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Journal Abstract Search

142 related items for PubMed ID: 12941042

  • 1. Endogenous alpha2-antiplasmin does not enhance glomerular fibrin deposition or injury in glomerulonephritis.
    Kitching AR, Turner AL, O'Sullivan KM, Dewerchin M, Lijnen HR.
    J Thromb Haemost; 2003 Sep; 1(9):1992-9. PubMed ID: 12941042
    [Abstract] [Full Text] [Related]

  • 2. Plasminogen and plasminogen activators protect against renal injury in crescentic glomerulonephritis.
    Kitching AR, Holdsworth SR, Ploplis VA, Plow EF, Collen D, Carmeliet P, Tipping PG.
    J Exp Med; 1997 Mar 03; 185(5):963-8. PubMed ID: 9120402
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  • 3. Glomerular fibrinolytic activity in anti-GBM glomerulonephritis in rabbits.
    Malliaros J, Holdsworth SR, Wojta J, Erlich J, Tipping PG.
    Kidney Int; 1993 Sep 03; 44(3):557-64. PubMed ID: 8231028
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  • 4. Plasminogen activator inhibitor-1 is a significant determinant of renal injury in experimental crescentic glomerulonephritis.
    Kitching AR, Kong YZ, Huang XR, Davenport P, Edgtton KL, Carmeliet P, Holdsworth SR, Tipping PG.
    J Am Soc Nephrol; 2003 Jun 03; 14(6):1487-95. PubMed ID: 12761249
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  • 5. Tissue factor initiates glomerular fibrin deposition and promotes major histocompatibility complex class II expression in crescentic glomerulonephritis.
    Erlich JH, Holdsworth SR, Tipping PG.
    Am J Pathol; 1997 Mar 03; 150(3):873-80. PubMed ID: 9060825
    [Abstract] [Full Text] [Related]

  • 6. Enhanced fibrinolytic potential in mice with combined homozygous deficiency of alpha2-antiplasmin and PAI-1.
    Dewerchin M, Collen D, Lijnen HR.
    Thromb Haemost; 2001 Aug 03; 86(2):640-6. PubMed ID: 11522016
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  • 8. Tissue factor pathway inhibitor expression in human crescentic glomerulonephritis.
    Cunningham MA, Ono T, Hewitson TD, Tipping PG, Becker GJ, Holdsworth SR.
    Kidney Int; 1999 Apr 03; 55(4):1311-8. PubMed ID: 10200995
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  • 9. Crescentic glomerulonephritis is diminished in fibrinogen-deficient mice.
    Drew AF, Tucker HL, Liu H, Witte DP, Degen JL, Tipping PG.
    Am J Physiol Renal Physiol; 2001 Dec 03; 281(6):F1157-63. PubMed ID: 11704568
    [Abstract] [Full Text] [Related]

  • 10. Macrophage-induced glomerular fibrin deposition in experimental glomerulonephritis in the rabbit.
    Holdsworth SR, Tipping PG.
    J Clin Invest; 1985 Oct 03; 76(4):1367-74. PubMed ID: 4056035
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  • 11. Deficiency of growth factor midkine exacerbates necrotizing glomerular injuries in progressive glomerulonephritis.
    Kojima H, Kosugi T, Sato W, Sato Y, Maeda K, Kato N, Kato K, Inaba S, Ishimoto T, Tsuboi N, Matsuo S, Maruyama S, Yuzawa Y, Kadomatsu K.
    Am J Pathol; 2013 Feb 03; 182(2):410-9. PubMed ID: 23201132
    [Abstract] [Full Text] [Related]

  • 12. Protease-activated receptor-2 augments experimental crescentic glomerulonephritis.
    Moussa L, Apostolopoulos J, Davenport P, Tchongue J, Tipping PG.
    Am J Pathol; 2007 Sep 03; 171(3):800-8. PubMed ID: 17640968
    [Abstract] [Full Text] [Related]

  • 13. Intrinsic renal cells are the major source of tumor necrosis factor contributing to renal injury in murine crescentic glomerulonephritis.
    Timoshanko JR, Sedgwick JD, Holdsworth SR, Tipping PG.
    J Am Soc Nephrol; 2003 Jul 03; 14(7):1785-93. PubMed ID: 12819238
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  • 14. Renal expression of tissue factor pathway inhibitor and evidence for a role in crescentic glomerulonephritis in rabbits.
    Erlich JH, Apostolopoulos J, Wun TC, Kretzmer KK, Holdsworth SR, Tipping PG.
    J Clin Invest; 1996 Jul 15; 98(2):325-35. PubMed ID: 8755641
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  • 16. Alpha2-antiplasmin gene deficiency in mice is associated with enhanced fibrinolytic potential without overt bleeding.
    Lijnen HR, Okada K, Matsuo O, Collen D, Dewerchin M.
    Blood; 1999 Apr 01; 93(7):2274-81. PubMed ID: 10090937
    [Abstract] [Full Text] [Related]

  • 17. Role of plasminogen system components in focal cerebral ischemic infarction: a gene targeting and gene transfer study in mice.
    Nagai N, De Mol M, Lijnen HR, Carmeliet P, Collen D.
    Circulation; 1999 May 11; 99(18):2440-4. PubMed ID: 10318667
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  • 18. Glomerular capillary and endothelial cell injury is associated with the formation of necrotizing and crescentic lesions in crescentic glomerulonephritis.
    Fujita E, Nagahama K, Shimizu A, Aoki M, Higo S, Yasuda F, Mii A, Fukui M, Kaneko T, Tsuruoka S.
    J Nippon Med Sch; 2015 May 11; 82(1):27-35. PubMed ID: 25797872
    [Abstract] [Full Text] [Related]

  • 19. Releasing the Brakes on the Fibrinolytic System in Pulmonary Emboli: Unique Effects of Plasminogen Activation and α2-Antiplasmin Inactivation.
    Singh S, Houng A, Reed GL.
    Circulation; 2017 Mar 14; 135(11):1011-1020. PubMed ID: 28028005
    [Abstract] [Full Text] [Related]

  • 20. Apoptosis in progressive crescentic glomerulonephritis.
    Shimizu A, Masuda Y, Kitamura H, Ishizaki M, Sugisaki Y, Yamanaka N.
    Lab Invest; 1996 May 14; 74(5):941-51. PubMed ID: 8642789
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