associated with impairment of nitric oxide and prostanoid dilator pathways in the human forearm.
    Singh N, Prasad S, Singer DR, MacAllister RJ.
    Clin Sci (Lond); 2002 May 31; 102(5):595-600. PubMed ID: 11980580
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  • 19. Inflammation-induced endothelial dysfunction involves reduced nitric oxide bioavailability and increased oxidant stress.
    Clapp BR, Hingorani AD, Kharbanda RK, Mohamed-Ali V, Stephens JW, Vallance P, MacAllister RJ.
    Cardiovasc Res; 2004 Oct 01; 64(1):172-8. PubMed ID: 15364625
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  • 20. Calcitonin gene-related peptide-induced vasodilation in the human forearm is antagonized by CGRP8-37: evaluation of a human in vivo pharmacodynamic model.
    Vanmolkot FH, Van der Schueren BJ, de Hoon JN.
    Clin Pharmacol Ther; 2006 Mar 01; 79(3):263-73. PubMed ID: 16513450
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