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Journal Abstract Search

79 related items for PubMed ID: 12880425

  • 1. Involvement of oxidative stress in apoptosis induced by a mixture of isothiazolinones in normal human keratinocytes.
    Ettorre A, Andreassi M, Anselmi C, Neri P, Andreassi L, Di Stefano A.
    J Invest Dermatol; 2003 Aug; 121(2):328-36. PubMed ID: 12880425
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  • 2. GSH depletion, protein S-glutathionylation and mitochondrial transmembrane potential hyperpolarization are early events in initiation of cell death induced by a mixture of isothiazolinones in HL60 cells.
    Di Stefano A, Frosali S, Leonini A, Ettorre A, Priora R, Di Simplicio FC, Di Simplicio P.
    Biochim Biophys Acta; 2006 Feb; 1763(2):214-25. PubMed ID: 16458373
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  • 3. Effects of antioxidants and caspase-3 inhibitor on the phenylethyl isothiocyanate-induced apoptotic signaling pathways in human PLC/PRF/5 cells.
    Wu SJ, Ng LT, Lin CC.
    Eur J Pharmacol; 2005 Aug 22; 518(2-3):96-106. PubMed ID: 16054126
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  • 4. Contribution of reactive oxygen species and caspase-3 to apoptosis and attenuated ICAM-1 expression by paclitaxel-treated MDA-MB-435 breast carcinoma cells.
    Fawcett H, Mader JS, Robichaud M, Giacomantonio C, Hoskin DW.
    Int J Oncol; 2005 Dec 22; 27(6):1717-26. PubMed ID: 16273228
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  • 12. Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis.
    Fulda S, Meyer E, Friesen C, Susin SA, Kroemer G, Debatin KM.
    Oncogene; 2001 Mar 01; 20(9):1063-75. PubMed ID: 11314043
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  • 13. Caspase-1 is not involved in CD95/Fas-induced apoptosis in Jurkat T cells.
    Chow SC, Slee EA, MacFarlane M, Cohen GM.
    Exp Cell Res; 1999 Feb 01; 246(2):491-500. PubMed ID: 9925765
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  • 14. Caspase inhibition switches the mode of cell death induced by cyanide by enhancing reactive oxygen species generation and PARP-1 activation.
    Prabhakaran K, Li L, Borowitz JL, Isom GE.
    Toxicol Appl Pharmacol; 2004 Mar 01; 195(2):194-202. PubMed ID: 14998685
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  • 15. Oxidative stress and mitochondrial-mediated apoptosis in dopaminergic cells exposed to methylcyclopentadienyl manganese tricarbonyl.
    Kitazawa M, Wagner JR, Kirby ML, Anantharam V, Kanthasamy AG.
    J Pharmacol Exp Ther; 2002 Jul 01; 302(1):26-35. PubMed ID: 12065696
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  • 16. Oxidative stress induces caspase-independent retinal apoptosis in vitro.
    Carmody RJ, Cotter TG.
    Cell Death Differ; 2000 Mar 01; 7(3):282-91. PubMed ID: 10745273
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  • 17. A rapid and transient ROS generation by cadmium triggers apoptosis via caspase-dependent pathway in HepG2 cells and this is inhibited through N-acetylcysteine-mediated catalase upregulation.
    Oh SH, Lim SC.
    Toxicol Appl Pharmacol; 2006 May 01; 212(3):212-23. PubMed ID: 16169029
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  • 18. The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.
    Hirsch T, Marchetti P, Susin SA, Dallaporta B, Zamzami N, Marzo I, Geuskens M, Kroemer G.
    Oncogene; 1997 Sep 25; 15(13):1573-81. PubMed ID: 9380409
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  • 19. Functional dissociation of DeltaPsim and cytochrome c release defines the contribution of mitochondria upstream of caspase activation during granzyme B-induced apoptosis.
    Waterhouse NJ, Sedelies KA, Sutton VR, Pinkoski MJ, Thia KY, Johnstone R, Bird PI, Green DR, Trapani JA.
    Cell Death Differ; 2006 Apr 25; 13(4):607-18. PubMed ID: 16167065
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  • 20. Differential caspase-9-dependent signaling pathway between tumor necrosis factor receptor- and Fas-mediated hepatocyte apoptosis in mice.
    Imao M, Nagaki M, Imose M, Moriwaki H.
    Liver Int; 2006 Feb 25; 26(1):137-46. PubMed ID: 16420519
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