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290 related items for PubMed ID: 12956712

  • 1. Increase in Bcl-2 phosphorylation and reduced levels of BH3-only Bcl-2 family proteins in kainic acid-mediated neuronal death in the rat brain.
    Korhonen L, Belluardo N, Mudo G, Lindholm D.
    Eur J Neurosci; 2003 Sep; 18(5):1121-34. PubMed ID: 12956712
    [Abstract] [Full Text] [Related]

  • 2. Bcl-2, Bax and Bcl-x expression following kainic acid administration at convulsant doses in the rat.
    López E, Pozas E, Rivera R, Ferrer I.
    Neuroscience; 1999 Sep; 91(4):1461-70. PubMed ID: 10391451
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  • 3. Domoic acid-induced neuronal damage in the rat hippocampus: changes in apoptosis related genes (bcl-2, bax, caspase-3) and microglial response.
    Ananth C, Thameem Dheen S, Gopalakrishnakone P, Kaur C.
    J Neurosci Res; 2001 Oct 15; 66(2):177-90. PubMed ID: 11592113
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  • 4. Mild traumatic brain injury induces apoptotic cell death in the cortex that is preceded by decreases in cellular Bcl-2 immunoreactivity.
    Raghupathi R, Conti AC, Graham DI, Krajewski S, Reed JC, Grady MS, Trojanowski JQ, McIntosh TK.
    Neuroscience; 2002 Oct 15; 110(4):605-16. PubMed ID: 11934469
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  • 5. The proapoptotic BCL-2 homology domain 3-only protein Bim is not critical for acute excitotoxic cell death.
    Theofilas P, Bedner P, Hüttmann K, Theis M, Steinhäuser C, Frank S.
    J Neuropathol Exp Neurol; 2009 Jan 15; 68(1):102-10. PubMed ID: 19104441
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  • 6. Brain-derived neurotrophic factor prevents changes in Bcl-2 family members and caspase-3 activation induced by excitotoxicity in the striatum.
    Pérez-Navarro E, Gavaldà N, Gratacòs E, Alberch J.
    J Neurochem; 2005 Feb 15; 92(3):678-91. PubMed ID: 15659237
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  • 11. DNA damage and nonhomologous end joining in excitotoxicity: neuroprotective role of DNA-PKcs in kainic acid-induced seizures.
    Neema M, Navarro-Quiroga I, Chechlacz M, Gilliams-Francis K, Liu J, Lamonica K, Lin SL, Naegele JR.
    Hippocampus; 2005 Feb 15; 15(8):1057-71. PubMed ID: 16216017
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  • 12. Radiation-induced apoptosis in developing rats and kainic acid-induced excitotoxicity in adult rats are associated with distinctive morphological and biochemical c-Jun/AP-1 (N) expression.
    Ferrer I, Planas AM, Pozas E.
    Neuroscience; 1997 Sep 15; 80(2):449-58. PubMed ID: 9284348
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  • 13. Bcl-2 and Bax proteins are increased in neocortical but not in thalamic apoptosis following devascularizing lesion of the cerebral cortex in the rat: an immunohistochemical study.
    Sulejczak D, Czarkowska-Bauch J, Macias M, Skup M.
    Brain Res; 2004 May 01; 1006(2):133-49. PubMed ID: 15051517
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  • 15. Kainic acid-induced excitotoxicity is associated with a complex c-Fos and c-Jun response which does not preclude either cell death or survival.
    Pozas E, Ballabriga J, Planas AM, Ferrer I.
    J Neurobiol; 1997 Sep 01; 33(3):232-46. PubMed ID: 9298762
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  • 16. Alterations in the cellular distribution of bcl-2, bcl-x and bax in the adult rat substantia nigra following striatal 6-hydroxydopamine lesions.
    Kramer BC, Mytilineou C.
    J Neurocytol; 2004 Mar 01; 33(2):213-23. PubMed ID: 15322379
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  • 17. Expression and activation of caspase 3 following status epilepticus in the rat.
    Narkilahti S, Pirttilä TJ, Lukasiuk K, Tuunanen J, Pitkänen A.
    Eur J Neurosci; 2003 Sep 01; 18(6):1486-96. PubMed ID: 14511328
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  • 18. Quantification and localization of kainic acid-induced neurotoxicity employing a new biomarker of cell death: cleaved microtubule-associated protein-tau (C-tau).
    Zemlan FP, Mulchahey JJ, Gudelsky GA.
    Neuroscience; 2003 Sep 01; 121(2):399-409. PubMed ID: 14521998
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  • 19. c-Jun N-terminal kinase signaling pathway in excitotoxic cell death following kainic acid-induced status epilepticus.
    Spigolon G, Veronesi C, Bonny C, Vercelli A.
    Eur J Neurosci; 2010 Apr 01; 31(7):1261-72. PubMed ID: 20345908
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  • 20. Neuroprotection of adenoviral-vector-mediated GDNF expression against kainic-acid-induced excitotoxicity in the rat hippocampus.
    Yoo YM, Lee CJ, Lee U, Kim YJ.
    Exp Neurol; 2006 Aug 01; 200(2):407-17. PubMed ID: 16690057
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