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285 related items for PubMed ID: 12884289

  • 21. Interferon-gamma-induced sensitization of colon carcinomas to ZD9331 targets caspases, downstream of Fas, independent of mitochondrial signaling and the inhibitor of apoptosis survivin.
    Geller J, Petak I, Szucs KS, Nagy K, Tillman DM, Houghton JA.
    Clin Cancer Res; 2003 Dec 15; 9(17):6504-15. PubMed ID: 14695155
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  • 22. IL-7 inhibits dexamethasone-induced apoptosis via Akt/PKB in mature, peripheral T cells.
    Sade H, Sarin A.
    Eur J Immunol; 2003 Apr 15; 33(4):913-9. PubMed ID: 12672057
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  • 23. Inhibition of caspase activity delays apoptosis in a transfected NS/0 myeloma cell line.
    McKenna SL, Cotter TG.
    Biotechnol Bioeng; 2000 Jan 20; 67(2):165-76. PubMed ID: 10592514
    [Abstract] [Full Text] [Related]

  • 24. Caspase-dependent chemotherapy-induced death of glioma cells requires mitochondrial cytochrome c release.
    Glaser T, Weller M.
    Biochem Biophys Res Commun; 2001 Feb 23; 281(2):322-7. PubMed ID: 11181049
    [Abstract] [Full Text] [Related]

  • 25. Smac induces cytochrome c release and apoptosis independently from Bax/Bcl-x(L) in a strictly caspase-3-dependent manner in human carcinoma cells.
    Hasenjäger A, Gillissen B, Müller A, Normand G, Hemmati PG, Schuler M, Dörken B, Daniel PT.
    Oncogene; 2004 Jun 03; 23(26):4523-35. PubMed ID: 15064710
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  • 26. Lovastatin inhibits tumor growth and lung metastasis in mouse mammary carcinoma model: a p53-independent mitochondrial-mediated apoptotic mechanism.
    Shibata MA, Ito Y, Morimoto J, Otsuki Y.
    Carcinogenesis; 2004 Oct 03; 25(10):1887-98. PubMed ID: 15180944
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  • 27. The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.
    Hirsch T, Marchetti P, Susin SA, Dallaporta B, Zamzami N, Marzo I, Geuskens M, Kroemer G.
    Oncogene; 1997 Sep 25; 15(13):1573-81. PubMed ID: 9380409
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  • 28. Functional dissociation of DeltaPsim and cytochrome c release defines the contribution of mitochondria upstream of caspase activation during granzyme B-induced apoptosis.
    Waterhouse NJ, Sedelies KA, Sutton VR, Pinkoski MJ, Thia KY, Johnstone R, Bird PI, Green DR, Trapani JA.
    Cell Death Differ; 2006 Apr 25; 13(4):607-18. PubMed ID: 16167065
    [Abstract] [Full Text] [Related]

  • 29. The BH3 domain is required for caspase-independent cell death induced by Bax and oligomycin.
    Fitch ME, Chang CM, Parslow TG.
    Cell Death Differ; 2000 Apr 25; 7(4):338-49. PubMed ID: 10773818
    [Abstract] [Full Text] [Related]

  • 30. Apoptotic crosstalk between the endoplasmic reticulum and mitochondria controlled by Bcl-2.
    Häcki J, Egger L, Monney L, Conus S, Rossé T, Fellay I, Borner C.
    Oncogene; 2000 May 04; 19(19):2286-95. PubMed ID: 10822379
    [Abstract] [Full Text] [Related]

  • 31. Phosphatidylserine externalization during differentiation-triggered apoptosis of erythroleukemic cells.
    Diaz C, Lee AT, McConkey DJ, Schroit AJ.
    Cell Death Differ; 1999 Mar 04; 6(3):218-26. PubMed ID: 10200572
    [Abstract] [Full Text] [Related]

  • 32. Acceleration of apoptotic cell death after the cleavage of Bcl-XL protein by caspase-3-like proteases.
    Fujita N, Nagahashi A, Nagashima K, Rokudai S, Tsuruo T.
    Oncogene; 1998 Sep 10; 17(10):1295-304. PubMed ID: 9771973
    [Abstract] [Full Text] [Related]

  • 33. Bcl-2 regulates chondrocyte morphology and aggrecan gene expression independent of caspase activation and full apoptosis.
    Feng L, Balakir R, Precht P, Horton WE.
    J Cell Biochem; 1999 Sep 15; 74(4):576-86. PubMed ID: 10440927
    [Abstract] [Full Text] [Related]

  • 34. Bcl-2 protects against hyperoxia-induced apoptosis through inhibition of the mitochondria-dependent pathway.
    Métrailler-Ruchonnet I, Pagano A, Carnesecchi S, Ody C, Donati Y, Barazzone Argiroffo C.
    Free Radic Biol Med; 2007 Apr 01; 42(7):1062-74. PubMed ID: 17349933
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  • 35. Diosgenin induces apoptosis in HeLa cells via activation of caspase pathway.
    Hou R, Zhou QL, Wang BX, Tashiro S, Onodera S, Ikejima T.
    Acta Pharmacol Sin; 2004 Aug 01; 25(8):1077-82. PubMed ID: 15301743
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  • 36. Apoptosis of hematopoietic cells induced by growth factor withdrawal is associated with caspase-9 mediated cleavage of Raf-1.
    Cornelis S, Bruynooghe Y, Van Loo G, Saelens X, Vandenabeele P, Beyaert R.
    Oncogene; 2005 Feb 24; 24(9):1552-62. PubMed ID: 15674327
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  • 37. Blocking granule-mediated death by primary human NK cells requires both protection of mitochondria and inhibition of caspase activity.
    Sedelies KA, Ciccone A, Clarke CJ, Oliaro J, Sutton VR, Scott FL, Silke J, Susanto O, Green DR, Johnstone RW, Bird PI, Trapani JA, Waterhouse NJ.
    Cell Death Differ; 2008 Apr 24; 15(4):708-17. PubMed ID: 18202705
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  • 38. Feedback regulation of mitochondria by caspase-9 in the B cell receptor-mediated apoptosis.
    Eeva J, Nuutinen U, Ropponen A, Mättö M, Eray M, Pellinen R, Wahlfors J, Pelkonen J.
    Scand J Immunol; 2009 Dec 24; 70(6):574-83. PubMed ID: 19906200
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  • 39. Cancer-specific toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mitochondrial cell-death mediators by a Nur77-dependent pathway.
    Maddika S, Booy EP, Johar D, Gibson SB, Ghavami S, Los M.
    J Cell Sci; 2005 Oct 01; 118(Pt 19):4485-93. PubMed ID: 16179607
    [Abstract] [Full Text] [Related]

  • 40. Inhibition of N-methyl-D-aspartate receptors increases paraoxon-induced apoptosis in cultured neurons.
    Wu X, Tian F, Okagaki P, Marini AM.
    Toxicol Appl Pharmacol; 2005 Oct 01; 208(1):57-67. PubMed ID: 16164961
    [Abstract] [Full Text] [Related]


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